Adverse events associated with drug-related thiamine deficiency: A pharmacovigilance study.

Thiamine (Vitamin B1) metabolism in schistosomes.

Thiamine Metabolism in Microbes and Strategies for Biomanufacturing.

Genetic dissection of stool frequency implicates vitamin B1 metabolism and other actionable pathways in the modulation of gut motility.

A Rare Familial Case of Pseudohypoparathyroidism Type 1b in Two Brothers Presenting With Recurrent Leg Cramps and Learning Difficulties.

Ameliorative Effects of Vitamin E and Lutein on Hydrogen Peroxide-Triggered Oxidative Cytotoxicity via Combined Transcriptome and Metabolome Analysis.

Vitamin A acts as a potent suppressor of selenoprotein P with potential relevance for multivitamin supplementation.

Targeting mitochondrial transporters and metabolic reprogramming for disease treatment.

Effect of henagliflozin on aging biomarkers in patients with type 2 diabetes: A multicenter, randomized, double-blind, placebo-controlled study.

Exploring neuropsychiatric manifestations of vitamin B complex deficiencies.

Structure-based discovery of thiamine uptake inhibitors.

Structures of Listeria monocytogenes MenD in ThDP-bound and in-crystallo captured intermediate I-bound forms.

Investigating prolactin-induced protein and its role in modulating the metabolic state of the keratoconus microenvironment.

The role of SLC19A2 variants in the wide spectrum of non-autoimmune abnormalities of glucose homeostasis.

A Systems Hypothesis of Lipopolysaccharide-Induced Vitamin Transport Suppression and Metabolic Reprogramming in Autism Spectrum Disorders: An Open Call for Validation and Therapeutic Translation.

Thiamin deficiency in children with chronic kidney disease on peritoneal dialysis and its association with dialysis duration and transport peritoneal membrane status.

Metabolomic Profiling of Aqueous Humor From Glaucoma Patients Identifies Metabolites With Anti-Inflammatory and Neuroprotective Potential in Mice.

Elevated thiamine level is associated with activating interaction between HIF-1α and SLC19A3 in experimental myopic guinea pigs.

Thiamine, gastrointestinal beriberi and acetylcholine signaling.

Genetic Variants of the Human Thiamine Transporter (SLC19A3, THTR2)-Potential Relevance in Metabolic Diseases.

Combined Administration of Metformin and Amprolium to Rats Affects Metabolism of Free Amino Acids in the Brain, Altering Behavior, and Heart Rate.

Research on the Therapeutic Effect of Qizhu Anti Cancer Recipe on Colorectal Cancer Based on RNA Sequencing Analysis.

Thiamine-Responsive Megaloblastic Anemia Syndrome Mimicking Myelodysplastic Neoplasm.

IQGAP-2: a novel interacting partner with the human colonic thiamin pyrophosphate transporter.

Structural basis of thiamine transport and drug recognition by SLC19A3.

Bacterial lipopolysaccharide inhibits free thiamin uptake along the intestinal tract via interference with membrane expression of thiamin transporters 1 and 2.

Rare Diseases Linked to Mutations in Vitamin Transporters Expressed in the Human Blood-Brain Barrier.

Phospholipid scrambling induced by an ion channel/metabolite transporter complex.

Integration of network pharmacology, UHPLC-Q exactive orbitrap HRMS technique and metabolomics to elucidate the active ingredients and mechanisms of compound danshen pills in treating hypercholesterolemic rats.

Role of gut/liver metabolites and gut microbiota in liver fibrosis caused by cholestasis.

Exploring the Therapeutic Potential of Benfotiamine in a Sporadic Alzheimer's-Like Disease Rat Model: Insights into Insulin Signaling and Cognitive function.

SLC25A19 is required for NADH homeostasis and mitochondrial respiration.

Biotin Homeostasis and Human Disorders: Recent Findings and Perspectives.

Pan-Genome Analysis of Wolbachia, Endosymbiont of Diaphorina citri, Reveals Independent Origin in Asia and North America.

Vitamin B1 deficiency leads to high oxidative stress and mtDNA depletion caused by SLC19A3 mutation in consanguineous family with Leigh syndrome.

Goldenseal-Mediated Inhibition of Intestinal Uptake Transporters Decreases Metformin Systemic Exposure in Mice.

Improvement effect of biochar on soil microbial community structure and metabolites of decline disease bayberry.

Acute Encephalopathy Caused by Inherited Metabolic Diseases.

The Influence of Alcohol Consumption on Intestinal Nutrient Absorption: A Comprehensive Review.

Integrated analysis of metabolomic and transcriptomic profiling reveals the effect of Buyang Huanwu decoction on Parkinson's disease in mice.

Beyond the caudate nucleus: Early atypical neuroimaging findings in biotin-thiamine- responsive basal ganglia disease.

Case report: biotin-thiamine-responsive basal ganglia disease with severe subdural hematoma on magnetic resonance imaging.

