Raras
Buscar doenças, sintomas, genes...
Paralisia periódica
ORPHA:206976CID-10 · G72.3CID-11 · 8C74.1DOENÇA RARA

A doença de Parkinson (DP) é uma doença degenerativa crónica do sistema nervoso central que afeta principalmente a coordenação motora. Os sintomas vão-se manifestando de forma lenta e gradual ao longo do tempo. Na fase inicial da doença, os sintomas mais óbvios são tremores, rigidez, lentidão de movimentos e dificuldade em caminhar. Podem também ocorrer problemas de raciocínio e comportamentais. Nos estádios avançados da doença é comum a presença de demência. Cerca de 30% das pessoas manifestam depressão e ansiedade.

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Introdução

O que você precisa saber de cara

📋

Doença neuromuscular rara caracterizada por episódios de fraqueza muscular flácida, podendo apresentar face triangular, retenção urinária e constipação. Associada a alterações eletrolíticas e cardíacas, como hipofosfatemia transitória e bloqueio atrioventricular.

Pesquisas ativas
2 ensaios
11 total registrados no ClinicalTrials.gov
Publicações científicas
2.530 artigos
Último publicado: 2026 Mar
🏥
SUS: Cobertura mínimaScore: 15%
CID-10: G72.3
🇧🇷Dados SUS / DATASUS
PROCEDIMENTOS SIGTAP (2)
0202010694
Sequenciamento completo do exoma (WES)genetic_test
0301070040
Atendimento em reabilitação — doenças rarasrehabilitation
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Sinais e sintomas

O que aparece no corpo e com que frequência cada sintoma acontece

Partes do corpo afetadas

💪
Músculos
18 sintomas
😀
Face
15 sintomas
🦴
Ossos e articulações
12 sintomas
❤️
Coração
11 sintomas
🧠
Neurológico
8 sintomas
📏
Crescimento
8 sintomas

+ 53 sintomas em outras categorias

Características mais comuns

Face triangular
Retenção urinária
Constipação
Doença de Graves
Hipofosfatemia transitória
Hiperidrose
142sintomas
Sem dados (142)

Os sintomas variam de pessoa para pessoa. Abaixo estão as 142 características clínicas mais associadas, ordenadas por frequência.

Face triangularTriangular face
Retenção urináriaUrinary retention
ConstipaçãoConstipation
Doença de GravesGraves disease
Hipofosfatemia transitóriaTransient hypophosphatemia

Linha do tempo da pesquisa

Publicações por ano — veja quando o interesse científico cresceu
Anos de pesquisa1desde 2026
Total histórico2.530PubMed
Últimos 10 anos200publicações
Pico202580 papers
Linha do tempo
2026Hoje · 2026🧪 1992Primeiro ensaio clínico📈 2025Ano de pico
Publicações por ano (últimos 10 anos)

Encontrou um erro ou informação desatualizada? Sugira uma correção →

Genética e causas

O que está alterado no DNA e como passa nas famílias

Genes associados

9 genes identificados com associação a esta condição.

KCNJ2Inward rectifier potassium channel 2Disease-causing germline mutation(s) inModerado
FUNÇÃO

Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it (PubMed:36149965, PubMed:7590287, PubMed:9490857). Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages (PubMed:7590287, PubMed:7696590). The inward rectification is mainly due to the blockage of outward current by inter

LOCALIZAÇÃO

Cell membraneCell membrane, sarcolemma, T-tubule

VIAS BIOLÓGICAS (5)
Activation of G protein gated Potassium channelsInhibition of voltage gated Ca2+ channels via Gbeta/gamma subunitsClassical Kir channelsPhase 4 - resting membrane potentialSensory perception of sour taste
MECANISMO DE DOENÇA

Long QT syndrome 7

A heart disorder characterized by a prolonged QT interval on the ECG and polymorphic ventricular arrhythmias. They cause syncope and sudden death in response to exercise or emotional stress, and can present with a sentinel event of sudden cardiac death in infancy. Long QT syndrome type 7 manifests itself as a clinical triad consisting of potassium-sensitive periodic paralysis, ventricular ectopy and dysmorphic features.

EXPRESSÃO TECIDUAL(Ubíquo)
Baço
10.1 TPM
Brain Spinal cord cervical c-1
9.1 TPM
Sangue
6.5 TPM
Coração - Ventrículo esquerdo
5.5 TPM
Mama
5.1 TPM
OUTRAS DOENÇAS (5)
short QT syndrome type 3Andersen-Tawil syndromeatrial fibrillation, familial, 9short QT syndrome
HGNC:6263UniProt:P63252
KCNJ5G protein-activated inward rectifier potassium channel 4Candidate gene tested inTolerante
FUNÇÃO

Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium. This potassium channel is control

LOCALIZAÇÃO

Membrane

VIAS BIOLÓGICAS (2)
Activation of G protein gated Potassium channelsInhibition of voltage gated Ca2+ channels via Gbeta/gamma subunits
MECANISMO DE DOENÇA

Long QT syndrome 13

A heart disorder characterized by a prolonged QT interval on the ECG and polymorphic ventricular arrhythmias. They cause syncope and sudden death in response to exercise or emotional stress, and can present with a sentinel event of sudden cardiac death in infancy.

