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Disqueratose congênita
ORPHA:1775CID-10 · Q82.8CID-11 · 3A70.0DOENÇA RARA

A disqueratose congênita (DC) é uma displasia ectodérmica rara que frequentemente se apresenta com a tríade clássica de displasia ungueal, alterações pigmentares da pele e leucoplasia oral associada a um alto risco de insuficiência da medula óssea (BMF) e câncer.

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Introdução

O que você precisa saber de cara

📋

A disqueratose congênita (DC) é uma displasia ectodérmica rara que frequentemente se apresenta com a tríade clássica de displasia ungueal, alterações pigmentares da pele e leucoplasia oral associada a um alto risco de insuficiência da medula óssea (BMF) e câncer.

Pesquisas ativas
7 ensaios
20 total registrados no ClinicalTrials.gov
Publicações científicas
1.251 artigos
Último publicado: 2026 Apr

Escala de raridade

CLASSIFICAÇÃO ORPHANET · BRASIL 2024
1-9 / 1 000 000
Ultra-rara
<1/50k
Muito rara
1/20k
Rara
1/10k
Pouco freq.
1/5k
Incomum
1/2k
Prevalência
0.1
Europe
Início
Adolescent
+ adult, childhood, infancy, neonatal
🏥
SUS: Cobertura mínimaScore: 35%
Centros em: PA, PR, SC, RS, ES +10CID-10: Q82.8
🇧🇷Dados SUS / DATASUS
PROCEDIMENTOS SIGTAP (5)
0202010503
Cariótipo — bandas G, Q ou Rgenetic_test
0202010600
Pesquisa de microdeleções/microduplicações por FISHlab_test
0202010694
Sequenciamento completo do exoma (WES)rehabilitation
0202010260
Dosagem de alfa-fetoproteína
0301070040
Atendimento em reabilitação — doenças raras
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Sinais e sintomas

O que aparece no corpo e com que frequência cada sintoma acontece

Partes do corpo afetadas

🧬
Pele e cabelo
29 sintomas
🫃
Digestivo
16 sintomas
🩸
Sangue
13 sintomas
🧠
Neurológico
12 sintomas
👁️
Olhos
12 sintomas
🦴
Ossos e articulações
12 sintomas

+ 62 sintomas em outras categorias

Características mais comuns

90%prev.
Mácula
Muito frequente (99-80%)
90%prev.
Morfologia anormal da unha
Muito frequente (99-80%)
90%prev.
Distrofia ungueal
Muito frequente (99-80%)
90%prev.
Anormalidade dos neutrófilos
Muito frequente (99-80%)
90%prev.
Bolhas anormais na pele
Muito frequente (99-80%)
90%prev.
Anemia
Muito frequente (99-80%)
185sintomas
Muito frequente (9)
Frequente (29)
Ocasional (24)
Sem dados (123)

Os sintomas variam de pessoa para pessoa. Abaixo estão as 185 características clínicas mais associadas, ordenadas por frequência.

MáculaMacule
Muito frequente (99-80%)90%
Morfologia anormal da unhaAbnormal fingernail morphology
Muito frequente (99-80%)90%
Distrofia unguealNail dystrophy
Muito frequente (99-80%)90%
Anormalidade dos neutrófilosAbnormality of neutrophils
Muito frequente (99-80%)90%
Bolhas anormais na peleAbnormal blistering of the skin
Muito frequente (99-80%)90%

Linha do tempo da pesquisa

Publicações por ano — veja quando o interesse científico cresceu
Anos de pesquisa1desde 2026
Total histórico1.251PubMed
Últimos 10 anos200publicações
Pico202351 papers
Linha do tempo
2026Hoje · 2026🧪 1994Primeiro ensaio clínico📈 2023Ano de pico
Publicações por ano (últimos 10 anos)

Encontrou um erro ou informação desatualizada? Sugira uma correção →

Genética e causas

O que está alterado no DNA e como passa nas famílias

Genes associados

15 genes identificados com associação a esta condição. Padrão de herança: Autosomal dominant, Autosomal recessive, X-linked recessive.

TINF2TERF1-interacting nuclear factor 2Disease-causing germline mutation(s) inTolerante
FUNÇÃO

Component of the shelterin complex (telosome) that is involved in the regulation of telomere length and protection. Shelterin associates with arrays of double-stranded TTAGGG repeats added by telomerase and protects chromosome ends; without its protective activity, telomeres are no longer hidden from the DNA damage surveillance and chromosome ends are inappropriately processed by DNA repair pathways. Plays a role in shelterin complex assembly. Isoform 1 may have additional role in tethering telo

LOCALIZAÇÃO

NucleusChromosome, telomereNucleus matrix

VIAS BIOLÓGICAS (10)
DNA Damage/Telomere Stress Induced SenescencePackaging Of Telomere EndsMeiotic synapsisInhibition of DNA recombination at telomereTelomere C-strand (Lagging Strand) Synthesis
MECANISMO DE DOENÇA

Dyskeratosis congenita, autosomal dominant, 3

A rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy.

EXPRESSÃO TECIDUAL(Ubíquo)
Útero
88.3 TPM
Nervo tibial
85.3 TPM
Artéria tibial
82.2 TPM
Baço
82.0 TPM
Glândula adrenal
81.4 TPM
OUTRAS DOENÇAS (4)
Revesz syndromedyskeratosis congenita, autosomal dominant 3dyskeratosis congenitaHoyeraal-Hreidarsson syndrome
HGNC:11824UniProt:Q9BSI4
ACDAdrenocortical dysplasia protein homologDisease-causing germline mutation(s) inTolerante
FUNÇÃO

Component of the shelterin complex (telosome) that is involved in the regulation of telomere length and protection. Shelterin associates with arrays of double-stranded TTAGGG repeats added by telomerase and protects chromosome ends. Without its protective activity, telomeres are no longer hidden from the DNA damage surveillance and chromosome ends are inappropriately processed by DNA repair pathways. Promotes binding of POT1 to single-stranded telomeric DNA. Modulates the inhibitory effects of P

LOCALIZAÇÃO

NucleusChromosome, telomere

VIAS BIOLÓGICAS (10)
DNA Damage/Telomere Stress Induced SenescencePackaging Of Telomere EndsMeiotic synapsisInhibition of DNA recombination at telomereTelomere C-strand (Lagging Strand) Synthesis
MECANISMO DE DOENÇA

Dyskeratosis congenita, autosomal dominant, 6

A form of dyskeratosis congenita, a rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy.

OUTRAS DOENÇAS (4)
dyskeratosis congenita, autosomal dominant 6inherited aplastic anemiafamilial melanomaHoyeraal-Hreidarsson syndrome
HGNC:25070UniProt:Q96AP0
DCLRE1B5' exonuclease ApolloDisease-causing germline mutation(s) inRestrito
FUNÇÃO

5'-3' exonuclease that plays a central role in telomere maintenance and protection during S-phase. Participates in the protection of telomeres against non-homologous end-joining (NHEJ)-mediated repair, thereby ensuring that telomeres do not fuse. Plays a key role in telomeric loop (T loop) formation by being recruited by TERF2 at the leading end telomeres and by processing leading-end telomeres immediately after their replication via its exonuclease activity: generates 3' single-stranded overhan

LOCALIZAÇÃO

Chromosome, telomereNucleusCytoplasm, cytoskeleton, microtubule organizing center, centrosome

VIAS BIOLÓGICAS (1)
Fanconi Anemia Pathway
MECANISMO DE DOENÇA

Dyskeratosis congenita, autosomal recessive, 8

A form of dyskeratosis congenita, a rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy. Additional DKCB8 features include microcephaly, intrauterine growth retardation, and developmental anomalies in some patients. DKCB8 patients exhibit normal global telemore length, although there is evidence of telomere instability.

VIAS REACTOME (1)
EXPRESSÃO TECIDUAL(Ubíquo)
Fibroblastos
12.9 TPM
Linfócitos
10.9 TPM
Cérebro - Hemisfério cerebelar
9.9 TPM
Cerebelo
7.8 TPM
Artéria tibial
7.8 TPM
OUTRAS DOENÇAS (1)
dyskeratosis congenita, autosomal recessive 8
HGNC:HGNC:17641UniProt:Q9H816
NOP10H/ACA ribonucleoprotein complex subunit 3Disease-causing germline mutation(s) inTolerante
FUNÇÃO

Required for ribosome biogenesis and telomere maintenance. Part of the H/ACA small nucleolar ribonucleoprotein (H/ACA snoRNP) complex, which catalyzes pseudouridylation of rRNA (PubMed:32554502). This involves the isomerization of uridine such that the ribose is subsequently attached to C5, instead of the normal N1. Each rRNA can contain up to 100 pseudouridine ('psi') residues, which may serve to stabilize the conformation of rRNAs. May also be required for correct processing or intranuclear tr

LOCALIZAÇÃO

Nucleus, nucleolusNucleus, Cajal body

VIAS BIOLÓGICAS (2)
Telomere Extension By TelomeraserRNA modification in the nucleus and cytosol
MECANISMO DE DOENÇA

Dyskeratosis congenita, autosomal recessive, 1

A rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy.

