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Xeroderma pigmentoso
ORPHA:910CID-10 · Q82.1CID-11 · LD27.1DOENÇA RARA

O xeroderma pigmentoso (XP) é uma genodermatose rara caracterizada por extrema sensibilidade às alterações induzidas pela radiação ultravioleta (UV) na pele e nos olhos, e múltiplos cânceres de pele. É subdividido em 8 grupos de complementação, de acordo com o gene afetado: XP clássico (XPA a XPG) e variante XP (XPV).

Mantido por Agente Raras·Colaborar como especialista →

Introdução

O que você precisa saber de cara

📋

O xeroderma pigmentoso (XP) é uma genodermatose rara caracterizada por extrema sensibilidade às alterações induzidas pela radiação ultravioleta (UV) na pele e nos olhos, e múltiplos cânceres de pele. É subdividido em 8 grupos de complementação, de acordo com o gene afetado: XP clássico (XPA a XPG) e variante XP (XPV).

Pesquisas ativas
2 ensaios
16 total registrados no ClinicalTrials.gov
Publicações científicas
4.829 artigos
Último publicado: 2026 Apr 10

Escala de raridade

CLASSIFICAÇÃO ORPHANET · BRASIL 2024
1-9 / 1 000 000
Ultra-rara
<1/50k
Muito rara
1/20k
Rara
1/10k
Pouco freq.
1/5k
Incomum
1/2k
Prevalência
0.1
United States
Início
All ages
🏥
SUS: Cobertura mínimaScore: 15%
CID-10: Q82.1
🇧🇷Dados SUS / DATASUS
PROCEDIMENTOS SIGTAP (5)
0202010503
Cariótipo — bandas G, Q ou Rgenetic_test
0202010600
Pesquisa de microdeleções/microduplicações por FISHlab_test
0202010694
Sequenciamento completo do exoma (WES)rehabilitation
0202010260
Dosagem de alfa-fetoproteína
0301070040
Atendimento em reabilitação — doenças raras
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Sinais e sintomas

O que aparece no corpo e com que frequência cada sintoma acontece

Partes do corpo afetadas

🧬
Pele e cabelo
18 sintomas
🧠
Neurológico
16 sintomas
👁️
Olhos
12 sintomas
📏
Crescimento
4 sintomas
🦴
Ossos e articulações
4 sintomas
👂
Ouvidos
3 sintomas

+ 49 sintomas em outras categorias

Características mais comuns

90%prev.
Febre
Muito frequente (99-80%)
90%prev.
Deficiência intelectual
Muito frequente (99-80%)
90%prev.
Anormalidade no EEG
Muito frequente (99-80%)
90%prev.
Telangiectasia da pele
Muito frequente (99-80%)
90%prev.
Pele fina
Muito frequente (99-80%)
90%prev.
Atrofia óptica
Muito frequente (99-80%)
111sintomas
Muito frequente (21)
Frequente (13)
Ocasional (26)
Sem dados (51)

Os sintomas variam de pessoa para pessoa. Abaixo estão as 111 características clínicas mais associadas, ordenadas por frequência.

FebreFever
Muito frequente (99-80%)90%
Deficiência intelectualIntellectual disability
Muito frequente (99-80%)90%
Anormalidade no EEGEEG abnormality
Muito frequente (99-80%)90%
Telangiectasia da peleTelangiectasia of the skin
Muito frequente (99-80%)90%
Pele finaThin skin
Muito frequente (99-80%)90%

Linha do tempo da pesquisa

Publicações por ano — veja quando o interesse científico cresceu
Anos de pesquisa1desde 2026
Total histórico4.829PubMed
Últimos 10 anos200publicações
Pico2025100 papers
Linha do tempo
2026Hoje · 2026🧪 1991Primeiro ensaio clínico📈 2025Ano de pico
Publicações por ano (últimos 10 anos)

Encontrou um erro ou informação desatualizada? Sugira uma correção →

Genética e causas

O que está alterado no DNA e como passa nas famílias

Genes associados

8 genes identificados com associação a esta condição. Padrão de herança: Autosomal recessive.

ERCC3General transcription and DNA repair factor IIH helicase/translocase subunit XPBDisease-causing germline mutation(s) inTolerante
FUNÇÃO

ATP-dependent 3'-5' DNA helicase/translocase (PubMed:17466626, PubMed:27193682, PubMed:33902107, PubMed:8465201, PubMed:8663148). Binds dsDNA rather than ssDNA, unzipping it in a translocase rather than classical helicase activity (PubMed:27193682, PubMed:33902107). Component of the general transcription and DNA repair factor IIH (TFIIH) core complex (PubMed:10024882, PubMed:17466626, PubMed:8157004, PubMed:8465201). When complexed to CDK-activating kinase (CAK), involved in RNA transcription by

LOCALIZAÇÃO

Nucleus

VIAS BIOLÓGICAS (10)
RNA Polymerase II Promoter EscapeRNA Polymerase II Transcription Initiation And Promoter ClearanceRNA Polymerase II HIV Promoter EscapeTranscription of the HIV genomemRNA Capping
MECANISMO DE DOENÇA

Xeroderma pigmentosum complementation group B

An autosomal recessive pigmentary skin disorder characterized by solar hypersensitivity of the skin, high predisposition for developing cancers on areas exposed to sunlight and, in some cases, neurological abnormalities. The skin develops marked freckling and other pigmentation abnormalities. Some XP-B patients present features of Cockayne syndrome, including cachectic dwarfism, pigmentary retinopathy, ataxia, decreased nerve conduction velocities. The phenotype combining xeroderma pigmentosum and Cockayne syndrome traits is referred to as XP-CS complex.

EXPRESSÃO TECIDUAL(Ubíquo)
Cérebro - Hemisfério cerebelar
90.1 TPM
Cerebelo
88.4 TPM
Útero
71.8 TPM
Cervix Endocervix
71.5 TPM
Nervo tibial
71.4 TPM
OUTRAS DOENÇAS (5)
xeroderma pigmentosum group Btrichothiodystrophy 2, photosensitivetrichothiodystrophyxeroderma pigmentosum
HGNC:3435UniProt:P19447
POLHDNA polymerase etaDisease-causing germline mutation(s) inTolerante
FUNÇÃO

DNA polymerase specifically involved in the DNA repair by translesion synthesis (TLS) (PubMed:10385124, PubMed:11743006, PubMed:16357261, PubMed:20388628, PubMed:24449906, PubMed:24553286, PubMed:38212351). Due to low processivity on both damaged and normal DNA, cooperates with the heterotetrameric (REV3L, REV7, POLD2 and POLD3) POLZ complex for complete bypass of DNA lesions. Inserts one or 2 nucleotide(s) opposite the lesion, the primer is further extended by the tetrameric POLZ complex. In th

LOCALIZAÇÃO

Nucleus

VIAS BIOLÓGICAS (3)
HDR through Homologous Recombination (HRR)Translesion Synthesis by POLHTermination of translesion DNA synthesis
MECANISMO DE DOENÇA

Xeroderma pigmentosum variant type

An autosomal recessive pigmentary skin disorder characterized by solar hypersensitivity of the skin, high predisposition for developing cancers on areas exposed to sunlight and, in some cases, neurological abnormalities. XPV shows normal nucleotide excision repair, but an exaggerated delay in recovery of replicative DNA synthesis. Most patients with the variant type of xeroderma pigmentosum do not develop clinical symptoms and skin neoplasias until a later age. Clinical manifestations are limited to photo-induced deterioration of the skin and eyes.