EXPRESSÃO TECIDUAL(Tecido-específico)
Glândula adrenal
62.9 TPM
Pituitária
25.4 TPM
Baço
10.2 TPM
Pâncreas
8.2 TPM
Rim - Medula
6.3 TPM
OUTRAS DOENÇAS (5)
familial hyperaldosteronism type IIIlong QT syndrome 13familial atrial fibrillationAndersen-Tawil syndrome
HGNC:6266UniProt:P48544
KCNJ18Inward rectifier potassium channel 18Candidate gene tested inTolerante
FUNÇÃO

Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium

LOCALIZAÇÃO

Cell membraneEndoplasmic reticulum

MECANISMO DE DOENÇA

Thyrotoxic periodic paralysis 2

A sporadic muscular disorder characterized by episodic weakness and hypokalemia during a thyrotoxic state. It is clinically similar to hereditary hypokalemic periodic paralysis, except for the fact that hyperthyroidism is an absolute requirement for disease manifestation. The disease presents with recurrent episodes of acute muscular weakness of the four extremities that vary in severity from paresis to complete paralysis. Attacks are triggered by ingestion of a high carbohydrate load or strenuous physical activity followed by a period of rest. Thyrotoxic periodic paralysis can occur in association with any cause of hyperthyroidism, but is most commonly associated with Graves disease.

EXPRESSÃO TECIDUAL(Tecido-específico)
Skin Not Sun Exposed Suprapubic
10.2 TPM
Skin Sun Exposed Lower leg
8.5 TPM
Testículo
1.2 TPM
Cérebro - Hemisfério cerebelar
0.1 TPM
Cerebelo
0.1 TPM
OUTRAS DOENÇAS (2)
thyrotoxic periodic paralysisthyrotoxic periodic paralysis, susceptibility to, 2
HGNC:39080UniProt:B7U540
KCNE3Potassium voltage-gated channel subfamily E member 3Candidate gene tested inModerado
FUNÇÃO

Ancillary protein that functions as a regulatory subunit of the voltage-gated potassium (Kv) channel complex composed of pore-forming and potassium-conducting alpha subunits and of regulatory beta subunits. KCNE3 beta subunit modulates the gating kinetics and enhances stability of the channel complex (PubMed:10646604, PubMed:11207363, PubMed:12954870). Alters the gating of the delayed rectifier Kv channel containing KCNB1 alpha subunit (PubMed:12954870). Associates with KCNC4/Kv3.4 alpha subunit

LOCALIZAÇÃO

Cell membraneCytoplasmPerikaryonCell projection, dendriteMembrane raft

VIAS BIOLÓGICAS (2)
Phase 3 - rapid repolarisationPhase 2 - plateau phase
MECANISMO DE DOENÇA

Brugada syndrome 6

A tachyarrhythmia characterized by right bundle branch block and ST segment elevation on an electrocardiogram (ECG). It can cause the ventricles to beat so fast that the blood is prevented from circulating efficiently in the body. When this situation occurs, the individual will faint and may die in a few minutes if the heart is not reset.

EXPRESSÃO TECIDUAL(Ubíquo)
Sangue
18.2 TPM
Intestino delgado
12.5 TPM
Baço
11.1 TPM
Ovário
10.2 TPM
Glândula salivar
10.1 TPM
OUTRAS DOENÇAS (3)
Brugada syndrome 6Brugada syndromehypokalemic periodic paralysis
HGNC:6243UniProt:Q9Y6H6
GABRA3Gamma-aminobutyric acid receptor subunit alpha-3Candidate gene tested inRestrito
FUNÇÃO

Alpha subunit of the heteropentameric ligand-gated chloride channel gated by gamma-aminobutyric acid (GABA), a major inhibitory neurotransmitter in the brain (PubMed:16412217, PubMed:29053855). GABA-gated chloride channels, also named GABA(A) receptors (GABAAR), consist of five subunits arranged around a central pore and contain GABA active binding site(s) located at the alpha and beta subunit interface(s) (By similarity). When activated by GABA, GABAARs selectively allow the flow of chloride an

LOCALIZAÇÃO

Postsynaptic cell membraneCell membrane

VIAS BIOLÓGICAS (1)
GABA receptor activation
MECANISMO DE DOENÇA

Epilepsy, X-linked 2, with or without impaired intellectual development and dysmorphic features

A neurologic disorder characterized by variable combinations of epileptic seizure, and a varying degree of intellectual disability and developmental delay. Some patients have dysmorphic facial features or mild skeletal anomalies. In general, males are more severely affected than females, although there is evidence for incomplete penetrance in both sexes.