EXPRESSÃO TECIDUAL(Ubíquo)
Fibroblastos
349.8 TPM
Linfócitos
344.0 TPM
Sangue
272.8 TPM
Pulmão
208.1 TPM
Baço
201.7 TPM
OUTRAS DOENÇAS (4)
cataracts, hearing impairment, nephrotic syndrome, and enterocolitis 2pulmonary fibrosis and/or bone marrow failure syndrome, telomere-related, 9dyskeratosis congenita, autosomal recessive 1dyskeratosis congenita
HGNC:14378UniProt:Q9NPE3
TYMSThymidylate synthaseDisease-causing germline mutation(s) inTolerante
FUNÇÃO

Catalyzes the reductive methylation of 2'-deoxyuridine 5'-monophosphate (dUMP) to thymidine 5'-monophosphate (dTMP), using the cosubstrate, 5,10- methylenetetrahydrofolate (CH2H4folate) as a 1-carbon donor and reductant and contributes to the mitochondrial and nuclear de novo thymidylate biosynthesis pathway

LOCALIZAÇÃO

NucleusCytoplasmMitochondrionMitochondrion matrixMitochondrion inner membrane

VIAS BIOLÓGICAS (2)
Interconversion of nucleotide di- and triphosphatesG1/S-Specific Transcription
MECANISMO DE DOENÇA

Dyskeratosis congenita, digenic

A form of dyskeratosis congenita, a rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy. DKCD transmission pattern is consistent with digenic inheritance.

EXPRESSÃO TECIDUAL(Ubíquo)
Linfócitos
198.0 TPM
Fibroblastos
63.1 TPM
Testículo
55.8 TPM
Esôfago - Mucosa
14.8 TPM
Intestino delgado
9.8 TPM
OUTRAS DOENÇAS (2)
dyskeratosis congenita, digenicdyskeratosis congenita
HGNC:12441UniProt:P04818
NHP2H/ACA ribonucleoprotein complex subunit 2Disease-causing germline mutation(s) inTolerante
FUNÇÃO

Required for ribosome biogenesis and telomere maintenance. Part of the H/ACA small nucleolar ribonucleoprotein (H/ACA snoRNP) complex, which catalyzes pseudouridylation of rRNA. This involves the isomerization of uridine such that the ribose is subsequently attached to C5, instead of the normal N1. Each rRNA can contain up to 100 pseudouridine ('psi') residues, which may serve to stabilize the conformation of rRNAs. May also be required for correct processing or intranuclear trafficking of TERC,

LOCALIZAÇÃO

Nucleus, nucleolusNucleus, Cajal body

VIAS BIOLÓGICAS (2)
Telomere Extension By TelomeraserRNA modification in the nucleus and cytosol
MECANISMO DE DOENÇA

Dyskeratosis congenita, autosomal recessive, 2

A rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy.

EXPRESSÃO TECIDUAL(Ubíquo)
Fibroblastos
177.2 TPM
Linfócitos
169.5 TPM
Esôfago - Mucosa
136.9 TPM
Ovário
119.1 TPM
Cervix Endocervix
115.3 TPM
OUTRAS DOENÇAS (2)
dyskeratosis congenita, autosomal recessive 2dyskeratosis congenita
HGNC:14377UniProt:Q9NX24
TERCDisease-causing germline mutation(s) inDesconhecido
LOCALIZAÇÃO

VIAS REACTOME (1)
OUTRAS DOENÇAS (5)
dyskeratosis congenita, autosomal dominant 1pulmonary fibrosis and/or bone marrow failure, Telomere-related, 2dyskeratosis congenitaidiopathic aplastic anemia
HGNC:11727
WRAP53Telomerase Cajal body protein 1Disease-causing germline mutation(s) inTolerante
FUNÇÃO

RNA chaperone that plays a key role in telomere maintenance and RNA localization to Cajal bodies (PubMed:29695869, PubMed:29804836). Specifically recognizes and binds the Cajal body box (CAB box) present in both small Cajal body RNAs (scaRNAs) and telomerase RNA template component (TERC) (PubMed:19285445, PubMed:20351177, PubMed:29695869, PubMed:29804836). Essential component of the telomerase holoenzyme complex, a ribonucleoprotein complex essential for the replication of chromosome termini tha

LOCALIZAÇÃO

Nucleus, Cajal bodyChromosome, telomereChromosome

VIAS BIOLÓGICAS (1)
Telomere Extension By Telomerase
MECANISMO DE DOENÇA

Dyskeratosis congenita, autosomal recessive, 3

A rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy.

EXPRESSÃO TECIDUAL(Ubíquo)
Linfócitos
13.6 TPM
Testículo
10.1 TPM
Fibroblastos
9.9 TPM
Baço
9.6 TPM
Cervix Endocervix
8.0 TPM
OUTRAS DOENÇAS (2)
dyskeratosis congenita, autosomal recessive 3dyskeratosis congenita
HGNC:25522UniProt:Q9BUR4
USB1U6 snRNA phosphodiesterase 1Disease-causing germline mutation(s) inTolerante
FUNÇÃO

3'-5' RNA exonuclease that trims the 3' end of oligo(U) and oligo(A) tracts of the pre-U6 small nuclear RNA (snRNA) molecule, leading to the formation of a mature U6 snRNA 3' end-terminated with a 2',3'-cyclic phosphate (PubMed:22899009, PubMed:23022480, PubMed:23190533, PubMed:26213367, PubMed:28887445, PubMed:30215753, PubMed:31832688). Participates in the U6 snRNA 3' end processing that prevents U6 snRNA degradation (PubMed:22899009, PubMed:23022480, PubMed:23190533, PubMed:26213367, PubMed:2

LOCALIZAÇÃO

Nucleus

MECANISMO DE DOENÇA

Poikiloderma with neutropenia

A genodermatosis characterized by poikiloderma, pachyonychia and chronic neutropenia. The disorder starts as a papular erythematous rash on the limbs during the first year of life. It gradually spreads centripetally and, as the papular rash resolves, hypo- and hyperpigmentation result, with development of telangiectasias. Another skin manifestation is pachyonychia, but alopecia and leukoplakia are distinctively absent. Patients have recurrent pneumonias that usually result in reactive airway disease and/or chronic cough. One of the most important extracutaneous symptoms is an increased susceptibility to infections, mainly affecting the respiratory system, primarily due to a chronic neutropenia and to neutrophil functional defects. Bone marrow abnormalities account for neutropenia and may evolve into myelodysplasia associated with the risk of leukemic transformation. Poikiloderma with neutropenia shows phenotypic overlap with Rothmund-Thomson syndrome.

EXPRESSÃO TECIDUAL(Ubíquo)
Sangue
54.4 TPM
Fibroblastos
29.1 TPM
Tireoide
26.3 TPM
Testículo
26.3 TPM
Esôfago - Muscular
25.6 TPM
OUTRAS DOENÇAS (2)
poikiloderma with neutropeniadyskeratosis congenita
HGNC:25792UniProt:Q9BQ65
CTC1CST complex subunit CTC1Disease-causing germline mutation(s) inTolerante
FUNÇÃO

Component of the CST complex proposed to act as a specialized replication factor promoting DNA replication under conditions of replication stress or natural replication barriers such as the telomere duplex. The CST complex binds single-stranded DNA with high affinity in a sequence-independent manner, while isolated subunits bind DNA with low affinity by themselves. Initially the CST complex has been proposed to protect telomeres from DNA degradation (PubMed:19854130). However, the CST complex ha

LOCALIZAÇÃO

NucleusChromosome, telomere

VIAS BIOLÓGICAS (2)
Polymerase switching on the C-strand of the telomereTelomere C-strand synthesis initiation
MECANISMO DE DOENÇA

Cerebroretinal microangiopathy with calcifications and cysts 1

An autosomal recessive pleiomorphic disorder characterized primarily by intracranial calcifications, leukodystrophy, and brain cysts, resulting in spasticity, ataxia, dystonia, seizures, and cognitive decline. Patients also have retinal telangiectasia and exudates (Coats disease) as well as extraneurologic manifestations, including osteopenia with poor bone healing and a high risk of gastrointestinal bleeding and portal hypertension caused by vasculature ectasias in the stomach, small intestine, and liver. Some individuals also have hair, skin, and nail changes, as well as anemia and thrombocytopenia.