EXPRESSÃO TECIDUAL(Ubíquo)
Linfócitos
39.4 TPM
Fibroblastos
31.8 TPM
Testículo
21.5 TPM
Cervix Ectocervix
11.3 TPM
Tireoide
11.2 TPM
OUTRAS DOENÇAS (1)
xeroderma pigmentosum variant type
HGNC:9181UniProt:Q9Y253
DDB2DNA damage-binding protein 2Disease-causing germline mutation(s) inTolerante
FUNÇÃO

Protein, which is both involved in DNA repair and protein ubiquitination, as part of the UV-DDB complex and DCX (DDB1-CUL4-X-box) complexes, respectively (PubMed:10882109, PubMed:11278856, PubMed:11705987, PubMed:12732143, PubMed:15882621, PubMed:16473935, PubMed:18593899, PubMed:32789493, PubMed:9892649). Core component of the UV-DDB complex (UV-damaged DNA-binding protein complex), a complex that recognizes UV-induced DNA damage and recruit proteins of the nucleotide excision repair pathway (t

LOCALIZAÇÃO

NucleusChromosome

VIAS BIOLÓGICAS (4)
DNA Damage Recognition in GG-NERTP53 Regulates Transcription of DNA Repair GenesNeddylationUb-specific processing proteases
MECANISMO DE DOENÇA

Xeroderma pigmentosum complementation group E

An autosomal recessive pigmentary skin disorder characterized by solar hypersensitivity of the skin, high predisposition for developing cancers on areas exposed to sunlight and, in some cases, neurological abnormalities. The skin develops marked freckling and other pigmentation abnormalities. XP-E patients show a mild phenotype with minimal or no neurologic features.

EXPRESSÃO TECIDUAL(Ubíquo)
Linfócitos
99.4 TPM
Skin Not Sun Exposed Suprapubic
65.1 TPM
Skin Sun Exposed Lower leg
63.4 TPM
Aorta
49.0 TPM
Útero
42.9 TPM
OUTRAS DOENÇAS (2)
xeroderma pigmentosum group Exeroderma pigmentosum
HGNC:2718UniProt:Q92466
XPCDNA repair protein complementing XP-C cellsDisease-causing germline mutation(s) inTolerante
FUNÇÃO

Involved in global genome nucleotide excision repair (GG-NER) by acting as damage sensing and DNA-binding factor component of the XPC complex (PubMed:10734143, PubMed:10873465, PubMed:12509299, PubMed:12547395, PubMed:19609301, PubMed:19941824, PubMed:20028083, PubMed:20649465, PubMed:20798892, PubMed:9734359). Has only a low DNA repair activity by itself which is stimulated by RAD23B and RAD23A. Has a preference to bind DNA containing a short single-stranded segment but not to damaged oligonucl

LOCALIZAÇÃO

NucleusChromosomeCytoplasm

VIAS BIOLÓGICAS (2)
DNA Damage Recognition in GG-NERSUMOylation of DNA damage response and repair proteins
MECANISMO DE DOENÇA

Xeroderma pigmentosum complementation group C

An autosomal recessive pigmentary skin disorder characterized by solar hypersensitivity of the skin, high predisposition for developing cancers on areas exposed to sunlight and, in some cases, neurological abnormalities. The skin develops marked freckling and other pigmentation abnormalities.

EXPRESSÃO TECIDUAL(Ubíquo)
Fibroblastos
82.0 TPM
Nervo tibial
64.0 TPM
Cervix Endocervix
62.8 TPM
Ovário
62.1 TPM
Útero
58.7 TPM
OUTRAS DOENÇAS (2)
xeroderma pigmentosum group Cxeroderma pigmentosum
HGNC:12816UniProt:Q01831
ERCC5DNA excision repair protein ERCC-5Disease-causing germline mutation(s) inTolerante
FUNÇÃO

Single-stranded structure-specific DNA endonuclease involved in DNA excision repair (PubMed:32522879, PubMed:32821917, PubMed:7651464, PubMed:8078765, PubMed:8090225, PubMed:8206890). Makes the 3'incision in DNA nucleotide excision repair (NER) (PubMed:32522879, PubMed:32821917, PubMed:8078765, PubMed:8090225). Binds and bends DNA repair bubble substrate and breaks base stacking at the single-strand/double-strand DNA junction of the DNA bubble (PubMed:32522879). Plays a role in base excision rep

LOCALIZAÇÃO

NucleusChromosome

VIAS BIOLÓGICAS (3)
Dual incision in TC-NERDual Incision in GG-NERFormation of Incision Complex in GG-NER
MECANISMO DE DOENÇA

Xeroderma pigmentosum complementation group G

An autosomal recessive pigmentary skin disorder characterized by solar hypersensitivity of the skin, high predisposition for developing cancers on areas exposed to sunlight and, in some cases, neurological abnormalities. The skin develops marked freckling and other pigmentation abnormalities. Some XP-G patients present features of Cockayne syndrome, cachectic dwarfism, pigmentary retinopathy, ataxia, decreased nerve conduction velocities. The phenotype combining xeroderma pigmentosum and Cockayne syndrome traits is referred to as XP-CS complex.

EXPRESSÃO TECIDUAL(Baixa expressão)
Linfócitos
4.7 TPM
Fibroblastos
3.5 TPM
Nervo tibial
3.4 TPM
Tireoide
3.3 TPM
Ovário
3.3 TPM
OUTRAS DOENÇAS (5)
xeroderma pigmentosum group Gcerebrooculofacioskeletal syndrome 3xeroderma pigmentosumxeroderma pigmentosum-Cockayne syndrome complex
HGNC:3437UniProt:P28715
XPADNA repair protein complementing XP-A cellsDisease-causing germline mutation(s) inTolerante
FUNÇÃO

Involved in DNA nucleotide excision repair (NER). Initiates repair by binding to damaged sites with various affinities, depending on the photoproduct and the transcriptional state of the region. Required for UV-induced CHEK1 phosphorylation and the recruitment of CEP164 to cyclobutane pyrimidine dimmers (CPD), sites of DNA damage after UV irradiation (PubMed:19197159). During NER stimulates the 5'-3' helicase activity of XPD/ERCC2 and the DNA translocase activity of XPB/ERCC3 (PubMed:31253769).

LOCALIZAÇÃO

Nucleus

VIAS BIOLÓGICAS (4)
Formation of TC-NER Pre-Incision ComplexDual incision in TC-NERDual Incision in GG-NERFormation of Incision Complex in GG-NER
MECANISMO DE DOENÇA

Xeroderma pigmentosum complementation group A

An autosomal recessive pigmentary skin disorder characterized by solar hypersensitivity of the skin, high predisposition for developing cancers on areas exposed to sunlight and, in some cases, neurological abnormalities. The skin develops marked freckling and other pigmentation abnormalities. XP-A patients show the most severe skin symptoms and progressive neurological disorders.

EXPRESSÃO TECIDUAL(Ubíquo)
Tireoide
50.2 TPM
Cólon sigmoide
36.7 TPM
Aorta
36.7 TPM
Artéria tibial
36.5 TPM
Nervo tibial
36.5 TPM
OUTRAS DOENÇAS (2)
xeroderma pigmentosum group Axeroderma pigmentosum
HGNC:12814UniProt:P23025
ERCC4DNA repair endonuclease XPFDisease-causing germline mutation(s) inTolerante
FUNÇÃO

Catalytic component of a structure-specific DNA repair endonuclease responsible for the 5-prime incision during DNA repair, and which is essential for nucleotide excision repair (NER) and interstrand cross-link (ICL) repair

LOCALIZAÇÃO

NucleusChromosome

VIAS BIOLÓGICAS (5)
HDR through Single Strand Annealing (SSA)Dual incision in TC-NERDual Incision in GG-NERFormation of Incision Complex in GG-NERFanconi Anemia Pathway
MECANISMO DE DOENÇA

Xeroderma pigmentosum complementation group F

An autosomal recessive pigmentary skin disorder characterized by solar hypersensitivity of the skin, high predisposition for developing cancers on areas exposed to sunlight and, in some cases, neurological abnormalities. The skin develops marked freckling and other pigmentation abnormalities. XP-F patients show a mild phenotype.