VIAS REACTOME (1)
EXPRESSÃO TECIDUAL(Tecido-específico)
Brain Frontal Cortex BA9
20.7 TPM
Brain Anterior cingulate cortex BA24
13.6 TPM
Hipotálamo
13.3 TPM
Córtex cerebral
12.6 TPM
Brain Nucleus accumbens basal ganglia
6.9 TPM
OUTRAS DOENÇAS (2)
epilepsy, X-linked 2, with or without impaired intellectual development and dysmorphic featuresthyrotoxic periodic paralysis
HGNC:4077UniProt:P34903
MT-ATP6ATP synthase F(0) complex subunit aCandidate gene tested inDesconhecido
FUNÇÃO

Subunit a, of the mitochondrial membrane ATP synthase complex (F(1)F(0) ATP synthase or Complex V) that produces ATP from ADP in the presence of a proton gradient across the membrane which is generated by electron transport complexes of the respiratory chain (Probable). ATP synthase complex consist of a soluble F(1) head domain - the catalytic core - and a membrane F(1) domain - the membrane proton channel (PubMed:37244256). These two domains are linked by a central stalk rotating inside the F(1

LOCALIZAÇÃO

Mitochondrion inner membrane

VIAS BIOLÓGICAS (4)
Mitochondrial translation terminationFormation of ATP by chemiosmotic couplingCristae formationMitochondrial protein degradation
MECANISMO DE DOENÇA

Neuropathy, ataxia, and retinitis pigmentosa

A syndrome characterized by variable combination of developmental delay, psychomotor retardation, hearing loss, optic atrophy and retinitis pigmentosa, dementia, seizures, ataxia, proximal neurogenic muscle weakness, and sensory neuropathy.

OUTRAS DOENÇAS (8)
mitochondrial diseasematernally-inherited Leigh syndromefamilial infantile bilateral striatal necrosismitochondrial proton-transporting ATP synthase complex deficiency
HGNC:7414UniProt:P00846
MT-ATP8ATP synthase F(0) complex subunit 8Candidate gene tested inDesconhecido
FUNÇÃO

Subunit 8, of the mitochondrial membrane ATP synthase complex (F(1)F(0) ATP synthase or Complex V) that produces ATP from ADP in the presence of a proton gradient across the membrane which is generated by electron transport complexes of the respiratory chain (PubMed:37244256). ATP synthase complex consist of a soluble F(1) head domain - the catalytic core - and a membrane F(1) domain - the membrane proton channel (PubMed:37244256). These two domains are linked by a central stalk rotating inside

LOCALIZAÇÃO

Mitochondrion membrane

VIAS BIOLÓGICAS (3)
Mitochondrial translation terminationFormation of ATP by chemiosmotic couplingCristae formation
MECANISMO DE DOENÇA

Mitochondrial complex V deficiency, mitochondrial 2

A mitochondrial disorder with heterogeneous clinical manifestations including neuropathy, ataxia, hypertrophic cardiomyopathy. Hypertrophic cardiomyopathy can present with negligible to extreme hypertrophy, minimal to extensive fibrosis and myocyte disarray, absent to severe left ventricular outflow tract obstruction, and distinct septal contours/morphologies with extremely varying clinical course.

OUTRAS DOENÇAS (4)
mitochondrial diseaseperiodic paralysis with later-onset distal motor neuropathymitochondrial proton-transporting ATP synthase complex deficiencyKearns-Sayre syndrome
HGNC:7415UniProt:P03928
CACNA1SVoltage-dependent L-type calcium channel subunit alpha-1SDisease-causing germline mutation(s) inTolerante
FUNÇÃO

Pore-forming, alpha-1S subunit of the voltage-gated calcium channel that gives rise to L-type calcium currents in skeletal muscle. Calcium channels containing the alpha-1S subunit play an important role in excitation-contraction coupling in skeletal muscle via their interaction with RYR1, which triggers Ca(2+) release from the sarcoplasmic reticulum and ultimately results in muscle contraction. Long-lasting (L-type) calcium channels belong to the 'high-voltage activated' (HVA) group

LOCALIZAÇÃO

Cell membrane, sarcolemma, T-tubule

VIAS BIOLÓGICAS (1)
NCAM1 interactions
MECANISMO DE DOENÇA

Periodic paralysis hypokalemic 1

An autosomal dominant disorder manifested by episodic flaccid generalized muscle weakness associated with falls of serum potassium levels.

VIAS REACTOME (1)
OUTRAS DOENÇAS (8)
congenital myopathy 18hypokalemic periodic paralysis, type 1malignant hyperthermia of anesthesiaobsolete periodic paralysis with transient compartment-like syndrome
HGNC:1397UniProt:Q13698
SCN4ASodium channel protein type 4 subunit alphaDisease-causing germline mutation(s) inTolerante
FUNÇÃO

Pore-forming subunit of Nav1.4, a voltage-gated sodium (Nav) channel that directly mediates the depolarizing phase of action potentials in excitable membranes. Navs, also called VGSCs (voltage-gated sodium channels) or VDSCs (voltage-dependent sodium channels), operate by switching between closed and open conformations depending on the voltage difference across the membrane. In the open conformation they allow Na(+) ions to selectively pass through the pore, along their electrochemical gradient.

LOCALIZAÇÃO

Cell membrane

VIAS BIOLÓGICAS (2)
Interaction between L1 and AnkyrinsPhase 0 - rapid depolarisation
MECANISMO DE DOENÇA

Paramyotonia congenita

An autosomal dominant channelopathy characterized by myotonia, increased by exposure to cold, intermittent flaccid paresis, not necessarily dependent on cold or myotonia, lability of serum potassium, non-progressive nature and lack of atrophy or hypertrophy of muscles. In some patients, myotonia is not increased by cold exposure (paramyotonia without cold paralysis). Patients may have a combination phenotype of PMC and HYPP.