EXPRESSÃO TECIDUAL(Ubíquo)
Cerebelo
50.0 TPM
Cérebro - Hemisfério cerebelar
43.2 TPM
Baço
39.7 TPM
Cervix Endocervix
30.6 TPM
Útero
30.5 TPM
OUTRAS DOENÇAS (3)
cerebroretinal microangiopathy with calcifications and cysts 1dyskeratosis congenitaCoats plus syndrome
HGNC:26169UniProt:Q2NKJ3
NPM1NucleophosminDisease-causing germline mutation(s) (loss of function) inAltamente restrito
FUNÇÃO

Involved in diverse cellular processes such as ribosome biogenesis, centrosome duplication, protein chaperoning, histone assembly, cell proliferation, and regulation of tumor suppressors p53/TP53 and ARF. Binds ribosome presumably to drive ribosome nuclear export. Associated with nucleolar ribonucleoprotein structures and bind single-stranded nucleic acids. Acts as a chaperonin for the core histones H3, H2B and H4. Stimulates APEX1 endonuclease activity on apurinic/apyrimidinic (AP) double-stran

LOCALIZAÇÃO

Nucleus, nucleolusNucleus, nucleoplasmCytoplasm, cytoskeleton, microtubule organizing center, centrosome

VIAS BIOLÓGICAS (4)
PKR-mediated signalingTP53 regulates transcription of additional cell cycle genes whose exact role in the p53 pathway remain uncertainNuclear import of Rev proteinNuclear events stimulated by ALK signaling in cancer
EXPRESSÃO TECIDUAL(Ubíquo)
Linfócitos
1517.5 TPM
Fibroblastos
1222.0 TPM
Ovário
1031.3 TPM
Cervix Endocervix
640.5 TPM
Útero
622.0 TPM
OUTRAS DOENÇAS (8)
acute myeloid leukemiaacute promyelocytic leukemiaacute myeloid leukemia with NPM1 somatic mutationsdyskeratosis congenita
HGNC:7910UniProt:P06748
RTEL1Regulator of telomere elongation helicase 1Disease-causing germline mutation(s) inTolerante
FUNÇÃO

A probable ATP-dependent DNA helicase implicated in telomere-length regulation, DNA repair and the maintenance of genomic stability. Acts as an anti-recombinase to counteract toxic recombination and limit crossover during meiosis. Regulates meiotic recombination and crossover homeostasis by physically dissociating strand invasion events and thereby promotes noncrossover repair by meiotic synthesis dependent strand annealing (SDSA) as well as disassembly of D loop recombination intermediates. Als

LOCALIZAÇÃO

Nucleus

VIAS BIOLÓGICAS (1)
Cytosolic iron-sulfur cluster assembly
MECANISMO DE DOENÇA

Dyskeratosis congenita, autosomal recessive, 5

A form of dyskeratosis congenita, a rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy. DKCB5 is characterized by onset of bone marrow failure and immunodeficiency in early childhood. Most patients also have growth and developmental delay and cerebellar hypoplasia, consistent with a clinical diagnosis of Hoyeraal-Hreidarsson syndrome.

EXPRESSÃO TECIDUAL(Ubíquo)
Cerebelo
61.0 TPM
Pituitária
57.3 TPM
Cérebro - Hemisfério cerebelar
51.0 TPM
Útero
40.6 TPM
Tireoide
35.6 TPM
OUTRAS DOENÇAS (5)
pulmonary fibrosis and/or bone marrow failure, Telomere-related, 3dyskeratosis congenita, autosomal recessive 5dyskeratosis congenitaidiopathic pulmonary fibrosis
HGNC:15888UniProt:Q9NZ71
DKC1H/ACA ribonucleoprotein complex subunit DKC1Disease-causing germline mutation(s) inAltamente restrito
FUNÇÃO

Catalytic subunit of H/ACA small nucleolar ribonucleoprotein (H/ACA snoRNP) complex, which catalyzes pseudouridylation of rRNA (PubMed:25219674, PubMed:32554502). This involves the isomerization of uridine such that the ribose is subsequently attached to C5, instead of the normal N1 (PubMed:25219674). Each rRNA can contain up to 100 pseudouridine ('psi') residues, which may serve to stabilize the conformation of rRNAs. Required for ribosome biogenesis and telomere maintenance (PubMed:19179534, P

LOCALIZAÇÃO

Nucleus, nucleolusNucleus, Cajal bodyCytoplasm

VIAS BIOLÓGICAS (2)
Telomere Extension By TelomeraserRNA modification in the nucleus and cytosol
MECANISMO DE DOENÇA

Dyskeratosis congenita, X-linked

A rare, progressive bone marrow failure syndrome characterized by the triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy.

EXPRESSÃO TECIDUAL(Ubíquo)
Linfócitos
106.0 TPM
Fibroblastos
67.6 TPM
Ovário
44.5 TPM
Esôfago - Mucosa
44.2 TPM
Testículo
44.1 TPM
OUTRAS DOENÇAS (4)
dyskeratosis congenita, X-linkedcataracts, hearing impairment, nephrotic syndrome, and enterocolitis 1dyskeratosis congenitaHoyeraal-Hreidarsson syndrome
HGNC:2890UniProt:O60832
PARNPoly(A)-specific ribonuclease PARNDisease-causing germline mutation(s) inTolerante
FUNÇÃO

3'-exoribonuclease that has a preference for poly(A) tails of mRNAs, thereby efficiently degrading poly(A) tails. Exonucleolytic degradation of the poly(A) tail is often the first step in the decay of eukaryotic mRNAs and is also used to silence certain maternal mRNAs translationally during oocyte maturation and early embryonic development. Interacts with both the 3'-end poly(A) tail and the 5'-end cap structure during degradation, the interaction with the cap structure being required for an eff

LOCALIZAÇÃO

NucleusCytoplasmNucleus, nucleolus

VIAS BIOLÓGICAS (3)
Deadenylation of mRNAATF4 activates genes in response to endoplasmic reticulum stressKSRP (KHSRP) binds and destabilizes mRNA
MECANISMO DE DOENÇA

Dyskeratosis congenita, autosomal recessive, 6

A form of dyskeratosis congenita, a rare multisystem disorder caused by defective telomere maintenance. It is characterized by progressive bone marrow failure, and the clinical triad of reticulated skin hyperpigmentation, nail dystrophy, and mucosal leukoplakia. Common but variable features include premature graying, aplastic anemia, low platelets, osteoporosis, pulmonary fibrosis, and liver fibrosis among others. Early mortality is often associated with bone marrow failure, infections, fatal pulmonary complications, or malignancy.

EXPRESSÃO TECIDUAL(Ubíquo)
Linfócitos
39.8 TPM
Testículo
34.1 TPM
Tireoide
33.5 TPM
Artéria tibial
33.3 TPM
Útero
33.0 TPM
OUTRAS DOENÇAS (5)
pulmonary fibrosis and/or bone marrow failure, Telomere-related, 4dyskeratosis congenita, autosomal recessive 6dyskeratosis congenitaidiopathic pulmonary fibrosis
HGNC:8609UniProt:O95453
TERTTelomerase reverse transcriptaseDisease-causing germline mutation(s) inRestrito
FUNÇÃO

Telomerase is a ribonucleoprotein enzyme essential for the replication of chromosome termini in most eukaryotes. Active in progenitor and cancer cells. Inactive, or very low activity, in normal somatic cells. Catalytic component of the teleromerase holoenzyme complex whose main activity is the elongation of telomeres by acting as a reverse transcriptase that adds simple sequence repeats to chromosome ends by copying a template sequence within the RNA component of the enzyme. Catalyzes the RNA-de

LOCALIZAÇÃO

Nucleus, nucleolusNucleus, nucleoplasmNucleusChromosome, telomereCytoplasmNucleus, PML body

VIAS BIOLÓGICAS (3)
Telomere Extension By TelomeraseFormation of the beta-catenin:TCF transactivating complexRegulation of MITF-M-dependent genes involved in DNA replication, damage repair and senescence
EXPRESSÃO TECIDUAL(Baixa expressão)
Testículo
2.7 TPM
Intestino delgado
0.7 TPM
Brain Caudate basal ganglia
0.6 TPM
Cólon transverso
0.5 TPM
Brain Nucleus accumbens basal ganglia
0.5 TPM
OUTRAS DOENÇAS (13)
dyskeratosis congenita, autosomal dominant 2pulmonary fibrosis and/or bone marrow failure, Telomere-related, 1adrenal cortex carcinomaclear cell sarcoma of kidney
HGNC:11730UniProt:O14746

Variantes genéticas (ClinVar)

197 variantes patogênicas registradas no ClinVar.

🧬 TINF2: NM_001099274.3(TINF2):c.399+60T>G ()
🧬 TINF2: NM_001099274.3(TINF2):c.1221+2T>G ()
🧬 TINF2: NM_001099274.3(TINF2):c.1222-6C>G ()
🧬 TINF2: NM_001099274.3(TINF2):c.1006G>T (p.Gly336Trp) ()
🧬 TINF2: NM_001099274.3(TINF2):c.399+1G>A ()
Ver todas no ClinVar

Classificação de variantes (ClinVar)

Distribuição de 12,239 variantes classificadas pelo ClinVar.

7343
4896
VUS (60.0%)
Benigna (40.0%)
VARIANTES MAIS SIGNIFICATIVAS
TERT: NM_198253.3(TERT):c.922C>T (p.Pro308Ser) [Uncertain significance]
LOC110806306: NR_001566.3(TERC):n.246G>C [Uncertain significance]
LOC110806306: NR_001566.3(TERC):n.334G>C [Uncertain significance]
CTC1: NM_025099.6(CTC1):c.1936C>A (p.Arg646=) [Uncertain significance]
CTC1: NM_025099.6(CTC1):c.2669+19C>G [Uncertain significance]

Vias biológicas (Reactome)

37 vias biológicas associadas aos genes desta condição.