EXPRESSÃO TECIDUAL(Ubíquo)
Testículo
13.8 TPM
Fibroblastos
6.2 TPM
Linfócitos
5.4 TPM
Cervix Endocervix
5.1 TPM
Fallopian Tube
5.1 TPM
OUTRAS DOENÇAS (7)
Fanconi anemia complementation group Qxeroderma pigmentosum group FXFE progeroid syndromexeroderma pigmentosum
HGNC:3436UniProt:Q92889
ERCC2General transcription and DNA repair factor IIH helicase subunit XPDDisease-causing germline mutation(s) inTolerante
FUNÇÃO

ATP-dependent 5'-3' DNA helicase (PubMed:31253769, PubMed:8413672, PubMed:9771713). Component of the general transcription and DNA repair factor IIH (TFIIH) core complex, not absolutely essential for minimal transcription in vitro (PubMed:10024882, PubMed:17466626, PubMed:9771713). Required for transcription-coupled nucleotide excision repair (NER) of damaged DNA; recognizes damaged bases (PubMed:17466626, PubMed:23352696, PubMed:9771713). Sequestered in chromatin on UV-damaged DNA (PubMed:23352

LOCALIZAÇÃO

NucleusCytoplasm, cytoskeleton, spindle

VIAS BIOLÓGICAS (1)
Cytosolic iron-sulfur cluster assembly
MECANISMO DE DOENÇA

Xeroderma pigmentosum complementation group D

An autosomal recessive pigmentary skin disorder characterized by solar hypersensitivity of the skin, high predisposition for developing cancers on areas exposed to sunlight and, in some cases, neurological abnormalities. The skin develops marked freckling and other pigmentation abnormalities. Some XP-D patients present features of Cockayne syndrome, including cachectic dwarfism, pigmentary retinopathy, ataxia, decreased nerve conduction velocities. The phenotype combining xeroderma pigmentosum and Cockayne syndrome traits is referred to as XP-CS complex.

EXPRESSÃO TECIDUAL(Ubíquo)
Fibroblastos
40.4 TPM
Testículo
31.6 TPM
Linfócitos
20.0 TPM
Pituitária
18.6 TPM
Tireoide
17.6 TPM
OUTRAS DOENÇAS (7)
xeroderma pigmentosum group Dtrichothiodystrophy 1, photosensitivecerebrooculofacioskeletal syndrome 2xeroderma pigmentosum
HGNC:3434UniProt:P18074

Variantes genéticas (ClinVar)

283 variantes patogênicas registradas no ClinVar.

🧬 ERCC3: NM_000122.2(ERCC3):c.700C>T (p.Arg234Ter) ()
🧬 ERCC3: NM_000122.2(ERCC3):c.1827+2T>G ()
🧬 ERCC3: NM_000122.2(ERCC3):c.1028-2A>G ()
🧬 ERCC3: NM_000122.2(ERCC3):c.335dup (p.His112fs) ()
🧬 ERCC3: NM_000122.2(ERCC3):c.1558C>T (p.Arg520Trp) ()
Ver todas no ClinVar

Classificação de variantes (ClinVar)

Distribuição de 2,231 variantes classificadas pelo ClinVar.

1004
1227
VUS (45.0%)
Benigna (55.0%)
VARIANTES MAIS SIGNIFICATIVAS
ERCC4: NM_005236.3(ERCC4):c.675A>G (p.Ile225Met) [Uncertain significance]
ERCC4: NM_005236.3(ERCC4):c.316G>A (p.Val106Ile) [Uncertain significance]
ERCC4: NM_005236.3(ERCC4):c.1492G>T (p.Val498Leu) [Uncertain significance]
ERCC4: NM_005236.3(ERCC4):c.175G>T (p.Val59Leu) [Uncertain significance]
ERCC4: NM_005236.3(ERCC4):c.2455A>G (p.Ser819Gly) [Uncertain significance]

Vias biológicas (Reactome)

39 vias biológicas associadas aos genes desta condição.

Formation of RNA Pol II elongation complex Formation of the Early Elongation Complex Formation of HIV elongation complex in the absence of HIV Tat Formation of the HIV-1 Early Elongation Complex RNA Pol II CTD phosphorylation and interaction with CE during HIV infection HIV Transcription Initiation RNA Polymerase II HIV Promoter Escape Transcription of the HIV genome Formation of HIV-1 elongation complex containing HIV-1 Tat Tat-mediated elongation of the HIV-1 transcript NoRC negatively regulates rRNA expression Formation of Incision Complex in GG-NER Dual Incision in GG-NER RNA Polymerase II Pre-transcription Events Formation of TC-NER Pre-Incision Complex Transcription-Coupled Nucleotide Excision Repair (TC-NER) Dual incision in TC-NER Gap-filling DNA repair synthesis and ligation in TC-NER TP53 Regulates Transcription of DNA Repair Genes mRNA Capping RNA Polymerase I Transcription Initiation RNA Polymerase I Promoter Escape RNA Polymerase II Promoter Escape RNA Polymerase II Transcription Pre-Initiation And Promoter Opening RNA Polymerase I Transcription Termination RNA Polymerase II Transcription Initiation RNA Polymerase II Transcription Elongation RNA Polymerase II Transcription Initiation And Promoter Clearance RNA Pol II CTD phosphorylation and interaction with CE Translesion Synthesis by POLH Termination of translesion DNA synthesis HDR through Homologous Recombination (HRR) Ub-specific processing proteases DNA Damage Recognition in GG-NER Neddylation SUMOylation of DNA damage response and repair proteins HDR through Single Strand Annealing (SSA) Fanconi Anemia Pathway Cytosolic iron-sulfur cluster assembly

Diagnóstico

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·Pré-clínico7
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🇧🇷 Atendimento SUS — Xeroderma pigmentoso

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Ensaios clínicos abertos e novidades científicas recentes

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16 ensaios clínicos encontrados, 2 ativos.

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Publicações mais relevantes

Timeline de publicações
1.046 papers (10 anos)

Mostrando amostra de 200 publicações de um total de 1.046

#1

DNA repair-associated nucleases induce double-strand breaks following sequential exposure to UVA1 and UVB.

Photochemical &amp; photobiological sciences : Official journal of the European Photochemistry Association and the European Society for Photobiology2026 Mar 12

Solar UV comprises UVA and UVB; each exerts distinct biological effects, while their combined impact is not yet fully understood. We previously reported that human keratinocytes exposed to UVA1 followed by UVB irradiation exhibited severe cell death accompanied by DNA double-strand break (DSB) formation. In this study, we confirmed the DSB formation following sequential exposure to UVA1 and UVB and investigated the underlying mechanisms. The occurrence of DSBs was validated by biased sinusoidal field gel electrophoresis and the detection of phosphorylated histone H2AX and RPA. Notably, DSB induction was absent in xeroderma pigmentosum (XP) mutant cell lines, suggesting that nucleotide excision repair (NER) of UVB-induced pyrimidine dimers serves as a trigger for DSB formation. RPA, which binds to single-stranded DNA (ssDNA) gaps, and the replication factor PCNA rapidly accumulated at UV-damaged sites and persisted for an extended period in cells pre-irradiated with UVA1, indicating that NER-mediated ssDNA gaps were stabilized by UVA1 exposure. Furthermore, DSB formation was markedly suppressed by knockdown of the nucleases, EXO1 and MRE11. Inhibition of MRE11 endonuclease activity with PFM01 suppressed DSB formation after sequential exposure to UVA1 and UVB, whereas inhibition of its exonuclease activity with Mirin had no significant effect. These findings suggest that ssDNA gaps stabilized by UVA1 pre-irradiation are extended by EXO1, while MRE11 introduces a nick, ultimately leading to DSB formation.

#2

Pre-incision structures reveal principles of DNA nucleotide excision repair.