EXPRESSÃO TECIDUAL(Tecido-específico)
Músculo esquelético
80.7 TPM
Adipose Visceral Omentum
29.9 TPM
Mama
21.0 TPM
Tecido adiposo
18.1 TPM
Tireoide
10.4 TPM
OUTRAS DOENÇAS (12)
potassium-aggravated myotoniacongenital myopathy 22A, classiccongenital myopathy 22B, severe fetalhyperkalemic periodic paralysis
HGNC:10591UniProt:P35499

Variantes genéticas (ClinVar)

305 variantes patogênicas registradas no ClinVar.

🧬 KCNJ2: NM_000891.3(KCNJ2):c.647A>G (p.Asn216Ser) ()
🧬 KCNJ2: NM_000891.3(KCNJ2):c.351del (p.Glu118fs) ()
🧬 KCNJ2: NM_000891.3(KCNJ2):c.557C>G (p.Pro186Arg) ()
🧬 KCNJ2: NM_000891.3(KCNJ2):c.*2776G>T ()
🧬 KCNJ2: NM_000891.3(KCNJ2):c.1009T>C (p.Tyr337His) ()
Ver todas no ClinVar

Classificação de variantes (ClinVar)

Distribuição de 4,433 variantes classificadas pelo ClinVar.

1773
2660
VUS (40.0%)
Benigna (60.0%)
VARIANTES MAIS SIGNIFICATIVAS
SCN4A: NM_000334.4(SCN4A):c.1508G>A (p.Gly503Asp) [Uncertain significance]
SCN4A: NM_000334.4(SCN4A):c.451A>G (p.Ser151Gly) [Uncertain significance]
GH-LCR: NM_000334.4(SCN4A):c.3560A>G (p.Tyr1187Cys) [Uncertain significance]
GH-LCR: NM_000334.4(SCN4A):c.4514C>T (p.Ala1505Val) [Uncertain significance]
GH-LCR: NM_000334.4(SCN4A):c.2144C>T (p.Ala715Val) [Uncertain significance]

Diagnóstico

Os sinais que médicos procuram e os exames que confirmam

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Tratamento e manejo

Remédios, cuidados de apoio e o que precisa acompanhar

Pipeline de tratamentos
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·Pré-clínico4
Medicamentos catalogadosEnsaios clínicos· 0 medicamentos · 8 ensaios
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Ensaios clínicos abertos e novidades científicas recentes

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Outros ensaios clínicos

11 ensaios clínicos encontrados, 2 ativos.

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Publicações mais relevantes

Timeline de publicações
730 papers (10 anos)

Mostrando amostra de 200 publicações de um total de 730

#1

Case Report of Andersen-Tawil Syndrome: Rare Presentation of a Rare Disease.

Annals of Indian Academy of Neurology2026 Jan 01

Andersen-Tawil syndrome (ATS) is a rare genetic disorder characterized by a triad of periodic paralysis, cardiac arrhythmias, and dysmorphic features, typically presenting in the first two decades of life. A 45-year-old man presented with acute lower motor neuron-type quadriparesis. He reported similar self-resolving episodes over the past 2 years, triggered by rest after exercise. There were no cardiac symptoms, dysmorphic features, or relevant family history. Biochemical evaluation revealed hypokalemia. Genetic testing confirmed a heterozygous nonsense mutation in the KCNJ2 gene (c.13C>T, p.Arg5Ter). Cardiac and dental evaluations were normal. He was treated with potassium supplementation and later acetazolamide, with no recurrence over 2 years. This case highlights a rare adult-onset presentation of ATS without cardiac or dysmorphic features and a negative family history, contributing to the limited adult ATS literature.

#2

Novel CACNA1S Mutation c.3491 A>C in Hypokalemic Periodic Paralysis: First Report with Functional Validation.

Journal of molecular neuroscience : MN2026 Feb 04

Hypokalemic periodic paralysis (HypoPP) is a muscle disease caused by abnormal ion channels and is characterized by recurrent skeletal muscle relaxation paralysis and hypokalemia. There are obvious triggers before disease onset, such as cold, excessive exercise, excessive consumption of sugary and high-energy foods, and overeating. The aim of this study was to elucidate the pathogenic mechanism of novel mutations in the voltage-dependent L-type calcium channel subunit alpha-1 S (CACNA1S) gene associated with HypoPP. Method: Whole-exome sequencing and American College of Medical Genetics and Genomics (ACMG) compliance analysis were performed, supplemented by serum potassium and blood biochemistry tests for bioinformatics analysis. We report a 13-year-old adolescent male patient with hypokalemic periodic paralysis, who complained of limb muscle weakness accompanied by pain for 10 h. Whole-exome sequencing revealed a mutation in the CACNA1S gene (NM_000069.3: exon27: c.3491 A>C [p. Glu1164Ala]), which was classified as an uncertain mutation. The clinical presentation and protein structure prediction of the gene mutation confirmed its pathogenic role and mechanism. The mutation caused a conformational change in the calcium ion channel. This study revealed a new mutation site in the HypoPP gene and proposed the possibility of a new pathogenesis. Moreover, obesity and low magnesium are two factors that induce HypoPP, which may increase the risk of disease.

#3

The Critical Care Phenotype of Hypokalemic Paralysis: Etiology, Outcomes, and Predictors of Respiratory Failure in a Retrospective Cohort Study.