Recognition and association of DNA glycosylase with site containing an affected pyrimidine Cleavage of the damaged pyrimidine Recognition and association of DNA glycosylase with site containing an affected purine Cleavage of the damaged purine Meiotic synapsis Packaging Of Telomere Ends Telomere Extension By Telomerase Polymerase switching on the C-strand of the telomere Processive synthesis on the C-strand of the telomere Telomere C-strand (Lagging Strand) Synthesis Telomere C-strand synthesis initiation Removal of the Flap Intermediate from the C-strand DNA Damage/Telomere Stress Induced Senescence Inhibition of DNA recombination at telomere Fanconi Anemia Pathway rRNA modification in the nucleus and cytosol Interconversion of nucleotide di- and triphosphates G1/S-Specific Transcription Telomere Maintenance Association of TriC/CCT with target proteins during biosynthesis Nuclear import of Rev protein SUMOylation of transcription cofactors Deposition of new CENPA-containing nucleosomes at the centromere TP53 regulates transcription of additional cell cycle genes whose exact role in the p53 pathway remain uncertain TFAP2A acts as a transcriptional repressor during retinoic acid induced cell differentiation SARS-CoV-1-host interactions ALK mutants bind TKIs Signaling by ALK fusions and activated point mutants Nuclear events stimulated by ALK signaling in cancer PKR-mediated signaling Cytosolic iron-sulfur cluster assembly Resolution of D-loop Structures through Synthesis-Dependent Strand Annealing (SDSA) ATF4 activates genes in response to endoplasmic reticulum stress Deadenylation of mRNA KSRP (KHSRP) binds and destabilizes mRNA Formation of the beta-catenin:TCF transactivating complex Regulation of MITF-M-dependent genes involved in DNA replication, damage repair and senescence

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Centros de Referência SUS

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Centros para Disqueratose congênita

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Hospital Universitário Prof. Edgard Santos (HUPES)

R. Dr. Augusto Viana, s/n - Canela, Salvador - BA, 40110-060 · CNES 0003808

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Anomalias CongênitasErros Inatos do Metabolismo

Hospital Infantil Albert Sabin

R. Tertuliano Sales, 544 - Vila União, Fortaleza - CE, 60410-794 · CNES 2407876

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Hospital de Apoio de Brasília (HAB)

AENW 3 Lote A Setor Noroeste - Plano Piloto, Brasília - DF, 70684-831 · CNES 0010456

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Hospital Estadual Infantil e Maternidade Alzir Bernardino Alves (HIABA)

Av. Min. Salgado Filho, 918 - Soteco, Vila Velha - ES, 29106-010 · CNES 6631207

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Hospital das Clínicas da UFG

Rua 235 QD. 68 Lote Área, Nº 285, s/nº - Setor Leste Universitário, Goiânia - GO, 74605-050 · CNES 2338424

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Hospital Universitário da UFJF

R. Catulo Breviglieri, Bairro - s/n - Santa Catarina, Juiz de Fora - MG, 36036-110 · CNES 2297442

Atenção Especializada

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Hospital das Clínicas da UFMG

Av. Prof. Alfredo Balena, 110 - Santa Efigênia, Belo Horizonte - MG, 30130-100 · CNES 2280167

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Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital Universitário Julio Müller (HUJM)

R. Luis Philippe Pereira Leite, s/n - Alvorada, Cuiabá - MT, 78048-902 · CNES 2726092

Atenção Especializada

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Anomalias Congênitas

Hospital Universitário João de Barros Barreto

R. dos Mundurucus, 4487 - Guamá, Belém - PA, 66073-000 · CNES 2337878

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Hospital Universitário Lauro Wanderley (HULW)

R. Tabeliao Estanislau Eloy, 585 - Castelo Branco, João Pessoa - PB, 58050-585 · CNES 0002470

Atenção Especializada

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Anomalias Congênitas

Instituto de Medicina Integral Prof. Fernando Figueira (IMIP)

R. dos Coelhos, 300 - Boa Vista, Recife - PE, 50070-902 · CNES 0000647

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Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital Pequeno Príncipe

R. Des. Motta, 1070 - Água Verde, Curitiba - PR, 80250-060 · CNES 3143805

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Hospital Universitário Regional de Maringá (HUM)

Av. Mandacaru, 1590 - Parque das Laranjeiras, Maringá - PR, 87083-240 · CNES 2216108

Atenção Especializada

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Hospital de Clínicas da UFPR

R. Gen. Carneiro, 181 - Alto da Glória, Curitiba - PR, 80060-900 · CNES 2364980

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Hospital Universitário Pedro Ernesto (HUPE-UERJ)

Blvd. 28 de Setembro, 77 - Vila Isabel, Rio de Janeiro - RJ, 20551-030 · CNES 2280221

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Instituto Nacional de Saúde da Mulher, da Criança e do Adolescente Fernandes Figueira (IFF/Fiocruz)

Av. Rui Barbosa, 716 - Flamengo, Rio de Janeiro - RJ, 22250-020 · CNES 2269988

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Hospital São Lucas da PUCRS

Av. Ipiranga, 6690 - Jardim Botânico, Porto Alegre - RS, 90610-000 · CNES 2232928

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Anomalias CongênitasErros Inatos do Metabolismo

Hospital de Clínicas de Porto Alegre (HCPA)

Rua Ramiro Barcelos, 2350 Bloco A - Av. Protásio Alves, 211 - Bloco B e C - Santa Cecília, Porto Alegre - RS, 90035-903 · CNES 2237601

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Hospital Universitário da UFSC (HU-UFSC)

R. Profa. Maria Flora Pausewang - Trindade, Florianópolis - SC, 88036-800 · CNES 2560356

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Hospital das Clínicas da FMUSP

R. Dr. Ovídio Pires de Campos, 225 - Cerqueira César, São Paulo - SP, 05403-010 · CNES 2077485

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Hospital de Base de São José do Rio Preto

Av. Brg. Faria Lima, 5544 - Vila Sao Jose, São José do Rio Preto - SP, 15090-000 · CNES 2079798

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Hospital de Clínicas da UNICAMP

R. Vital Brasil, 251 - Cidade Universitária, Campinas - SP, 13083-888 · CNES 2748223

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Hospital de Clínicas de Ribeirão Preto (HCRP-USP)

R. Ten. Catão Roxo, 3900 - Vila Monte Alegre, Ribeirão Preto - SP, 14015-010 · CNES 2082187

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UNIFESP / Hospital São Paulo

R. Napoleão de Barros, 715 - Vila Clementino, São Paulo - SP, 04024-002 · CNES 2688689

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Publicações mais relevantes

Timeline de publicações
539 papers (10 anos)
#1

Monoallelic PARN mutation presenting as pancytopenia, hepatic fibrosis and idiopathic pulmonary fibrosis.

BMJ case reports2026 Feb 25

A woman in her late 20s presented with a 5-year history of progressive fatigue and generalised weakness. Examination revealed signs of premature ageing, anaemia, neuropathy and hepatosplenomegaly.Investigations showed pancytopenia, dimorphic anaemia, severe vitamin B12 deficiency, hepatic fibrosis and pulmonary fibrosis. Despite correction of nutritional deficiencies, the constellation of cytopenia, premature greying, osteoporosis, hepatic and pulmonary fibrosis raised suspicion of a genetic disorder. Clinical exome sequencing identified a monoallelic PARN missense variant (c.613T>C; p.Cys205Arg), classified as a Variant of Uncertain Significance. Germline pathogenic variants in PARN have been associated with dyskeratosis congenita, autosomal recessive 6 (DKCB6) and with telomere-related pulmonary fibrosis and/or bone marrow failure 4. The clinical-genetic correlation in this case supported a diagnosis of a telomere biology disorder (TBD). This case highlights the importance of considering TBDs in young adults with unexplained multisystem disease, even when classical mucocutaneous features are absent. Early recognition is crucial for guiding genetic counselling, surveillance and consideration of bone marrow transplantation in progressive cases.

#2

RNA Analysis Uncovers Pathogenic PARN Variant in Dyskeratosis Congenita.

Clinical genetics2026 Feb

Dyskeratosis congenita (DC) is a rare genetic disorder caused by impaired telomere maintenance, leading to diverse clinical manifestations, including bone marrow failure, mucocutaneous abnormalities, and multi-organ dysfunction. Here, we report a 14-year-old male patient presenting with microcephaly, developmental delay, synostoses, cerebellar hypoplasia with ataxia, and an immunodeficiency condition, but lacking classical DC features such as nail dystrophy and skin hyperpigmentation. WGS revealed two variants in the PARN gene: a known pathogenic missense variant (c.1045C > T, p.Arg349Trp) and a novel intronic variant (c.178-28T > C). Functional RNA analysis demonstrated that the intronic variant disrupts the branch point sequence, leading to exon 4 skipping and nonsense-mediated decay (NMD) of a significant proportion of transcripts. This study confirms the pathogenicity of the intronic variant and underscores the importance of functional validation in interpreting noncoding variants, particularly in genetically heterogeneous disorders like DC. Our findings expand the molecular and phenotypic spectrum of PARN-related DC and highlight the utility of WGS reanalysis and RNA studies in resolving diagnostically challenging cases.