Nature2026 Feb 11

Nucleotide excision repair (NER) removes bulky adducts from genomic DNA and prevents the ultraviolet light-sensitivity disease xeroderma pigmentosum, cancer and premature ageing1. After initial lesion recognition by XPC in global genome repair or by stalled RNA polymerases in transcription-coupled repair, a lesion and surrounding DNA duplex are unwound by TFIIH, which includes the ATPases XPB and XPD, and additional NER factors XPA, XPF, XPG and RPA, to form a DNA bubble2 comprising around 27 nucleotides. The double strand-single strand (ds-ss) junction-specific endonucleases XPF and XPG cleave DNA on the 5' and 3' sides of the lesion, respectively. Here we report the functional steps and atomic structures of the ATPase-driven and lesion-dependent DNA bubble formation and arrangement of the complete NER factors for dual incision. The unwinding of nearly 30 base pairs of DNA depends mainly on the double strand DNA translocase XPB and the duplex dividers XPA and XPF. XPD binds the lesion strand with XPF at the 5' ds-ss junction. XPF cuts the lesion strand only after XPG binds the 3' ds-ss junction. The ERCC1 subunit of XPF facilitates DNA strand separation and recruitment of RPA to the non-lesion strand. These findings provide insights on the causes of human diseases and potential targets for enhancing chemotherapeutic efficacy.

#3

Unusual Disease-Progression in Two Siblings With Xeroderma Pigmentosum Group G.

Clinical genetics2026 Jan 02

Protein truncation mutations in the gene for XPG nuclease cause a very severe clinical phenotype. Two siblings have splicing mutations, which result in in-frame deletions and a less severe phenotype.

#4

Association of XPC rs2228001, ESR2 rs1256030, and rs4986938 Gene Polymorphisms With Breast Cancer Risk in Bangladeshi Women: A Case-Control Study.

Clinical breast cancer2026 Feb 21

Breast cancer is one of the most destructive diseases among females worldwide, especially in developing countries. XPC and ESR2 genes have been identified in multiple malignancies. Therefore, we aimed to determine the association of XPC (rs2228001) and ESR2 (rs1256030/rs4986938) polymorphisms with breast cancer susceptibility in the Bangladeshi population. This case-control study was carried out on 220 breast cancer patients and 208 healthy volunteers. Genotyping was performed using the polymerase chain reaction (PCR)-restriction fragment length polymorphism (PCR-RFLP) technique. Analyses were conducted using the statistical software application SPSS (version 25.0). Logistic regression was employed to assess the genetic association, employing the odds ratio (OR) and 95% confidence intervals (CIs). The XPC rs2228001 polymorphism demonstrated a significant association with breast cancer risk, with the CC genotype (OR = 3.27, P = .009), dominant model (OR = 1.67, P = .011), recessive model (OR = 2.87, P = .019), and allelic model (OR = 1.69, P = .002). The ESR2 rs1256030 variant exhibited a significantly elevated risk of breast cancer across all genetic models (P < .05). Whereas, the ESR2 rs4986938 polymorphism showed a significant correlation with breast cancer susceptibility in the TT genotype under the additive model 2 (OR = 3.83, P = .011) and the recessive model (OR = 3.73, P = .021). Our study concluded that the XPC rs2228001, ESR2 rs1256030, and ESR2 rs4986938 gene polymorphisms might be associated with breast cancer risk in the Bangladeshi women. Larger studies across diverse populations are recommended to validate these findings.

#5

XAB2: a link between RNA metabolism, DNA damage repair, and human health.

Transcription2026 Mar 18

Cells have evolved multiple mechanisms to preserve genome integrity, collectively known as DNA damage response (DDR). Rather than acting separately, the DDR often interacts with transcription and mRNA splicing; however, the underlying molecular mechanisms of this cross-talk are still poorly understood. Consistent with this, components of the splicing machinery are increasingly being recognized as factors with a direct role in sensing, signaling, and repairing DNA damage. Xeroderma pigmentosum group A-binding protein 2 (XAB2), which plays a well-characterized role in mRNA splicing, has also been implicated in the repair of transcription-blocking DNA lesions, transcription elongation, mRNA export, RNA surveillance, and R-loop processing. XAB2 is critical for a wide variety of biological processes, including the mitotic cell cycle, cell differentiation, stress responses, tissue homeostasis, and cellular senescence. However, the mechanism by which XAB2 functions outside of mRNA splicing remains unclear. In this review, we summarize the current knowledge of the biological processes affected by XAB2 in different cellular contexts. Furthermore, we discuss the link between XAB2 and human health, with a particular focus on cancer. This review aims to emphasize the importance of XAB2 and raise awareness of its physiological contributions.

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📚 EuropePMC2.350 artigos no totalmostrando 197

2026

From Xeroderma Pigmentosum to malignant melanoma: case of an 8-year-old boy.

Oxford medical case reports
2026

Association of XPC rs2228001, ESR2 rs1256030, and rs4986938 Gene Polymorphisms With Breast Cancer Risk in Bangladeshi Women: A Case-Control Study.

Clinical breast cancer
2026

Treatment With Sonidegib for Multiple Basal Cell Carcinomas in a Patient With Xeroderma Pigmentosum.

Actas dermo-sifiliograficas
2026

XAB2: a link between RNA metabolism, DNA damage repair, and human health.

Transcription
2026

Feasibility of Allogeneic Stem Cell Transplantation in Xeroderma Pigmentosum Patients With Hematologic Malignancies, a Report From the SFGM-TC (Société Francophone de Greffe de Moelle et de Thérapie Cellulaire).

American journal of hematology
2026

Lip and oral involvement in 116 patients with xeroderma pigmentosum in the UK.

Clinical and experimental dermatology
2026

DNA repair-associated nucleases induce double-strand breaks following sequential exposure to UVA1 and UVB.

Photochemical &amp; photobiological sciences : Official journal of the European Photochemistry Association and the European Society for Photobiology
2026

Cutaneous Clearance in a Patient With Xeroderma Pigmentosum Under Treatment With Nivolumab.

Actas dermo-sifiliograficas
2026

Pre-incision structures reveal principles of DNA nucleotide excision repair.

Nature
2025

[Clinical characterization and genetic analysis of a patient with Xeroderma pigmentosum in conjunct with basal cell carcinoma and melanoma due to variants of XPC gene].

Zhonghua yi xue yi chuan xue za zhi = Zhonghua yixue yichuanxue zazhi = Chinese journal of medical genetics
2026

The interaction of XPG with TFIIH through p62 and XPD is required for the completion of nucleotide excision repair.

Nucleic acids research
2026

Arabidopsis XPD functions upstream of CDKA;1 to regulate stomatal development.

The New phytologist
2026

Xeroderma pigmentosum with multiple skin carcinoma and a homogenous XPC mutation: A case report from China and literature review.

The Journal of international medical research
2026

Type I interferon signaling defines a novel disease signature in xeroderma pigmentosum C human keratinocytes.

Scientific reports
2025

Successful adjuvant nivolumab therapy in a toddler with xeroderma pigmentosum and stage III melanoma.

European journal of dermatology : EJD
2026

Pembrolizumab Therapy Leads to Lentigines Resolution and Tumour Control in a Xeroderma Pigmentosum patient with Advanced Cutaneous Squamous Cell Carcinoma.

Clinical and experimental dermatology
2026

Cockayne syndrome mutation in XPG activate the integrated stress response.

Human genetics
2025

The association between DNA repair genes polymorphisms and cisplatin-induced ototoxicity in cancer patients: a systematic review.

Personalized medicine
2026

Antibody Deficiency in Xeroderma Pigmentosum.

Journal of clinical immunology
2026

Transcriptomic differences in immune- and stress-related pathways associated with artificial rearing in the endangered hog deer (Axis porcinus).

BMC genomics
2026

Genotype-phenotype associations in a robust cohort of 69 patients with xeroderma pigmentosum across Türkiye: a multicentre study.

The British journal of dermatology
2026

Unusual Disease-Progression in Two Siblings With Xeroderma Pigmentosum Group G.