Alzheimer's disease is associated with disruption in thiamin transport physiology: A potential role for neuroinflammation.

Biotin-Responsive Basal Ganglia Disease: Treatable Metabolic Disorder with SLC19A3 Mutation Presenting as Rapidly Progressive Dementia.

Genes Differentially Expressed by Haemophilus ducreyi during Anaerobic Growth Significantly Overlap Those Differentially Expressed during Experimental Infection of Human Volunteers.

Mitochondrial transport and metabolism of the vitamin B-derived cofactors thiamine pyrophosphate, coenzyme A, FAD and NAD+ , and related diseases: A review.

Clinical and genetic studies of thiamine metabolism dysfunction syndrome-4: case series and review of the literature.

Biotin-thiamine-responsive basal ganglia disease: A case report.

Biotin-thiamine responsive basal ganglia disease in the era of COVID-19 outbreak diagnosis not to be missed: A case report.

SLC gene mutations and pediatric neurological disorders: diverse clinical phenotypes in a Saudi Arabian population.

Early treatment of biotin-thiamine-responsive basal ganglia disease improves the prognosis.

Identification and functional analysis of novel SLC25A19 variants causing thiamine metabolism dysfunction syndrome 4.

CPEB alteration and aberrant transcriptome-polyadenylation lead to a treatable SLC19A3 deficiency in Huntington's disease.

The Effects of Genetic Mutations and Drugs on the Activity of the Thiamine Transporter, SLC19A2.

Magnetic resonance imaging pattern recognition in childhood bilateral basal ganglia disorders.

SLC25A19 deficiency and bilateral striatal necrosis with polyneuropathy: a new case and review of the literature.

Region-selective permeability of the blood-brain barrier to α-aminoisobutyric acid during thiamine deficiency and following its reversal.

Fursultiamine Alleviates Choroidal Neovascularization by Suppressing Inflammation and Metabolic Reprogramming.

Unusual case of biotin-thiamine responsive encephalopathy without basal ganglia involvement.

Eleven novel mutations and clinical characteristics in seven Chinese patients with thiamine metabolism dysfunction syndrome.

3'-UTR Polymorphisms of Vitamin B-Related Genes Are Associated with Osteoporosis and Osteoporotic Vertebral Compression Fractures (OVCFs) in Postmenopausal Women.

G6036A substitution in mitochondrial COX I gene compromises cytochrome c oxidase activity in thiamine responsive Leigh syndrome patients.

Novel p.P298L SURF1 mutation in thiamine deficient Leigh syndrome patients compromises cytochrome c oxidase activity.

Neonatal form of biotin-thiamine-responsive basal ganglia disease. Clues to diagnosis.

Organic Cation Transporters in Health and Disease.

Serum Metabolomics Reveals Personalized Metabolic Patterns for Macular Neovascular Disease Patient Stratification.

News and views in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS): The role of co-morbidity and novel treatments.

Functional analysis of the third identified SLC25A19 mutation causative for the thiamine metabolism dysfunction syndrome 4.

Citreoviridin induces myocardial apoptosis through PPAR-γ-mTORC2-mediated autophagic pathway and the protective effect of thiamine and selenium.

Aggravated effects of coexisting marginal thiamine deficits and zinc excess on SN56 neuronal cells.

TRMA syndrome with a severe phenotype, cerebral infarction, and novel compound heterozygous SLC19A2 mutation: a case report.

Bilateral Striatal Necrosis with Polyneuropathy with a Novel SLC25A19 (Mitochondrial Thiamine Pyrophosphate Carrier OMIMI*606521) Mutation: Treatable Thiamine Metabolic Disorder-A Report of Two Indian Cases.

Thiamine deficiency affects glucose transport and β-oxidation in rats.

Whole exome sequencing identifies a new mutation in the SLC19A2 gene leading to thiamine-responsive megaloblastic anemia in an Egyptian family.

Genetic defects of thiamine transport and metabolism: A review of clinical phenotypes, genetics, and functional studies.

Neurological, Psychiatric, and Biochemical Aspects of Thiamine Deficiency in Children and Adults.

Reduced Folate Carrier: an Entry Receptor for a Novel Feline Leukemia Virus Variant.

Effects of Chronic Voluntary Alcohol Drinking on Thiamine Concentrations, Endoplasmic Reticulum Stress, and Oxidative Stress in the Brain of Crossed High Alcohol Preferring Mice.

Comprehensive proteomic and metabolomic profiling of mcr-1-mediated colistin resistance in Escherichia coli.

Symposium review: The importance of the ruminal epithelial barrier for a healthy and productive cow.

New molecular aspects in the mechanism of oromaxillofacial cleft prevention by B-vitamins.

Thiamine Responsive Megaloblastic Anaemia, Diabetes Mellitus and Sensorineural Hearing Loss in a Child.

A Novel Mutation of SLC19A2 in a Chinese Zhuang Ethnic Family with Thiamine-Responsive Megaloblastic Anemia.