#3

Avascular Necrosis and Minimal Trauma Fractures in Telomere Biology Disorders.

Clinical genetics2026 Feb

Avascular necrosis (AVN) and minimal trauma fractures (MTF) cause significant morbidity in patients with telomere biology disorders (TBDs). TBDs are associated with very high risks of bone marrow failure, pulmonary fibrosis, cancer, and many other complications due to pathogenic germline variants in genes essential for telomere function and maintenance. To understand the extent to which AVN and MTF occur in TBDs and identify areas requiring more research in the role of telomeres in bone biology. We assessed the occurrence of AVN and MTF in 233 patients with TBDs. An age, gender, and gene-matched TBD patient control group was used to assess associations between AVN/MTF and clinical characteristics. Forty-two (18%) patients with TBD developed at least one AVN and/or MTF event with 19 patients experiencing their first event in childhood. AVN and MTF were most common in patients with autosomal or X-linked recessive, or heterozygous TINF2 disease (19/36 AVN and 17/19 MTF). Androgen and corticosteroid use were more common in patients with AVN compared with matched patient controls (41.2% vs. 16.3%, p < 0.05 and 41.2% vs. 14%, p < 0.01, respectively); however, 57.1% of patients experienced AVN and/or MTF events in the absence of androgen or corticosteroid use. Severe bone marrow failure and hematopoietic cell transplantation history were significantly more common in MTF patients than in controls (44.2% and 30.2% respectively, p < 0.05). There were no statistically significant associations between low bone mineral density or vitamin D deficiency and AVN or MTF. AVN and MTFs are common, debilitating complications in TBDs and frequently occur independently of androgen or corticosteroid use. Our results underscore the need for disease-specific translational studies as well as improved prevention and therapeutic options for patients with TBDs. Trial Registration: ClinicalTrials.gov identifier: NCT00027274.

#4

Dyskerin dysfunction in cancer development: from telomere dysregulation to immune deficiency.

American journal of cancer research2026

Dyskerin, encoded by the dyskerin pseudouridine synthase 1 (DKC1) gene, is a core component of the H/ACA ribonucleoprotein complex and plays essential roles in telomerase activity maintenance, rRNA pseudouridylation, and ribosome biogenesis. Loss of DKC1 function represents a major pathogenic basis of dyskeratosis congenita (DC) and is associated with a markedly increased risk of malignancy, particularly head and neck squamous cell carcinoma and oral squamous cell carcinoma. Traditionally, cancer susceptibility in DC has been largely attributed to telomere shortening and the resulting genomic instability; however, this explanation does not fully account for the heterogeneity observed across different genetic subtypes and clinical phenotypes. In this review, we systematically integrate three key mechanisms through which dyskerin dysfunction contributes to DC-associated carcinogenesis: disruption of telomere homeostasis, defects in selective translation regulation dependent on RNA pseudouridylation, and progressive impairment of T-cell-mediated immune surveillance. We highlight how DKC1 deficiency leads to insufficient rRNA pseudouridylation, selectively affecting the translation of internal ribosome entry site (IRES)-dependent transcripts, thereby attenuating the stress-induced expression of critical tumor suppressor proteins. In parallel, evidence from patient cohort studies is discussed to support a potentially dominant role of immunodeficiency in tumor development. Finally, we propose that future studies on DC and short telomere syndromes should emphasize genetic stratification and long-term clinical outcomes to refine cancer risk assessment and optimize preventive and therapeutic strategies.

#5

Beyond iron deficiency: A comprehensive national survey of anaemia etiology in Sri Lankan young adults.

Scientific reports2026 Mar 19

Previous studies in Sri Lanka have explored the prevalence and causes of anaemia, mainly emphasizing iron deficiency while overlooking other important factors such as enzymopathies, membranopathies, and haemoglobinopathies. Moreover, many studies reported unexplained cases of anaemia even after detailed evaluation. This study aimed to determine the prevalence and underlying causes of anaemia among community-based young adults in Sri Lanka. A descriptive cross-sectional study was conducted from January 2023 to December 2024 among young adults aged 18-30 years. Data and blood samples were collected using a pretested questionnaire. Anaemic individuals underwent serum iron studies, CRP, vitamin B12 and folate assays, and thalassaemia screening. Those with uncharacterized anaemia were further assessed using red cell enzyme assays, EMA dye binding assays, and whole-exome sequencing. The mean (± SD) age of the study participants was 23.90 ± 1.98 years. Anaemia prevalence was 15.0%. The main causes were iron deficiency (49.3%), folate deficiency (27.8%), vitamin B12 deficiency (14.4%), and haemoglobinopathy traits were identified in 25.6% of anaemic individuals, frequently in combination with nutritional deficiencies. A substantial proportion of anaemic individuals exhibited coexisting aetiologies rather than a single cause. Initially, 17.4% remained uncharacterized, but advanced testing identified variants suggestive of hereditary spherocytosis and congenital dyserythropoietic anaemia or dyskeratosis congenita. This first community-based Sri Lankan study integrating advanced diagnostics revealed that, although iron deficiency predominates, genetic and enzymatic disorders contribute notably, highlighting the need for broader diagnostic strategies in anaemia screening in the community.

Publicações recentes

Ver todas no PubMed

📚 EuropePMC699 artigos no totalmostrando 195

2026

Dyskerin dysfunction in cancer development: from telomere dysregulation to immune deficiency.

American journal of cancer research
2026

Beyond iron deficiency: A comprehensive national survey of anaemia etiology in Sri Lankan young adults.

Scientific reports
2026

Dyskeratosis Congenita: Clinical Phenotype and Genetic Features in a Sibling Pair.

Clinical, cosmetic and investigational dermatology
2026

Monoallelic PARN mutation presenting as pancytopenia, hepatic fibrosis and idiopathic pulmonary fibrosis.

BMJ case reports
2026

Dyskeratosis Congenita Due to a de novo TINF2 Mutation.

QJM : monthly journal of the Association of Physicians
2026

Cirrhosis of Liver in Patients With Dyskeratosis Congenita: A Report of Two Cases.

Clinical case reports
2026

Exosome-mediated decay of unstable long extended precursors of human telomerase RNA is dependent on 5'-cap trimethylation.

Genes &amp; development
2026

A rare case of dyskeratosis congenita with DKC1 mutation presenting initially as thrombocytopenia: Case report.

Medicine
2026

Oral squamous cell carcinoma risk and magnitude of association in inherited cancer predisposition syndromes: evidence from a large real-world cohort.

Oral surgery, oral medicine, oral pathology and oral radiology
2025

[Clinical and genetic analysis of a child with X-linked Hoyeraal-Hreidarsson syndrome due to variant of DKC1 gene and a literature review].

Zhonghua yi xue yi chuan xue za zhi = Zhonghua yixue yichuanxue zazhi = Chinese journal of medical genetics
2025

Recent progress in human telomerase structure and its therapeutic targeting.

Frontiers in molecular biosciences
2025

Interprotomer communication and functional asymmetry in H/ACA snoRNPs.

Proceedings of the National Academy of Sciences of the United States of America
2025

Case Report of Hair Abnormalities in Dyskeratosis Congenita: A Trichoscopic and Microscopic Analysis.

Skin appendage disorders
2025

Fueling for the finish line: Control of human telomerase activity by nucleotide metabolism.

DNA repair
2026

Case Series: Clinical Significance of Heterozygous Pathogenic RTEL1 Variants Identified via Routine Clinical Genetic Diagnostics.

American journal of medical genetics. Part A
2025

[Clinical and genetic characteristics of 6 cases of congenital dyskeratosis in children].

Zhonghua er ke za zhi = Chinese journal of pediatrics
2025

Segmental dyskeratosis congenita - A diagnostic challenge.

Anais brasileiros de dermatologia
2025

Beyond Hematologic Malignancies: Colorectal Cancer as a Solid Tumor Manifestation of Inherited Bone Marrow Failure Syndromes.

International journal of molecular sciences
2025

Ending diagnostic odyssey by reanalysis of whole exome sequencing data: reclassification of suspected Fanconi anemia cases to dyskeratosis congenita and Diamond-Blackfan anemia.

Orphanet journal of rare diseases
2025

Insights in bone marrow failure syndromes: take home messages from the 3rd ESH-EBMT-EHA-IPIG translational research conference.

Bone marrow transplantation
2025

Optical Genomic Mapping and Next-Generation Sequencing Identified Retrotransposon Insertion and Missense Variant Disrupting PARN Gene in Dyskeratosis Congenita.

Human mutation
2026

RNA Analysis Uncovers Pathogenic PARN Variant in Dyskeratosis Congenita.

Clinical genetics
2025

Telomerase activity in T-cells as a functional test for pathogenicity assessment of novel genetic variants in telomere biology disorders.

Scientific reports
2026

Avascular Necrosis and Minimal Trauma Fractures in Telomere Biology Disorders.