Clinical genetics
2026

Oral squamous cell carcinoma risk and magnitude of association in inherited cancer predisposition syndromes: evidence from a large real-world cohort.

Oral surgery, oral medicine, oral pathology and oral radiology
2026

Vitiligo in Xeroderma Pigmentosum: A Protective Phenomenon Against Skin Cancer?

Photodermatology, photoimmunology &amp; photomedicine
2026

Liposomal Delivery of Pyronaridine as a Repurposed Inhibitor of ERCC1/XPF for the Sensitization of Colorectal Cancer Cells to Platinum Chemotherapeutics.

Molecular pharmaceutics
2025

Karyotypic Profiling of Induced Pluripotent Stem Cells Derived from a Xeroderma Pigmentosum Group C Patient.

Cells
2025

Molecular basis of XPF-ERCC1 targeting to SLX4-dependent DNA repair pathways.

Nature communications
2026

Altered expression of nucleotide excision repair genes ERCC2 and ERCC5 in prostate cancer tissues.

Cancer genetics
2025

Inhibition of scheggia/SLC25A1 citrate transporter alleviates XPD deficits.

Scientific reports
2025

Combining spironolactone to antiretroviral therapy accelerates HIV decay in humanized mice.

Emerging microbes &amp; infections
2025

Translocation mechanism of xeroderma pigmentosum group D protein on single-stranded DNA and genetic disease etiology.

Nature communications
2025

A retrospective Case-Control study investigating the association of XPC Lys939Gln (rs2228001), XPD Lys751Gln (rs13181), and TP53 Arg72Pro (rs1042522) with papillary thyroid carcinoma susceptibility in the Bangladeshi population.

Molecular biology reports
2025

Efficacy of Minnelide in a Next-Generation Dual-Recombinase Regulated Genetically Engineered Mouse Model of CIC::DUX4 Sarcoma.

bioRxiv : the preprint server for biology
2025

CRISPR/Cas9-mediated editing of XPA in induced pluripotent stem cells: A model for investigating Xeroderma Pigmentosum and NER dysfunction.

Stem cell research
2025

Expanding the landscape of nucleotide excision repair disorders: from discovery to therapy.

The Journal of clinical investigation
2026

Ocular Manifestations and Genetic Spectrum of Xeroderma Pigmentosum: Insights From an Indian Cohort in a Global Context.

Cornea
2025

Clinical Spectrum of Xeroderma Pigmentosum: An Observational Study.

Cureus
2025

Xeroderma pigmentosum associated with recurrent squamous and basal cell carcinoma: a case report from Nepal.

Annals of medicine and surgery (2012)
2025

Hemophagocytic Lymphohistiocytosis due to Brucellosis in a Xeroderma Pigmentosum Pediatric Patient: A Case Report and Review of the Literature.

Case reports in hematology
2025

Clinical outcomes of 3D-total body photography and digital dermoscopy for surveillance of high-risk melanoma patients. A prospective longitudinal observational study.

European journal of cancer (Oxford, England : 1990)
2025

Contribution of Xeroderma Pigmentosum Complementation Group C Genotypes to Colorectal Cancer in Taiwanese.

Anticancer research
2025

Exploring the interaction between nucleotide excision repair pathways and Huntington disease: Implications for neurodegeneration and phenotypic overlap.

Parkinsonism &amp; related disorders
2025

Break-induced replication is activated to repair R-loop-associated double-strand breaks in SETX-deficient cells.

Cell reports
2025

Juvenile Granulosa Cell Tumor in an Adolescent With Xeroderma Pigmentosum: A Rare Case and Implications for Surveillance.

Cureus
2025

Mechanistic insights into alcohol-induced DNA crosslink repair by Slx4-Xpf-Ercc1 nuclease complex in the Fanconi anaemia pathway.

Communications biology
2026

A Splicing Variant in XPA Results in Delayed Onset of Clinical Features of Xeroderma Pigmentosum.

The Journal of investigative dermatology
2026

A Novel Technique in One-Staged Reconstruction of Large Lower Eyelid Defects: Orbicularis Oculi Myoperiosteal Flap.

Ophthalmic plastic and reconstructive surgery
2025

Reverse Isotopic Response: A Novel Phenomenon in Xeroderma Pigmentosum.

Indian dermatology online journal
2025

TFIIH-p52ΔC defines a ninth xeroderma pigmentosum complementation-group XP-J and restores TFIIH stability to p8-defective trichothiodystrophy.

The Journal of clinical investigation
2025

XP-J, a ninth xeroderma pigmentosum complementation group, results from mutations in GTF2H4, encoding TFIIH-p52 subunit.

The Journal of clinical investigation
2025

The aflatoxin B1-induced formamidopyrimidine adduct is repaired by transcription-coupled nucleotide excision repair in human cells.

NAR cancer
2025

Anti-tumorigenic properties by trichothiodystrophy mutations in melanocytic cells.

NAR cancer
2025

Ferritin-Conjugated PROTAC Strategy for ERCC1/XPF Degradation and Platinum Sensitization in Resistant Tumors.

Journal of medicinal chemistry
2025

Embryonal Rhabdomyosarcoma of the Uterine Cervix in a Patient With Xeroderma Pigmentosum: An Exceptional Association.

Cureus
2025

Quantitative functional profiling of ERCC2 mutations deciphers cisplatin sensitivity in bladder cancer.

The Journal of clinical investigation
2025

Xeroderma pigmentosum: a 12-year experience in digital dermoscopy and reflectance confocal microscopy follow-up at a Cancer Center in Brazil.

Anais brasileiros de dermatologia
2025

Uncovering the Role of DNA Repair Impairment in UVA-Induced Mutagenesis in Human Xeroderma Pigmentosum Variant Cells.

Molecular carcinogenesis
2025

Uniquely high spontaneous mutational load in blood cells of XP-C patients.

bioRxiv : the preprint server for biology
2025

Inhibition of nucleotide excision repair proteins associated with cancer chemotherapy.

Biochimica et biophysica acta. Reviews on cancer
2025

[Characterization of periocular basal cell carcinoma in patients ≤ 50 years from 1996-2024 : A retrospective analysis at a university eye clinic].

Die Ophthalmologie
2025

Trichothiodystrophy-causative pathogenic variants impair a cooperative action of TFIIH and DDX1 in R-loop processing.

Nucleic acids research
2025

Genome-wide mapping of formaldehyde-induced DNA-protein crosslinks reveals unique patterns of formation and transcription-coupled removal in mammalian cells.

Nucleic acids research
2025

Prediction of response to neoadjuvant chemotherapy in patients with muscle-invasive urothelial bladder cancer: role of immune-related gene expression.

Cancer immunology, immunotherapy : CII
2025

A new multisystem ERCC1-hepatorenal syndrome: insights from a clinical cohort, molecular pathogenesis, and management guidelines.

European journal of human genetics : EJHG
2025

Ophthalmic and Cutaneous Manifestation of Xeroderma Pigmentosum in a 21-Year-Old Man: A Case Report.

Cureus
2025

Skin Signals: Exploring the Intersection of Cancer Predisposition Syndromes and Dermatological Manifestations.

International journal of molecular sciences
2025

The Price of Exposure: Xeroderma Pigmentosum and Skin Cancer.

Cureus
2024

The clinical spectrum associated with ERCC5 mutations: Is there a relationship between phenotype and genotype?

Pediatric discovery
2025

Imaging-Based High-Content Screening with Clickable Probes Identifies XPB Inhibitors.

Angewandte Chemie (International ed. in English)
2025

Newborn genome screening (the Generation Study): the advent of presymptomatic diagnosis for genodermatoses.

The British journal of dermatology
2025

Oroxylin A inhibits UVB-induced non-melanoma skin cancer by regulating XPA degradation.

Chinese journal of natural medicines
2025

A case of rapidly growing conjunctival squamous cell carcinoma in a 3-year-old child with xeroderma pigmentosum: a case report.