Cureus2026 Feb

Hypokalemic periodic paralysis (HPP) presenting as acute quadriparesis is a neuromuscular emergency. While its etiology is described in general wards, its severe "critical care phenotype" in the intensive care unit (ICU) remains poorly characterized. We aimed to define this phenotype by analyzing the clinical profile, etiological spectrum, and predictors of life-threatening severity. A retrospective study was conducted of 12 patients (nine male, three female; median age: 31.5 years) admitted to a tertiary ICU (2015-2021) with acute quadriparesis and hypokalemia (median potassium: 1.75 mmol/L). We analyzed management and outcomes and compared patients requiring mechanical ventilation (MV+) with those who did not (MV-) using distribution-appropriate statistical methods to identify factors associated with respiratory failure. All patients presented with acute flaccid quadriparesis and areflexia. Five (41.7%) required invasive mechanical ventilation, defining a severe "critical care phenotype." A secondary cause was identified in eight patients (66.7%), including thyrotoxicosis (n=2), distal renal tubular acidosis (n=2), primary hyperaldosteronism, sepsis, dengue fever, and gastroenteritis. Critically, the need for mechanical ventilation was not associated with the degree of hypokalemia (MV+ 1.7 mmol/L vs. MV- 1.7 mmol/L, p=0.87) or other baseline characteristics. With potassium supplementation and targeted therapy, 11 patients (91.7%) achieved complete neurological recovery; one death occurred in a patient with sepsis. HPP in the ICU represents a distinct critical care phenotype with a high risk of respiratory failure. As the requirement for mechanical ventilation was not predicted by admission potassium levels, vigilant monitoring for respiratory muscle fatigue is warranted in all cases. Favorable outcomes are achievable with prompt correction and treatment of the underlying cause, reinforcing that HPP is a reversible ICU emergency.

#4

Hypokalemic Periodic Paralysis: A Case Report of Acute Flaccid Quadriparesis Mimicking Guillain-Barré Syndrome.

Cureus2026 Feb

Hypokalemic periodic paralysis (HPP) is an uncommon but reversible cause of acute flaccid paralysis. It can clinically resemble neurological emergencies such as Guillain-Barré syndrome (GBS), leading to potential misdiagnosis and delays in appropriate treatment. We report a 30-year-old man who presented with sudden-onset quadriparesis following an acute febrile illness with gastrointestinal symptoms characterized by fever, myalgia, arthralgia, and vomiting. Initial neurological assessment demonstrated proximal and distal weakness without sensory involvement. Laboratory tests revealed severe hypokalemia (2.2 mmol/L), mild hypomagnesemia, hypophosphatemia, and markedly elevated CRP and procalcitonin. An MRI of the brain and spine was unremarkable. Although GBS was considered, rapid improvement in muscle strength following intravenous (IV) and oral potassium supplementation supported the diagnosis of hypokalemic periodic paralysis. The patient made a full recovery with the correction of electrolytes. This case emphasizes the importance of early electrolyte evaluation in acute flaccid paralysis and highlights the need to distinguish hypokalemic paralysis from GBS, as timely potassium replacement leads to rapid and complete recovery.

#5

Sex determination and genetic screening of equine embryos with whole genome amplification and real-time PCR.

The Journal of reproduction and development2026 Mar 23

Reliable pre-implantation sex determination and genetic screening enables informed embryo transfer decisions in equine breeding while avoiding later interventions. We developed a streamlined workflow that couples rapid whole genome amplification (WGA) with a multiplex real-time PCR targeting ETSTY5 as a Y-specific marker and UBC as an autosomal control. On purified equine DNA, sex was correctly assigned down to 10 pg gDNA and to a single fibroblast cell. Direct testing of embryo biopsies without WGA yielded inconsistent results, whereas introducing a short WGA step produced tight allelic-discrimination clusters and 100% diagnostic calls, including in cloned embryos of known sex. The same WGA product supported targeted genotyping for inherited disease screening of hyperkalemic periodic paralysis (HYPP) and hereditary equine regional dermal asthenia (HERDA) alleles. This WGA plus real-time PCR pipeline supports robust and practical embryo sexing and targeted pre-implantation genetic diagnostics within in vitro produced (IVP) equine embryo and embryo transfer workflows.

Publicações recentes

Ver todas no PubMed

📚 EuropePMC1.853 artigos no totalmostrando 192

2026

The Critical Care Phenotype of Hypokalemic Paralysis: Etiology, Outcomes, and Predictors of Respiratory Failure in a Retrospective Cohort Study.

Cureus
2026

Hypokalemic Periodic Paralysis: A Case Report of Acute Flaccid Quadriparesis Mimicking Guillain-Barré Syndrome.

Cureus
2026

Sex determination and genetic screening of equine embryos with whole genome amplification and real-time PCR.

The Journal of reproduction and development
2026

Approach to the patient with severe hyperthyroidism-related complications.

The Journal of clinical endocrinology and metabolism
2026

Clinical features and advances in the genetics of periodic paralysis.

PeerJ
2026

Lithium-induced hypokalemic periodic paralysis: A rare and life-threatening complication in bipolar disorder management.