Clinical genetics
2025

Targeting DKC1/NF-κB axis suppresses tumorigenesis and enhances 5-FU sensitivity in gastric cancer.

Cancer cell international
2025

Severe Treatment-Related Toxicity in Oral Cavity Squamous Cell Carcinoma with Dyskeratosis Congenita: A Case Report and Critical Review of Radiation-Induced Complications.

Advances in radiation oncology
2025

The role of telomeres in leukemic stem cells function.

Regenerative therapy
2025

Identification and Computational Analysis of a Novel Pathogenic DKC1 Variant Underlying X-Linked Dyskeratosis Congenita.

Biochemical genetics
2025

[Clinical Analysis of Dyskeratosis Congenita in Children].

Zhongguo shi yan xue ye xue za zhi
2025

Intronic hexanucleotide repeat expansion in TYMS in monozygotic twins with congenital progressive universal melanosis.

Biomedical reports
2025

Multidisciplinary Approach to Familial Pulmonary Fibrosis.

Radiographics : a review publication of the Radiological Society of North America, Inc
2025

Bilateral, recurrent Cytomegalovirus retinitis in Dyskeratosis congenita healing with extensive dystrophic calcification.

International ophthalmology
2025

Separation of telomere protection from length regulation by two different point mutations at amino acid 492 of RTEL1.

Nucleic acids research
2025

[Dyskeratosis congenita combined with myeloproliferative disorder and trilineage cytopenia].

Zhonghua jie he he hu xi za zhi = Zhonghua jiehe he huxi zazhi = Chinese journal of tuberculosis and respiratory diseases
2025

Investigating telomere length in progeroid syndromes: implications for aging disorders.

Aging
2025

Germline PARN Variants in Telomere Biology Disorders and Challenges in Variant Curation.

Molecular genetics &amp; genomic medicine
2025

Analysis of Late Complications Associated With Hematopoietic Stem Cell Transplantation in Patients With Dyskeratosis Congenita.

Pediatric blood &amp; cancer
2025

Siblings with a Homozygous Variant in the NHP2 Gene: A Case Report and Review of Literature.

Molecular syndromology
2025

De Novo Splice-Site Variant in DKC1 in a Female With Clinical Features of Hoyeraal-Hreidarsson Syndrome.

American journal of medical genetics. Part A
2025

Extensive and persistent tongue ulceration is an early character of dyskeratosis congenita.

Orphanet journal of rare diseases
2026

Structural Biology of Telomerase and Associated Factors.

Cold Spring Harbor perspectives in biology
2025

Loss of Ten1 in mice induces telomere shortening and models human dyskeratosis congenita.

Science advances
2025

TYMS-ENOSF1 Dyskeratosis Congenita in a Patient With Ring Chromosome 18: A Case Report.

American journal of medical genetics. Part A
2025

Familial pulmonary fibrosis with dyskeratosis congenita associated with a rare RTEL1 gene mutation.

BMJ case reports
2025

Dyskeratosis Congenita Complicated by Pulmonary Fibrosis and Myelodysplastic Syndrome with a Germline Mutation of the DKC1 Gene and a Somatic Mutation of the U2AF1 Gene in Leukocytes.

Internal medicine (Tokyo, Japan)
2025

Expanding the clinical spectrum of NHP2 -related dyskeratosis congenita: a case with novel phenotypic features.

Clinical dysmorphology
2025

Linear hypermelanosis in narrow bands: a mosaic pattern manifestation of dyskeratosis congenita?

The British journal of dermatology
2025

Haploidentical Hematopoietic Cell Transplantation in Dyskeratosis Congenita with Myelodysplastic Syndrome/Acute Myeloid Leukemia.

Blood cell therapy
2025

Genetic variants in NHEJ1 and related DNA repair disorders: insights into phenotypic heterogeneity and links to hypoplastic myelodysplastic syndromes and familial hematological malignancies susceptibility.

Annals of hematology
2025

Clinical Use of ZSCAN4 for Telomere Elongation in Hematopoietic Stem Cells.

NEJM evidence
2025

Premature ageing of lung alveoli and bone marrow cells from Terc deficient mice with different telomere lengths.

Scientific reports
2025

Phenotype puzzle: the role of novel LMBRD1 gene variant in Cbl deficiency causing Dyskeratosis Congenita-like clinical manifestations.

Journal of human genetics
2025

Late-onset telomere biology disorders in adults: clinical insights and treatment outcomes from a retrospective registry cohort.

Blood advances
2025

Dyskeratosis congenita mistaken for lichen planus.

Archives of disease in childhood
2025

TERT de novo mutation-associated dyskeratosis congenita and porto-sinusoidal vascular disease: a case report.

Journal of medical case reports
2024

[Next generation sequencing in pediatric bone marrow failure: a valuable tool for accurate diagnosis].

Andes pediatrica : revista Chilena de pediatria
2024

Bilateral cytomegalovirus retinitis in a patient with dyskeratosis congenita.

International journal of ophthalmology
2024

"Crying in the Wilderness"-The Use of Web-Based Support in Telomere Biology Disorders: Thematic Analysis.

JMIR formative research
2024

Hoyeraal-Hreidarsson syndrome: a case report of dyskeratosis congenita with a novel PARN gene mutation.

Annals of medicine and surgery (2012)
2025

Telomere function and regulation from mouse models to human ageing and disease.

Nature reviews. Molecular cell biology
2024

A "rotating menu" of medical uncertainty for families affected by telomere biology disorders: A qualitative interview study.

SSM. Qualitative research in health
2025

Inducible pluripotent stem cell models to study bone marrow failure and MDS predisposition syndromes.

Experimental hematology
2024

Inherited Telomere Biology Disorders: Pathophysiology, Clinical Presentation, Diagnostics, and Treatment.

Transfusion medicine and hemotherapy : offizielles Organ der Deutschen Gesellschaft fur Transfusionsmedizin und Immunhamatologie
2024

Telomeres and immunodeficiencies.

Human immunology
2025

An Atypical Presentation of Dyskeratosis Congenita in a Child With a Familial RTEL1 Mutation.

Pediatric dermatology
2025

Germline RTEL1 Variants in Telomere Biology Disorders.

American journal of medical genetics. Part A
2024

A comprehensive in silico investigation into the pathogenic SNPs in the RTEL1 gene and their biological consequences.

PloS one
2024

Locally advanced rectal cancer in a young adult affected with dyskeratosis congenita (Zinsser-Cole-Engman syndrome): a case report.

Surgical case reports
2025

Reticulated pigmentary changes and Terry's nails in a patient with a TERT variant-associated telomere biology disorder.

Pediatric dermatology
2024

The gray boundaries of aberrant shortening of the cellular timekeepers' edges.

EMBO molecular medicine
2024

The evolving genetic landscape of telomere biology disorder dyskeratosis congenita.

EMBO molecular medicine
2024

Telomere biology disorders: from dyskeratosis congenita and beyond.

Postgraduate medical journal
2024

Presumptive Cytomegalovirus Retinitis as a Complication of Dyskeratosis Congenita: A Case Report.

Case reports in ophthalmology
2024

An algorithmic approach towards diagnosis of patients with hereditary reticulate pigmentary disorders: a narrative review.

Clinical and experimental dermatology
2024

Mucosal Cancers Arising in Potentially Malignant Lesions of the Oral Mucosa Are Marjolin Ulcers: New Insights Into Old Concepts.

Dermatology practical &amp; conceptual
2024

Nucleotide sugars correlate with leukocyte telomere length as part of a dyskeratosis congenita metabolomic plasma signature.

Haematologica
2024

A case of dermatopathia pigmentosa reticularis masquerading as dyskeratosis congenita: the importance of nailing the correct diagnosis.

Clinical and experimental dermatology
2024

Diseases with oral malignant potential: Need for change to inform research, policy, and practice.

Journal of oral pathology &amp; medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
2024

A Novel Autosomal Recessive Candidate Gene Responsible for RASopathy-Like Phenotype and Bone Marrow Failure: RASA3.

Journal of pediatric genetics
2024

Bilateral retinal vasculopathy in a patient with aplastic anemia due to dyskeratosis congenita.

Journal francais d'ophtalmologie
2024

MASCC/ISOO Clinical Practice Statement: The risk of secondary oral cancer following hematopoietic cell transplantation.

Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer
2024

Reduced anti-Müllerian hormone levels in males with inherited bone marrow failure syndromes.

Endocrine connections
2025

Dyskeratosis congenita with squamous cell carcinoma of the tongue: A rare case report.

Indian journal of pathology &amp; microbiology
2024

Allogeneic Hematopoietic Cell Transplant For Bone Marrow Failure or Myelodysplastic Syndrome in Dyskeratosis Congenita/Telomere Biology Disorders: Single-Center, Single-Arm, Open-Label Trial of Reduced-Intensity Conditioning Without Radiation.

Transplantation and cellular therapy
2024

Dyskeratosis congenita associated with a novel missense variant in TERT: Approach for the dermatologists.

Archives of dermatological research
2024

Congenital nail abnormalities.