Journal of medical case reports
2025

EPHX1 and ERCC2 polymorphisms are associated with cisplatin-induced nephrotoxicity and prognosis in Thai cancer patients.

PloS one
2025

Pediatric Ocular Surface Squamous Neoplasia in the Absence of Known Risk Factors and Systemic Conditions: A Case Series.

Ocular oncology and pathology
2025

An unusual presentation of Huntington's disease-like syndrome in a patient with Xeroderma pigmentosum type F: Case report and review of the literature.

Clinical parkinsonism &amp; related disorders
2025

Role of alphaII-spectrin and XPF interaction in Hepatocellular carcinoma.

Gene
2025

Ebselen protects XPC deficient cells from H2O2 induced oxidative stress through a potentially mitohormetic mechanism.

Free radical biology &amp; medicine
2025

CHIR99021 Enhances the Proliferation of Mouse Female Germline Stem Cells Via Modulating ERCC2.

Reproductive sciences (Thousand Oaks, Calif.)
2025

Biological Models of Oxidative Purine DNA Damage in Neurodegenerative Disorders.

Antioxidants (Basel, Switzerland)
2025

Xeroderma Pigmentosam: Case Report of Siblings.

International journal of clinical pediatric dentistry
2025

Comprehensive Neuropsychological Assessment of Confirmed Xeroderma Pigmentosum a Variant with Neurological Manifestations: Case Report.

Archives of clinical neuropsychology : the official journal of the National Academy of Neuropsychologists
2025

Effective Sequential and Rotational Therapy in a Patient With Xeroderma Pigmentosum.

Dermatology practical &amp; conceptual
2025

Cigarette smoke and decreased DNA repair by Xeroderma Pigmentosum Group C use a double hit mechanism for epithelial cell lung carcinogenesis.

Oncotarget
2025

Distribution of genetic polymorphisms of nucleotide excision repair genes and risk of gastrointestinal cancer: Findings from a case-control study.

Indian journal of cancer
2025

XPD Regulates MIAT/miR-29a-3p/COL4A1 Axis to Impede Hepatocellular Carcinoma Development.

FASEB journal : official publication of the Federation of American Societies for Experimental Biology
2025

Effect of Repair Gene Polymorphism on the Risk of Malignant Neoplasm Development after Chronic Radiation Exposure.

Doklady. Biochemistry and biophysics
2025

Xeroderma pigmentosa associated with spindle cell carcinoma of lung: a case report.

Annals of medicine and surgery (2012)
2025

Treatment of corneoscleral mixed hemangioma by intrastromal lenticule transplantation in a case of xeroderma pigmentosum: a case report.

BMC ophthalmology
2025

Four germline POLH variants, including two found in skin tumors, impair DNA polymerase η function and cellular tolerance to UV radiation and cisplatin.

Chemico-biological interactions
2025

Amy and Friends: improving the lives of individuals affected by DNA repair disorders.

FEBS letters
2025

A Rare Case of Xeroderma Pigmentosum: Nivolumab Treatment for Three Cutaneous Malignancies with Clinical and Metabolic Imaging Correlation.

Diagnostics (Basel, Switzerland)
2025

CARM1/PRMT4 facilitates XPF-ERCC1 heterodimer assembly and maintains nucleotide excision repair activity.

Nucleic acids research
2025

Xeroderma pigmentosum type C with prominent cutaneous manifestations and subclinical neuroimaging abnormalities.

BMJ case reports
2025

Identification of a novel pathogenic XPC:c.2420 + 1 G>C variant in a patient with xeroderma pigmentosum.

DNA repair
2025

Thirteen-year-old Child Develops Squamous Cell Carcinoma Without Underlying Causes.

Plastic and reconstructive surgery. Global open
2025

The molecular landscape of hereditary ataxia: a single-center study.

Human genetics
2025

Exploring potential treatment opportunities in a head and neck tumor patient with AdCC: A novel germline ERCC2 mutation case report.

Medicine
2025

Transcription blocking properties and transcription-coupled repair of N2-alkylguanine adducts as a model for aldehyde-induced DNA damage.

The Journal of biological chemistry
2025

Circular RNA circ_0004470 accelerates the occurrence of lung cancer by promoting DNA damage and cell cycle arrest.

The Journal of biological chemistry
2025

Basal Cell Carcinoma in an Adolescent Male Child with Xeroderma Pigmentosum.

Indian dermatology online journal
2025

SLX4 and XPF are involved in cell migration and EMT in a cell-specific manner.

Biochemical pharmacology
2025

Germline variants in CDKN2A wild-type melanoma prone families.

Molecular oncology
2025

Association of XRCC1 (rs1799782) and XPD (rs13181) gene polymorphisms with renal failure risk in a sample of Iraqi population: a case-control study.

Molecular biology reports
2025

Immune checkpoint inhibitors for children with xeroderma pigmentosum and advanced cutaneous squamous cell carcinoma: A case presentation and brief review.

Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG
2025

Molecular insights into genodermatoses: Genetic findings from 43 patients.

Archives of dermatological research
2025

Nucleotide Excision Repair: Insights into Canonical and Emerging Functions of the Transcription/DNA Repair Factor TFIIH.

Genes
2025

Clinico-tomographic-pathological correlation in nodulo-ulcerative ocular surface squamous neoplasia: a study of 16 cases.

International ophthalmology
2025

Trichothiodystrophy due to ERCC2 Variants: Uncommon Contributor to Progressive Hypomyelinating Leukodystrophy.

Molecular genetics &amp; genomic medicine
2025

Association of ERCC2/XPD polymorphisms and the risk of head and neck carcinoma: a systematic review, meta-analysis, trial sequential analysis, network analysis, and functional effects.

BMC oral health
2025

Melatonin mitigates UV-induced tumorigenesis and suppresses hearing function deterioration in Xpa-deficient mice.

Journal of dermatological science
2025

Nano-delivery of a novel inhibitor of ERCC1-XPF for targeted sensitization of colorectal cancer to platinum-based chemotherapeutics.

Drug delivery and translational research
2025

Metastatic Squamous Cell Carcinoma Treated With Pembrolizumab in a Patient With Xeroderma Pigmentosum.

Pediatric dermatology
2025

Mutagenicity, DNA Repair Gene Polymorphism, and Differentially Expressed Plasma Protein Fractions Among Textile Dyeing Workers.

Journal of occupational and environmental medicine
2024

The Significance of the Response: Beyond the Mechanics of DNA Damage and Repair-Physiological, Genetic, and Systemic Aspects of Radiosensitivity in Higher Organisms.

International journal of molecular sciences
2024

Tualang Honey Has a Protective Effect Against Photodamage and Skin Cancer: An In Vivo Study.

Nutrients
2024

Beyond Nucleotide Excision Repair: The Importance of XPF in Base Excision Repair and Its Impact on Cancer, Inflammation, and Aging.

International journal of molecular sciences
2024

Unveiling Secondary Mutations in Blended Phenotypes: Dual ERCC4 and OTOA Pathogenic Variants Through WES Analysis.

International journal of molecular sciences
2025

Genetic signatures of ERCC1 and ERCC2 expression, along with SNPs variants, unveil favorable prognosis in SCLC patients undergoing platinum-based chemotherapy.

Oncology research
2025

Evaluation of Photoaging and Sun Protection Behavior in Children With Xeroderma Pigmentosum, Group C: A Prospective Analysis.

Pediatric dermatology
2024

Generation and characterization of CRISPR-Cas9-mediated XPC gene knockout in human skin cells.

Scientific reports
2025

Transcription-coupled repair - mechanisms of action, regulation, and associated human disorders.

FEBS letters
2025

The association of rs25487 of the XRCC1 gene and rs13181 of the ERCC2 gene polymorphisms with the ovarian cancer risk.

Biomolecules &amp; biomedicine
2025

A Case of Xeroderma Pigmentosum Variant Type With a Novel Mutation Diagnosed in Early Childhood.