Organic cation transporter 1 (OCT1) modulates multiple cardiometabolic traits through effects on hepatic thiamine content.

PET Imaging Analysis of Vitamin B1 Kinetics with [11C]Thiamine and its Derivative [11C]Thiamine Tetrahydrofurfuryl Disulfide in Rats.

[Defect of thiamine transport and activation and related disease].

SLC19A3 related disorder: Treatment implication and clinical outcome of 2 new patients.

Case 38-2017. A 20-Year-Old Woman with Seizures and Progressive Dystonia.

Impairment of Thiamine Transport at the GUT-BBB-AXIS Contributes to Wernicke's Encephalopathy.

Biotin Thiamin Responsive Basal Ganglia Disease in Siblings.

Reduced activities of thiamine-dependent and cytochrome c oxidase enzymes in cerebral cortex of cattle affected by sulfur-induced polioencephalomalacia.

Role of thiamine in Huntington's disease pathogenesis: In vitro studies.

SLC19A3 Gene Defects Sorting the Phenotype and Acronyms: Review.

Thiamine Acquisition Strategies Impact Metabolism and Competition in the Gut Microbe Bacteroides thetaiotaomicron.

Transketolase Activity but not Thiamine Membrane Transport Change in Response to Hyperglycaemia and Kidney Dysfunction.

Therapies for mitochondrial diseases and current clinical trials.

Thiamine metabolism is critical for regulating correlated growth of dendrite arbors and neuronal somata.

Compound heterozygous SLC19A3 mutations further refine the critical promoter region for biotin-thiamine-responsive basal ganglia disease.

High-dose thiamine prevents brain lesions and prolongs survival of Slc19a3-deficient mice.

Alcohol and thiamine deficiency trigger differential mitochondrial transition pore opening mediating cellular death.

Biotin-thiamine responsive basal ganglia disease: Identification of a pyruvate peak on brain spectroscopy, novel mutation in SLC19A3, and calculation of prevalence based on allele frequencies from aggregated next-generation sequencing data.

Treatable Inborn Errors of Metabolism Due to Membrane Vitamin Transporters Deficiency.

Biochemical Analyses of Cerebrospinal Fluid for the Diagnosis of Neurometabolic Conditions. What Can We Expect?

Treatment of rats with the JAK-2 inhibitor fedratinib does not lead to experimental Wernicke's encephalopathy.

Brain endothelial dysfunction following pyrithiamine induced thiamine deficiency in the rat.

Lyme disease spirochaete Borrelia burgdorferi does not require thiamin.

Distinct metabolic network states manifest in the gene expression profiles of pediatric inflammatory bowel disease patients and controls.

Treatment of genetic defects of thiamine transport and metabolism.

Thiamine Deficiency Increases Ca2+ Current and CaV1.2 L-type Ca2+ Channel Levels in Cerebellum Granular Neurons.

VdThit, a Thiamine Transport Protein, Is Required for Pathogenicity of the Vascular Pathogen Verticillium dahliae.

Neurological Disorders Associated with Striatal Lesions: Classification and Diagnostic Approach.

Inhibition of Intestinal Thiamin Transport in Rat Model of Sepsis.

The compact genome of the plant pathogen Plasmodiophora brassicae is adapted to intracellular interactions with host Brassica spp.

Evaluation of Th1/Th2-Related Immune Response against Recombinant Proteins of Brucella abortus Infection in Mice.

Biotin and Thiamine Responsive Basal Ganglia Disease--A vital differential diagnosis in infants with severe encephalopathy.

Novel SLC19A3 Promoter Deletion and Allelic Silencing in Biotin-Thiamine-Responsive Basal Ganglia Encephalopathy.

Identification of critical variants within SLC44A4, an ulcerative colitis susceptibility gene identified in a GWAS in north Indians.

Variation in SLC19A3 and Protection From Microvascular Damage in Type 1 Diabetes.

Free-thiamine is a potential biomarker of thiamine transporter-2 deficiency: a treatable cause of Leigh syndrome.

Extensive Modulation of the Fecal Metagenome in Children With Crohn's Disease During Exclusive Enteral Nutrition.

Endoplasmic Reticulum Stress and Ethanol Neurotoxicity.

Sudden infant death syndrome and abnormal metabolism of thiamin.

Chronic alcohol exposure affects pancreatic acinar mitochondrial thiamin pyrophosphate uptake: studies with mouse 266-6 cell line and primary cells.

Neuromyelitis optica spectrum disorders may be misdiagnosed as Wernicke's encephalopathy.

A Therapeutic Fast for Lymphoma Resulting in Wernicke Encephalopathy.

Synthesis, characterization, and efficacy evaluation of a new anti-diabetic vanadyl(II) thiamine hydrochloride complex in streptozotocin-induced diabetic rats.

Spatial cognitive deficits in an animal model of Wernicke-Korsakoff syndrome are related to changes in thalamic VDAC protein concentrations.

PKR downregulation prevents neurodegeneration and β-amyloid production in a thiamine-deficient model.