Hand surgery &amp; rehabilitation
2024

Germline thymidylate synthase deficiency impacts nucleotide metabolism and causes dyskeratosis congenita.

American journal of human genetics
2024

Overexpression of human SAMD9 inhibits protein translation and alters MYC signaling resulting in cell cycle arrest.

Experimental hematology
2024

Skin cancer-associated genodermatoses in skin of color patients: a review.

Archives of dermatological research
2024

Dyskeratosis congenita: a rare case report.

Oxford medical case reports
2024

Integrated proteogenomic analysis for inherited bone marrow failure syndrome.

Leukemia
2024

Cancer Precursor Syndromes and Their Detection in the Head and Neck.

Hematology/oncology clinics of North America
2024

A naturally occurring canine model of syndromic congenital microphthalmia.

G3 (Bethesda, Md.)
2024

Clinical mutations in the TERT and TERC genes coding for telomerase components induced oxidative stress, DNA damage at telomeres and cell apoptosis besides decreased telomerase activity.

Human molecular genetics
2024

A Review of the Repair of DNA Double Strand Breaks in the Development of Oral Cancer.

International journal of molecular sciences
2024

Multiple-Digit Pigmented Bowen's Disease Induced by Human Papillomavirus in an Immunocompetent Child.

Skin appendage disorders
2024

X-linked Dyskeratosis Congenita Case with Mutation 1058C>T(p.Ala353Val) in Dyskerine Gene.

Indian journal of dermatology
2025

Liver involvement in dyskeratosis congenita-Case series and concise literature review.

Indian journal of gastroenterology : official journal of the Indian Society of Gastroenterology
2024

Can telomeric changes orchestrate the development of autoinflammatory skin diseases?

Italian journal of dermatology and venereology
2024

A new variant in the ZCCHC8 gene: diverse clinical phenotypes and expression in the lung.

ERJ open research
2024

Linking Gene Fusions to Bone Marrow Failure and Malignant Transformation in Dyskeratosis Congenita.

International journal of molecular sciences
2023

Inherited bone marrow failure syndromes: phenotype as a tool for early diagnostic suspicion at a major reference center in Mexico.

Frontiers in genetics
2024

The Use of Social Media to Express and Manage Medical Uncertainty in Dyskeratosis Congenita: Content Analysis.

JMIR infodemiology
2024

Characterization of novel mutations in the TEL-patch domain of the telomeric factor TPP1 associated with telomere biology disorders.

Human molecular genetics
2023

Prevalence of Oral Submucous Fibrosis With Other Oral Potentially Malignant Disorders: A Clinical Retrospective Study.

Cureus
2024

Telomere length and cancer risk: finding Goldilocks.

Biogerontology
2023

A pan-cancer analysis of Dyskeratosis congenita 1 (DKC1) as a prognostic biomarker.

Hereditas
2023

Digital telomere measurement by long-read sequencing distinguishes healthy aging from disease.

bioRxiv : the preprint server for biology
2023

Minimal intensity conditioning strategies for bone marrow failure: is it time for "preventative" transplants?

Hematology. American Society of Hematology. Education Program
2023

Posttransplant complications in patients with marrow failure syndromes: are we improving long-term outcomes?

Hematology. American Society of Hematology. Education Program
2024

The metabolic basis of inherited neutropenias.

British journal of haematology
2023

Cytogenetics in the management of bone marrow failure syndromes: Guidelines from the Groupe Francophone de Cytogénétique Hématologique (GFCH).

Current research in translational medicine
2023

X-linked genodermatoses from diagnosis to tailored therapy.

La Clinica terapeutica
2023

Telomere biology disorders may manifest as common variable immunodeficiency (CVID).

Clinical immunology (Orlando, Fla.)
2023

Conformational plasticity and allosteric communication networks explain Shelterin protein TPP1 binding to human telomerase.

Communications chemistry
2024

A rare homozygous variant in TERT gene causing variable bone marrow failure, fragility fractures, rib anomalies and extremely short telomere lengths with high serum IgE.

British journal of haematology
2024

Severe Thrombocytopenia Due to Bone Marrow Failure in Children With Dyskeratosis Congenita Does Not Respond to Eltrombopag Treatment: Case Series.

Journal of pediatric hematology/oncology
2024

The C-terminal extension of dyskerin is a dyskeratosis congenita mutational hotspot that modulates interaction with telomerase RNA and subcellular localization.

Human molecular genetics
2024

Fyn-mediated phosphorylation of Menin disrupts telomere maintenance in stem cells.

bioRxiv : the preprint server for biology
2023

Germline landscape of RPA1, RPA2 and RPA3 variants in pediatric malignancies: identification of RPA1 as a novel cancer predisposition candidate gene.

Frontiers in oncology
2023

Compound heterozygous mutations in the helicase RTEL1 causing Hoyeraal-Hreidarsson syndrome with Blake`s pouch cyst: a case report.

The Turkish journal of pediatrics
2023

p53 in the Molecular Circuitry of Bone Marrow Failure Syndromes.

International journal of molecular sciences
2023

Genetic Counseling and Family Screening Recommendations in Patients with Telomere Biology Disorders.

Current hematologic malignancy reports
2023

New Insights into Dyskerin-CypA Interaction: Implications for X-Linked Dyskeratosis Congenita and Beyond.

Genes
2023

Telomerase RNA-based aptamers restore defective myelopoiesis in congenital neutropenic syndromes.

Nature communications
2023

CRISPR screen identifies CEBPB as contributor to dyskeratosis congenita fibroblast senescence via augmented inflammatory gene response.

G3 (Bethesda, Md.)
2023

A case report of dyskeratosis congenita caused by a novel TERC mutation.

Annals of hematology
2023

AC125611.3 promotes the progression of colon cancer by recruiting DKC1 to stabilize CTNNB1.

Arab journal of gastroenterology : the official publication of the Pan-Arab Association of Gastroenterology
2023

A systematic approach identifies p53-DREAM pathway target genes associated with blood or brain abnormalities.

Disease models &amp; mechanisms
2023

The Molecular and Genetic Mechanisms of Inherited Bone Marrow Failure Syndromes: The Role of Inflammatory Cytokines in Their Pathogenesis.

Biomolecules
2023

Dyskeratosis congenita: natural history of the disease through the study of a cohort of patients diagnosed in childhood.

Frontiers in pediatrics
2023

[Dyskeratosis congenita und Ösophagusstriktur bei einem Kleinkind].

Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG
2023

The PARN, TOE1, and USB1 RNA deadenylases and their roles in non-coding RNA regulation.

The Journal of biological chemistry
2023

Inherited bone marrow failure syndromes and germline predisposition to myeloid neoplasia: A practical approach for the pathologist.

Seminars in diagnostic pathology
2023

Control of protein synthesis through mRNA pseudouridylation by dyskerin.

Science advances
2023

Dyskeratosis Congenita: A Case Report of a Patient With Coronary Artery Disease.

Cureus
2023

Inherited Reticulate Pigmentary Disorders.

Genes
2023

The distribution and accumulation of the shortest telomeres in telomere biology disorders.

British journal of haematology
2023

Patient-Induced Pluripotent Stem Cell-Derived Hepatostellate Organoids Establish a Basis for Liver Pathologies in Telomeropathies.

Cellular and molecular gastroenterology and hepatology
2023

Hematopoietic cell transplantation for telomere biology diseases: A retrospective single-center cohort study.

European journal of haematology
2023

Spectrum of Liver Pathology in Dyskeratosis Congenita.

The American journal of surgical pathology
2023

Endoscopic Assessment and Serial Balloon Dilatation in a Toddler With Dyskeratosis Congenita-Hoyeraal-Hreidarsson Syndrome Following Bone Marrow Transplant: A Case Report.

JPGN reports
2023

What is the future of telomere length testing in telomere biology disorders?

Expert review of hematology
2023

Size matters in telomere biology disorders ‒ expanding phenotypic spectrum in patients with long or short telomeres.

Hereditary cancer in clinical practice
2023

Progression of liver disease and portal hypertension in dyskeratosis congenita and related telomere biology disorders.

Hepatology (Baltimore, Md.)
2022

A Primary Gastrointestinal Presentation and Novel Genetic Variant of Dyskeratosis Congenita in a Pediatric Patient.

JPGN reports
2023

Buildup from birth onward of short telomeres in human hematopoietic cells.

Aging cell
2023

Identification of Adult Patients With Classical Dyskeratosis Congenita or Cryptic Telomere Biology Disorder by Telomere Length Screening Using Age-modified Criteria.

HemaSphere
2023

A de novo TINF2, R282C Mutation in a Case of Dyskeratosis Congenital Founded by Next-Generation Sequencing.

Iranian biomedical journal
2023

Idiopathic non-cirrhotic portal hypertension in dyskeratosis congenita with rare variant of NHP2.

QJM : monthly journal of the Association of Physicians
2023

Case report: A novel mutation in RTEL1 gene in dyskeratosis congenita.

Frontiers in oncology
2023

Boosting NAD ameliorates hematopoietic impairment linked to short telomeres in vivo.