Photodermatology, photoimmunology &amp; photomedicine
2024

Identification of novel variants of XPA and POLH/XPV genes in xeroderma pigmentosum patients in Vietnam.

Personalized medicine
2024

Beyond Huntington's Disease - Late-Onset Chorea Caused by a Homozygous Variant in ERCC4.

Cerebellum (London, England)
2024

Co-infection of HSV-1 amplicons containing the XPC gene and a human artificial chromosome vector into primary XPC deficient fibroblast cells.

Biochemistry and biophysics reports
2024

Different germline variants in the XPA gene are associated with severe, intermediate, or mild neurodegeneration in xeroderma pigmentosum patients.

PLoS genetics
2024

Synthetic rescue of Xeroderma Pigmentosum C phenotype via PIK3C3 downregulation.

Cell death &amp; disease
2024

The Role of Dermoscopy in Diagnosing Xeroderma Pigmentosum: A Practical Tool in Low-resource Settings.

Acta dermato-venereologica
2024

Contribution of ERCC2 rs13181 (Lys751Gln) and rs1799793 (Asp312Asn) polymorphisms to the risk of bladder cancer in Bangladesh.

Cancer genetics
2025

Frequent Occurrence of High-Risk Basal Cell Carcinoma in Xeroderma Pigmentosum: A Histopathological Insight From an Indian Cohort.

Journal of cutaneous pathology
2025

The synergy between alkylating agents and ERCC1-XPF inhibitors is p53 dependent.

Fundamental &amp; clinical pharmacology
2024

DNA Damage Response Alterations Predict for Neoadjuvant Chemotherapy Sensitivity in Muscle-Invasive Bladder Cancer: A Correlative Analysis of the SWOG S1314 Trial.

JCO precision oncology
2024

Curcumin derivative C210 induces Epstein-Barr virus lytic cycle and inhibits virion production by disrupting Hsp90 function.

Scientific reports
2024

Xeroderma pigmentosum protein XPD controls caspase-mediated stress responses.

Nature communications
2024

Case report: Neoadjuvant-intent pembrolizumab resulted in complete response in a xeroderma pigmentosum patient with locally advanced resectable cutaneous squamous cell carcinoma of the nose.

Frontiers in medicine
2024

Canonical and Non-Canonical Roles of Human DNA Polymerase η.

Genes
2024

Cutaneous Squamous Cell Carcinoma and Multiple Basal Cell Carcinomas in Xeroderma Pigmentosum-Variant Type Treated with Imiquimod 5% Cream and Radiotherapy: A Case Report.

Acta medica Philippina
2024

Understanding and managing locally advanced basal cell carcinoma: insights into pathogenesis, therapeutic strategies, and the role of hedgehog pathway inhibitors.

Italian journal of dermatology and venereology
2024

Genetic variations in NER pathway gene polymorphisms and Wilms tumor risk: A six-center case-control study in East China.

IUBMB life
2024

Xeroderma Pigmentosum Type C Primary Skin Fibroblasts Overexpress HGF and Promote Squamous Cell Carcinoma Invasion in the Absence of Genotoxic Stress.

Cancers
2025

A personalized and systematically designed adherence intervention improves photoprotection in adults with xeroderma pigmentosum (XP): results of the XPAND randomized controlled trial.

The British journal of dermatology
2024

Epidemiologic trends in cutaneous squamous cell carcinoma from 2011 to 2021 at All Africa Leprosy, Tuberculosis, and Rehabilitation Training Center (ALERT) in Addis Ababa, Ethiopia.

JAAD international
2025

The Distinct Roles of NEIL1 and XPA in Limiting Aflatoxin B1-Induced Mutagenesis in Mice.

Molecular cancer research : MCR
2024

Molecular architecture and functional dynamics of the pre-incision complex in nucleotide excision repair.

Nature communications
2024

Generation of XPA p.Arg228T mutant LUMCi004-A cell line for modeling Xeroderma pigmentosum group A.

Stem cell research
2025

Germline Variants in Patients Affected by Both Uveal and Cutaneous Melanoma.

Pigment cell &amp; melanoma research
2024

A Case of Severe Facial Swelling Mimicking Facial Nerve Paralysis after Cryotherapy to the Basal Cell Carcinoma in a Xeroderma Pigmentosum Patient.

Indian journal of dermatology
2024

Update on Recommendations for Cancer Screening and Surveillance in Children with Genomic Instability Disorders.

Clinical cancer research : an official journal of the American Association for Cancer Research
2024

Foveal Hypoplasia in Presumed Xeroderma Pigmentosum: A Case Report.

Beyoglu eye journal
2024

Treatment of ocular surface squamous neoplasia in an Indian rural facility: a study of 38 eyes.

BMC ophthalmology
2025

Synchronous multiple primary malignancies or transdifferentiation?-A diagnostic challenge in a case of Xeroderma pigmentosum.

Indian journal of pathology &amp; microbiology
2024

Bioinformatics analysis of ERCC family in pan-cancer and ERCC2 in bladder cancer.

Frontiers in immunology
2024

Cell cycle arrest combined with CDK1 inhibition suppresses genome-wide mutations by activating alternative DNA repair genes during genome editing.

The Journal of biological chemistry
2024

Genomic patterns of somatic mutations provide new prognostic, therapeutic, and biological insights in cancer.

Cell genomics
2024

Case report: Xeroderma pigmentosum Group A with erythropoietic protoporphyria in a young Chinese patient.

Frontiers in endocrinology
2024

Dermoscopy and In Vivo Confocal Microscopy Findings of Basal Cell Carcinomas in Xeroderma Pigmentosum Patients.

Indian journal of dermatology
2024

Xeroderma Pigmentosum with a Rapidly Proliferating Squamous Cell Carcinoma in a 4-Year Old Kid: A Rare Entity in Indian Subcontinent.

Journal of maxillofacial and oral surgery
2024

XRCC1 and XPD polymorphisms: clinical outcomes and risk of prostate cancer in Bangladeshi population.

Molecular biology reports
2024

Diseases with oral malignant potential: Need for change to inform research, policy, and practice.

Journal of oral pathology &amp; medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
2024

ERCC2 mutations alter the genomic distribution pattern of somatic mutations and are independently prognostic in bladder cancer.

Cell genomics
2024

Investigation of Polymorphisms in Global Genome Repair Genes in Patients With Ovarian Cancer in the Turkish Population.

Cancer control : journal of the Moffitt Cancer Center
2024

Sequential post-translational modifications regulate damaged DNA-binding protein DDB2 function.

Journal of biochemistry
2024

Does the XPA-FEN1 Interaction Concern to Nucleotide Excision Repair or Beyond?

Biomolecules
2024

Impaired B-cell function in ERCC2 deficiency.

Frontiers in immunology
2024

A novel algorithm for the virtual screening of extensive small molecule libraries against ERCC1/XPF protein-protein interaction for the identification of resistance-bypassing potential anticancer molecules.

Turkish journal of biology = Turk biyoloji dergisi
2024

"Immunopeeling" Using Imiquimod for Xeroderma Pigmentosum.

Indian dermatology online journal
2024

Skin Cancer Induction by the Antimycotic Drug Voriconazole Is Caused by Impaired DNA Damage Detection Due to Chromatin Compaction.

The Journal of investigative dermatology
2024

Revealing the UV response of melanocytes in xeroderma pigmentosum group A using patient-derived induced pluripotent stem cells.

Journal of dermatological science
2024

Protein-protein interactions in the core nucleotide excision repair pathway.

DNA repair
2024

Genome-wide analysis of transcription-coupled repair reveals novel transcription events in Caenorhabditis elegans.

PLoS genetics
2024

Correlative Analysis of ATM, RB1, ERCC2, and FANCC Mutations and Pathologic Complete Response After Neoadjuvant Chemotherapy in Patients with Muscle-invasive Bladder Cancer: Results from the SWOG S1314 Trial.