Indian journal of psychiatry
2025

Case Report: Dominant deafness-onychodystrophy syndrome and hypokalemic periodic paralysis in a single patient: a rare syndromic overlap.

Frontiers in pediatrics
2026

The Clinical, Histological, and Genetic Spectrum of RYR1 Variants-A Multi-Center Israeli Cohort Study.

Journal of clinical medicine
2026

Unmasking malignancy-thyrotoxic hypokalemic periodic paralysis and papillary thyroid carcinoma in a young male with Graves' disease: a case report.

AME case reports
2026

Steroid-induced Hypokalemic Periodic Paralysis.

Annals of African medicine
2026

Case Report of Andersen-Tawil Syndrome: Rare Presentation of a Rare Disease.

Annals of Indian Academy of Neurology
2026

Novel CACNA1S Mutation c.3491 A>C in Hypokalemic Periodic Paralysis: First Report with Functional Validation.

Journal of molecular neuroscience : MN
2026

Silent electrolyte imbalance unmasked by paralysis: a case of hypokalemia in a middle-aged woman.

Clinical biochemistry
2026

Hypokalemic periodic paralysis type 1 with respiratory involvement: A case report and critical review of the diagnostic and therapeutic approach.

Medicina clinica
2026

Coexistence of McArdle disease and hypokalemic periodic paralysis: A case report.

Medicina clinica
2026

Efficacy of a K+ Channel Agonist, XEN1101, For Preserving Contractility in Mouse Models of Hypokalemic Periodic Paralysis.

Muscle & nerve
2026

Hypokalemic Periodic Paralysis Associated With a Rare CACNA1S Variant (p.Leu1243Val): Expanding the Mutational Spectrum.

Case reports in genetics
2026

Efficacy of Retigabine in Treating Weakness in a Mouse Model of Hypokalemic Periodic Paralysis.

Muscle & nerve
2025

A Pediatric Case of Hypokalemic Periodic Paralysis With Fatigue and Myalgia.

Cureus
2025

Thyrotoxic Periodic Paralysis (TPP): A Comprehensive Review with Regional Insights from the Middle East.

Cureus
2026

[Functional analysis of the mutant channels associated with skeletal muscle channelopathies].

Nihon yakurigaku zasshi. Folia pharmacologica Japonica
2026

Perinatal Management of Andersen-Tawil Syndrome Using a Wearable Cardioverter-Defibrillator: A Case Report.

The journal of obstetrics and gynaecology research
2025

A Trajectory of Thyroid Function: From Thyrotoxic Paralysis to Post-ablative Hypothyroidism.

Cureus
2025

Thyrotoxic periodic paralysis: diagnostic and management considerations.

BMJ case reports
2025

Thyrotoxic Hypokalemic Periodic Paralysis: Pathophysiological Mechanisms.

Endocrinology and metabolism (Seoul, Korea)
2026

Safety and efficacy of dichlorphenamide in patients with periodic paralysis: A systematic review and meta-analysis.

Neuromuscular disorders : NMD
2026

Nonthyroidal illness syndrome and diagnostic utility of CSF mNGS: insights from a case series of neurological scrub typhus.

International journal of infectious diseases : IJID : official publication of the International Society for Infectious Diseases
2025

Thyrotoxic Periodic Paralysis in an Asian Male With Graves' Disease.

Cureus
2025

Exploring Etiologies of Hypokalemic Paralysis: A Case Series.

The Journal of the Association of Physicians of India
2025

Guillain-Barré Syndrome and Viral Thyroiditis-Coexisting Together or Viral Thyroiditis as a Cause of Guillain-Barré Syndrome: An Unsolved Enigma!

The Journal of the Association of Physicians of India
2025

Isolated Periodic-Paralysis-Like Syndrome in a Kinship With a Pathogenic MT-ATP6 Variant: A Case Report.

Muscle & nerve
2026

Genetic Study of a Greek Family with Hypokalemic Periodic Paralysis in Four Generations.

Advances in experimental medicine and biology
2025

Hypokalemic Periodic Paralysis: A Case Report.

Cureus
2025

Sudden Tetraplegia from Hypokalaemic Periodic Paralysis Due to Cacna1s Mutation: Should Genetic Testing be Performed More Often?

European journal of case reports in internal medicine
2025

Unilateral Upper Extremity Paralysis Secondary to Hypokalemia and Fasting: A Case Report.

Clinical practice and cases in emergency medicine
2025

Severe Hypokalemic Paralysis Unmasking Renal Tubular Acidosis in a Patient With Sjögren's Syndrome.

Cureus
2025

Muscle Channelopathies and Rhabdomyolysis.

Continuum (Minneapolis, Minn.)
2025

Hyperthyroid Hypokalemic Periodic Paralysis in a Nepali Male; A Case Report.

Clinical case reports
2025

Genetic analysis of a family with skeletal muscle ion channelopathy and hereditary spastic paraplegia type 7 caused by SCN4A and SPG7 double mutations.

Gene
2025

Glucocorticoid-induced Thyrotoxic Periodic Paralysis Following Lumbar Nerve Root Block.

Internal medicine (Tokyo, Japan)
2025

Respiratory Failure in Hyperthyroidism: Focus on Thyrotoxic Periodic Paralysis.

Cureus
2025

Clinical and molecular mechanistic insights into the WDR72 mutation.