GeroScience
2023

Neonatal intestinal obstruction in Hoyeraal-Hreidarsson syndrome with novel RTEL1 variants.

Pediatric blood &amp; cancer
2023

Dyskeratosis congenita with heterozygous RTEL1 mutations presenting with fibrotic hypersensitivity pneumonitis.

Respiratory medicine case reports
2023

Dyskeratosis Congenita Links Telomere Attrition to 
Age-Related Systemic Energetics.

The journals of gerontology. Series A, Biological sciences and medical sciences
2023

Germline Pathogenic Variants in Squamous Cell Carcinoma of the Head and Neck.

Folia biologica
2023

Subretinal drusenoid deposits and bilateral capillary peripheral occlusion in a patient with dyskeratosis congenita.

Canadian journal of ophthalmology. Journal canadien d'ophtalmologie
2022

Autoimmune Neutropenia and Immune-Dysregulation in a Patient Carrying a TINF2 Variant.

International journal of molecular sciences
2022

Dyskeratosis congenita and telomere biology disorders.

Hematology. American Society of Hematology. Education Program
2022

Novel TINF2 gene mutation in dyskeratosis congenita with extremely short telomeres: A case report.

World journal of clinical cases
2022

Dermoscopic and Onychoscopic Features of Dyskeratosis Congenita.

Indian dermatology online journal
2022

Recent advances in understanding telomere diseases.

Faculty reviews
2022

A missense variant in the nuclear localization signal of DKC1 causes Hoyeraal-Hreidarsson syndrome.

NPJ genomic medicine
2022

Late Presentation of Dyskeratosis Congenita: Germline Predisposition to Adult-Onset Secondary Acute Myeloid Leukemia.

Hematology reports
2022

Painful thickened skin on the soles of the feet.

JAAD case reports
2023

Regulator of telomere elongation helicase 1 gene and its association with malignancy.

Cancer reports (Hoboken, N.J.)
2021

de Novo TINF2 C.845G>A: Pathogenic Variant in Patient with Dyskeratosis Congenita.

Balkan journal of medical genetics : BJMG
2023

Preretinal Microvascular Tufts Associated with Dyskeratosis Congenita.

Ophthalmology
2022

[Bone marrow failure and TP53 activating mutations].

[Rinsho ketsueki] The Japanese journal of clinical hematology
2022

Coats plus syndrome with new observation of drusenoid retinal pigment epithelial detachments in a teenager.

American journal of ophthalmology case reports
2022

Next-generation sequencing errors due to genetic variation in WRAP53 encoding TCAB1 on chromosome 17.

Human mutation
2022

A Novel Variant and a Missense Variant Identified in the DKC1 Gene in Three Chinese Familieswith Dyskeratosis Congenita.

Clinical, cosmetic and investigational dermatology
2022

Fanconi anemia and dyskeratosis congenita/telomere biology disorders: Two inherited bone marrow failure syndromes with genomic instability.

Frontiers in oncology
2022

Severe immunochemotherapy-induced toxicities in a patient with dyskeratosis congenita and literature review.

Hematology (Amsterdam, Netherlands)
2022

Telomere biology disorders: time for moving towards the clinic?

Trends in molecular medicine
2022

Domain specific mutations in dyskerin disrupt 3' end processing of scaRNA13.

Nucleic acids research
2022

The biology and management of dyskeratosis congenita and related disorders of telomeres.

Expert review of hematology
2022

Challenges in the interpretation of a germline TERT variant in a patient with juvenile myelomonocytic leukemia.

Pediatric blood &amp; cancer
2022

Pediatric cystectomy for refractory cystitis post-bone marrow transplant in dyskeratosis congenita: A case report.

Urology case reports
2021

NAD-Linked Metabolism and Intervention in Short Telomere Syndromes and Murine Models of Telomere Dysfunction.

Frontiers in aging
2022

Effects of Recloning on the Telomere Lengths of Mouse Terc+/- Nuclear Transfer-Derived Embryonic Stem Cells.

Stem cells and development
2022

Chemical Modifications of Ribosomal RNA.

Methods in molecular biology (Clifton, N.J.)
2022

Dysplastic megakaryocytes in dyskeratosis congenita with variant in PARN.

Blood
2022

Silencing circular RNA-friend leukemia virus integration 1 restrained malignancy of CC cells and oxaliplatin resistance by disturbing dyskeratosis congenita 1.

Open life sciences
2022

Recent advances in hematopoietic cell transplantation for inherited bone marrow failure syndromes.

International journal of hematology
2022

Dyskerin Downregulation Can Induce ER Stress and Promote Autophagy via AKT-mTOR Signaling Deregulation.

Biomedicines
2022

Inherited bone marrow failure in the pediatric patient.

Blood
2023

Truncating TINF2 p.Tyr312Ter variant and inherited breast cancer susceptibility.

Familial cancer
2022

GSK3 inhibition rescues growth and telomere dysfunction in dyskeratosis congenita iPSC-derived type II alveolar epithelial cells.

eLife
2022

Genetics and genomics of bone marrow failure syndrome.

Blood research
2022

Are Dyskeratosis Congenita patients at higher risk of symptomatic COVID-19?

Medical hypotheses
2022

Case Report: Novel Biallelic Variants in DNAJC21 Causing an Inherited Bone Marrow Failure Spectrum Phenotype: An Odyssey to Diagnosis.

Frontiers in genetics
2022

Case Report: A Missense Mutation in Dyskeratosis Congenita 1 Leads to a Benign Form of Dyskeratosis Congenita Syndrome With the Mucocutaneous Triad.

Frontiers in pediatrics
2022

Sex differences in telomere length, lifespan, and embryonic dyskerin levels.

Aging cell
2022

Editing TINF2 as a potential therapeutic approach to restore telomere length in dyskeratosis congenita.

Blood
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Doenças relacionadas

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Referências e fontes

Bases de dados externas citadas neste artigo

Publicações científicas

Artigos indexados no PubMed ligados a esta doença no grafo RarasNet — título, periódico e PMID direto da fonte, sem intermediação de IA.

  1. Monoallelic PARN mutation presenting as pancytopenia, hepatic fibrosis and idiopathic pulmonary fibrosis.
    BMJ case reports· 2026· PMID 41741124mais citado
  2. RNA Analysis Uncovers Pathogenic PARN Variant in Dyskeratosis Congenita.
    Clinical genetics· 2026· PMID 40859110mais citado
  3. Avascular Necrosis and Minimal Trauma Fractures in Telomere Biology Disorders.
    Clinical genetics· 2026· PMID 40762130mais citado
  4. Dyskerin dysfunction in cancer development: from telomere dysregulation to immune deficiency.
    American journal of cancer research· 2026· PMID 41868676mais citado
  5. Beyond iron deficiency: A comprehensive national survey of anaemia etiology in Sri Lankan young adults.
    Scientific reports· 2026· PMID 41857086mais citado
  6. Adriamycin, Vinblastine and Dacarbazine With Immunotherapy Achieves Complete Metabolic Response in a Patient With Classical Hodgkin Lymphoma and Dyskeratosis Congenita.
    J Hematol· 2026· PMID 41983152recente
  7. Late-onset dyskeratosis congenita due to a TERC (n.269G > C) variant-first reported case from Indonesia: a case report.
    J Med Case Rep· 2026· PMID 41965821recente
  8. Rare somatic manifestations of telomere biology disorders.
    Semin Hematol· 2026· PMID 41956864recente
  9. Bone marrow failure in telomere biology disorders: Current understanding and the emerging landscape of non-transplant therapies.
    Semin Hematol· 2026· PMID 41904107recente
  10. Clonal hematopoiesis in telomere biology disorders.
    Semin Hematol· 2026· PMID 41896073recente

Bases de dados e fontes oficiais

Identificadores e referências canônicas usadas para montar este verbete.

  1. ORPHA:1775(Orphanet)
  2. MONDO:0015780(MONDO)
  3. GARD:10905(GARD (NIH))
  4. Variantes catalogadas(ClinVar)
  5. Busca completa no PubMed(PubMed)
  6. Q3709312(Wikidata)

Dados compilados pelo RarasNet a partir de fontes abertas (Orphanet, OMIM, MONDO, PubMed/EuropePMC, ClinicalTrials.gov, DATASUS, PCDT/MS). Este conteúdo é informativo e não substitui avaliação médica.

Conteúdo mantido por Agente Raras · Médicos e pesquisadores podem colaborar

Disqueratose congênita
Compêndio · Raras BR

Disqueratose congênita

ORPHA:1775 · MONDO:0015780
Prevalência
1-9 / 1 000 000
Herança
Autosomal dominant, Autosomal recessive, X-linked recessive
CID-10
Q82.8 · Outras malformações congênitas especificadas da pele
CID-11
Ensaios
7 ativos
Início
Adolescent, Adult, Childhood, Infancy, Neonatal
Prevalência
0.1 (Europe)
MedGen
UMLS
C0265965
Repurposing
3 candidatos
ingenolPKC activator
retinolretinoid receptor ligand
ureahydroxy radical formation stimulant
EuropePMC
Wikidata
Papers 10a
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