European urology
2024

Adult-Onset Neuropsychiatric Symptoms as the Presenting Feature of Xeroderma Pigmentosum Group G: A Report of a Rare Case.

Cureus
2024

A case of xeroderma pigmentosum group C with rare compound heterozygous mutations.

European journal of dermatology : EJD
2024

Diffuse large B-cell lymphoma with cutaneous involvement in a patient with xeroderma pigmentosum type C.

JAAD case reports
2024

The association between XPD rs13181 and rs1799793 polymorphism and oral cancer risk: evidence from a meta-analysis.

BMC cancer
2025

Faulty Gap Filling in Nucleotide Excision Repair Leads to Double-Strand Break Formation in Senescent Cells.

The Journal of investigative dermatology
2024

The Expression of PRAME as an Aid for Diagnosis and Evaluation of Histologic Margins of Intraepidermal Cutaneous Melanoma in Xeroderma Pigmentosum Patients.

Applied immunohistochemistry &amp; molecular morphology : AIMM
2024

Subungual mass in a patient of xeroderma pigmentosum: Looking beyond malignant transformation.

Indian journal of dermatology, venereology and leprology
2024

XPD stalled on cross-linked DNA provides insight into damage verification.

Nature structural &amp; molecular biology
2024

Insights from multi-omic modeling of neurodegeneration in xeroderma pigmentosum using an induced pluripotent stem cell system.

Cell reports
2024

XPC Protects against Carcinogen-Induced Histologic Progression to Lung Squamous Cell Carcinoma by Reduced Basal Epithelial Cell Proliferation.

Cancers
2024

DNA repair ability in a patient with voriconazole-related squamous cell carcinoma that required differential diagnosis from xeroderma pigmentosum.

Journal of dermatological science
2024

Persistent TFIIH binding to non-excised DNA damage causes cell and developmental failure.

Nature communications
2024

Impression cytology of ocular surface in xeroderma pigmentosum.

Arquivos brasileiros de oftalmologia
2024

DNA Repair Genetics and the Risk of Radiation Pneumonitis in Patients With Lung Cancer: A Systematic Review and Meta-analysis.

Clinical oncology (Royal College of Radiologists (Great Britain))
2024

Clinical prognostic significance of xeroderma pigmentosum group C and IFN‑γ in non‑small cell lung cancer.

Oncology letters
2024

Two siblings with Fanconi anemia (FANCQ, ERCC4/XPF) presenting with tumor-mimicking lesions in the brain and acute neurological deterioration.

Pediatric blood &amp; cancer
2024

Evidence for persistent UV-induced DNA damage and altered DNA damage response in xeroderma pigmentosa patient corneas.

Experimental eye research
2024

PERIOCULAR HIGH RISK BCCS AFTER ADDITIONAL/PARALLEL INTAKE OF TORASEMIDE, MOXONIDINE AND MIRABEGRON: IMPORTANT LINKS TO SKIN CANCER RELATED (PHOTO-) NITROSOGENESIS IN THE CONTEXT OF PHARMACO-ONCOGENESIS.

Georgian medical news
2024

DMC-siERCC2 hybrid nanoparticle enhances TRAIL sensitivity by inducing cell cycle arrest for glioblastoma treatment.

Biomedicine &amp; pharmacotherapy = Biomedecine &amp; pharmacotherapie
2024

Proteome characterization of XPC-deficient melanocytes generated by CRISPR-Cas9 technology reveals alteration in the expression of several hundred proteins.

Journal of dermatological science
2024

Relationship between XPA, XPB/ERCC3, XPF/ERCC4, and XPG/ERCC5 Polymorphisms and the Susceptibility to Head and Neck Carcinoma: A Systematic Review, Meta-Analysis, and Trial Sequential Analysis.

Medicina (Kaunas, Lithuania)
2024

p53 regulates diverse tissue-specific outcomes to endogenous DNA damage in mice.

Nature communications
2024

Complex Genomic Rearrangement Patterns in Malignant Pleural Mesothelioma due to Environmental Asbestos Exposure.

Journal of environmental pathology, toxicology and oncology : official organ of the International Society for Environmental Toxicology and Cancer
2024

XPA tumor variant leads to defects in NER that sensitize cells to cisplatin.

NAR cancer
2024

Evaluation of Meibomian gland dysfunction using meibography in patients with xeroderma pigmentosum.

Arquivos brasileiros de oftalmologia
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Comunidades

Grupos ativos de quem convive com esta doença aqui no Raras

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Doenças relacionadas

Doenças com sintomas parecidos — ajudam quem ainda está buscando diagnóstico

Referências e fontes

Bases de dados externas citadas neste artigo

Publicações científicas

Artigos indexados no PubMed ligados a esta doença no grafo RarasNet — título, periódico e PMID direto da fonte, sem intermediação de IA.

  1. DNA repair-associated nucleases induce double-strand breaks following sequential exposure to UVA1 and UVB.
    Photochemical &amp; photobiological sciences : Official journal of the European Photochemistry Association and the European Society for Photobiology· 2026· PMID 41817842mais citado
  2. Pre-incision structures reveal principles of DNA nucleotide excision repair.
    Nature· 2026· PMID 41673165mais citado
  3. Unusual Disease-Progression in Two Siblings With Xeroderma Pigmentosum Group G.
    Clinical genetics· 2026· PMID 41482699mais citado
  4. Association of XPC rs2228001, ESR2 rs1256030, and rs4986938 Gene Polymorphisms With Breast Cancer Risk in Bangladeshi Women: A Case-Control Study.
    Clinical breast cancer· 2026· PMID 41864051mais citado
  5. XAB2: a link between RNA metabolism, DNA damage repair, and human health.
    Transcription· 2026· PMID 41848795mais citado
  6. Clinicopathological and molecular characterization of tumor-associated macrophages in sporadic and Xeroderma Pigmentosum-related cutaneous melanoma.
    BMC Cancer· 2026· PMID 41963845recente
  7. Successful Interstitial HDR Brachytherapy for Nasal Skin SCC in Xeroderma Pigmentosum: An Eight-Year Follow-Up Case Report.
    Clin Case Rep· 2026· PMID 41958690recente
  8. High cancer-associated mutational burden in normal blood of xeroderma pigmentosum group C, but not groups A, D, or F.
    Blood Neoplasia· 2026· PMID 41938744recente
  9. Th17 cells require the DNA repair sensor xeroderma pigmentosum complementation Group C to control oxidative DNA damage in a murine model.
    Nat Commun· 2026· PMID 41922349recente
  10. Rare internal malignancies in xeroderma pigmentosum: A report of two cases from Tunisia and analysis of driver mutations.
    Cancer Pathog Ther· 2026· PMID 41908666recente

Bases de dados e fontes oficiais

Identificadores e referências canônicas usadas para montar este verbete.

  1. ORPHA:910(Orphanet)
  2. MONDO:0019600(MONDO)
  3. GARD:7910(GARD (NIH))
  4. Variantes catalogadas(ClinVar)
  5. Busca completa no PubMed(PubMed)
  6. Artigo Wikipedia(Wikipedia)
  7. Q612693(Wikidata)

Dados compilados pelo RarasNet a partir de fontes abertas (Orphanet, OMIM, MONDO, PubMed/EuropePMC, ClinicalTrials.gov, DATASUS, PCDT/MS). Este conteúdo é informativo e não substitui avaliação médica.

Conteúdo mantido por Agente Raras · Médicos e pesquisadores podem colaborar

Xeroderma pigmentoso
Compêndio · Raras BR

Xeroderma pigmentoso

ORPHA:910 · MONDO:0019600
Prevalência
1-9 / 1 000 000
Herança
Autosomal recessive
CID-10
Q82.1 · Xeroderma pigmentoso
CID-11
Ensaios
2 ativos
Início
All ages
Prevalência
0.1 (United States)
MedGen
UMLS
C0043346
EuropePMC
Wikidata
Wikipedia
Papers 10a
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