BMJ case reports
2025

Twenty-year trend of thyrotoxicosis and thyrotoxic periodic paralysis: a population-based cohort study.

European thyroid journal
2025

Redefining periodic paralysis with CACNA1S mutation in a Spanish cohort.

Neurologia
2025

Hypokalemic Paralysis Is Not Always Periodic: A Case Series.

Case reports in medicine
2025

Hypokalemic Periodic Paralysis in a Patient With Primary Sjögren's Syndrome and Distal Renal Tubular Acidosis: A Case Report.

Clinical medicine insights. Case reports
2025

Hypokalemic Periodic Paralysis Can Be Attributed to Exogenous Hyperthyroidism Only After Exclusion of All Primary and Secondary Causes.

The American journal of medicine
2025

Generation of three iPSC lines from patients with CACNA1S related congenital myopathy.

Stem cell research
2025

Hyperthyroidism in Disguise: A Case of New-Onset Atrial Fibrillation and Acute Reversible Paralysis.

JCEM case reports
2025

Clinical, electromyographic, and biophysical characterization of the rare Nav1.4 channel mutation SCN4A L1436P.

Frontiers in physiology
2025

COVID-19 infection and intense physical activity in hypokalemic periodic paralysis.

Boletin medico del Hospital Infantil de Mexico
2025

Refractory Ventricular Arrhythmias in Thyrotoxic Periodic Paralysis: An Uncommon Presentation of Cardiogenic Shock.

Cureus
2025

Molecular genetics of skeletal muscle channelopathies.

Journal of human genetics
2025

Graves' disease and hypokalemic periodic paralysis: A case series.

The American journal of medicine
2025

Silent Paralysis: Recurrent Thyrotoxic Periodic Paralysis in a Young Hispanic Male With Graves' Disease.

AACE endocrinology and diabetes
2025

Discovery and Treatment of Action Potential-Independent Myotonia in Hyperkalemic Periodic Paralysis.

Annals of clinical and translational neurology
2025

Hypokalemic periodic paralysis associated with the atypical CACNA1S c.2690G>A (p.Arg897Lys) variant: description of 14 affected individuals from five families.

Neuromuscular disorders : NMD
2025

Rebound Hyperkalemia After Potassium Repletion in Thyrotoxic Periodic Paralysis: A Case Report and Review of Management Implications.

Cureus
2025

An Atypical Case of Sjögren's Syndrome: A Surprise Diagnosis.

Cureus
2025

Transient worsening of thyrotoxic myopathy following methimazole and metoprolol initiation in a 12-year-old girl: a case report and literature review.

Journal of pediatric endocrinology & metabolism : JPEM
2025

Hypokalemic Periodic Paralysis with Renal Tubular Acidosis in a Patient with Autoimmune Disorder.

The Journal of the Association of Physicians of India
2025

Anesthetic Considerations for Patients With Hypokalemic Periodic Paralysis Undergoing Ambulatory Surgery: A Case Report.

A&A practice
2025

Refractory ventricular fibrillation from thyrotoxic hypokalemic periodic paralysis.

The American journal of emergency medicine
2025

Rebound Hyperkalemia in Hypokalemic Thyrotoxic Periodic Paralysis.

The Neurohospitalist
2025

Unveiling Thyrotoxic Periodic Paralysis: A Rare Hyperthyroid Complication.

Cureus
2025

The association between renin and thyroid-related biomarkers with clinical characteristics and outcomes in hyperthyroid patients.

Science progress
2025

Exploring self-management of diet and physical activity in CACNA1S-related hypokalemic periodic paralysis: A qualitative interview study.

Journal of neuromuscular diseases
2025

Hypokelemic Periodic Paralysis in Pregnancy-A Rare Case Study.

Journal of obstetrics and gynaecology of India
2025

Comprehensive analysis of acute flaccid paralysis with and without myelitis in Taiwanese children.

Italian journal of pediatrics
2025

Recognizing a Rare Presentation: Hypokalemic Periodic Paralysis Secondary to Amphetamine Use.

Nephrology (Carlton, Vic.)
2025

When Potassium Takes a Break: A Case Series of 3 Cases on Hypokalemic Periodic Paralysis.

Clinical case reports
2025

Feasibility of 7 T 39 K/ 23 Na Magnetic Resonance Imaging for assessing muscular ion balance in hypokalemic periodic paralysis.

Investigative radiology
2025

Semaglutide reverses the chronic myopathy of hyperkalemic periodic paralysis: a case report.

BMC nephrology
2025

Atypical Electrophysiological Pattern in Hypokalemic Periodic Paralysis With CACNA1S Mutation: A Case Report.

Cureus
2025

Quetiapine-induced hypokalemic periodic paralysis in a pregnant woman: a case report.

Korean journal of family medicine
2025

Potassium-sensitive loss of muscle force in the setting of reduced inward rectifier K+ current: Implications for Andersen-Tawil syndrome.

Proceedings of the National Academy of Sciences of the United States of America
2025

Broadening the Phenotypic Range: KCNJ2 Variant Linked to Isolated Periodic Paralysis with Fixed Myopathy.

Annals of Indian Academy of Neurology
2025

A Rare Presentation of Sjögren's Syndrome With Hypokalemic Periodic Paralysis Treated Based on Renal Biopsy Findings.

Cureus
2025

STIM1 in-frame deletion of eight amino acids in a patient with moderate tubular aggregate myopathy/Stormorken syndrome.

Journal of medical genetics
2025

How nutrigenomics impacts equine health - A case study of vitamin E.

Journal of equine veterinary science
2025

CACNA1S Channelopathy - A Novel Missense Variant Causing Hypokalemic Periodic Paralysis.

Indian journal of pediatrics
2024

[Thyrotoxic hypokalemic periodic paralysis in a black African man in Abidjan (Côte d'Ivoire)].

Medecine tropicale et sante internationale
2025

Insulin-induced severe thyrotoxic periodic paralysis: A case report.

World journal of clinical cases
2025

Unraveling an Uncommon Encounter: Hypokalemic Periodic Paralysis with Brugada Phenocopy Amidst Hypokalemia.

European journal of case reports in internal medicine
2025

Hypokalemic Periodic Paralysis Induced by Factitious Hyperthyroidism for Weight Reduction.

The American journal of medicine
2025

CACNA1S-associated triadopathy presenting with myalgia, muscle weakness, and asymptomatic hyperCKemia.

Therapeutic advances in neurological disorders
2025

Hypokalemic periodic paralysis, a rare yet critical condition: A case report.

Medicine international
2025

Gitelman syndrome presenting with lower limb paralysis: a case report.

Journal of medical case reports
2024

Novel mutation in KCNJ2 gene causes long QT interval syndrome type 7 and learning disability: A case report.

Medicine
2025

A case of ascending paralysis in pregnancy: Thiamine deficiency mimicking Guillain-Barré syndrome.

Clinical medicine (London, England)
2025

Simultaneous Cases of Familial Hypokalemic Periodic Paralysis Induced by Illicit Injection of Betamethasone.

The Journal of emergency medicine
2024

[Hypokalemia: Not Just Tubulopathies].

Giornale italiano di nefrologia : organo ufficiale della Societa italiana di nefrologia
2025

Mechanisms underlying the distinct K+ dependencies of periodic paralysis.

The Journal of general physiology
2025

A Rare Case of Thyrotoxic Periodic Paralysis in a Patient With Concomitant Methimazole-Induced Agranulocytosis.

AACE clinical case reports
2025

Genetics of Muscle Disease.

The Veterinary clinics of North America. Equine practice
2025

Thyrotoxic periodic Paralysis With hypoxemia: A case report and a comprehensive review.

Radiology case reports
2024

Atypical Presentation of Andersen-Tawil Syndrome: Heart Failure with Reduced Ejection without Periodic Paralysis or Dysmorphic Features.

European journal of case reports in internal medicine
2024

Periodic paralysis across the life course: age-related phenotype transition and sarcopenia overlap.

Frontiers in neurology
2025

"A Paralyzing Snack": An Endocrine Cause of Paralysis.

Acta medica (Hradec Kralove)
2025

Anesthesia and Myopathies of Horses.

The Veterinary clinics of North America. Equine practice
2025

Muscle Contractility in Hypokalemic Periodic Paralysis.

Muscle & nerve
2026

Thyrotoxic Hypokalemic Periodic Paralysis Induced by High-Dose Insulin in an Adolescent Male with Type 1 Diabetes Mellitus.

Hormone research in paediatrics
2024

Acute Muscle Weakness in Graves' Disease: A Case Report of Hypokalemic Thyrotoxic Periodic Paralysis.

Cureus
2024

Thyrotoxic Periodic Paralysis With Graves' Disease: A Case Report.

Cureus
2024

Thyrotoxic Periodic Paralysis: A Unique Case Highlighting the Diagnostic Challenges and Management.

Cureus
2024

Quantitative Muscle MRI to Monitor Disease Progression in Hypokalemic Period Paralysis.

Neurology. Genetics
2024

Bartter Syndrome With Recurrent Hypokalemic Periodic Paralysis: A Case Report.

Cureus
2024

A pediatric case of Andersen-Tawil syndrome with slowly progressive myopathy.

Pediatrics international : official journal of the Japan Pediatric Society
2024

Hypokalemic periodic paralysis presenting as cardiac arrest.

BMJ case reports
2024

A Case Report on Hypokalemic Periodic Paralysis.

Cureus
2024

Thyrotoxic Periodic Paralysis: A Rare Cause of Quadriparesis in a Young and Seemingly Healthy Patient.

Medicina (Kaunas, Lithuania)
2024

Periodic Paralysis: A Case Series with a Literature Review.

Case reports in neurology
2024

[Andersen-Tawil Syndrome, a differential of bidirectional ventricular tachycardia: a case report].

Archivos peruanos de cardiologia y cirugia cardiovascular
2024

Thyrotoxic periodic paralysis - a retrospective study from Southern India.

European thyroid journal
2024

A Novel CLCN1 Gene Mutation Associated with Hypokalemic Periodic Paralysis in a Pregnant Woman.

The Israel Medical Association journal : IMAJ
2025

Periodic Paralysis in a Child With Thermosensitive Mitochondrial Trifunctional Protein Deficiency.

American journal of medical genetics. Part A
2024

Clinical, myopathological, and genetic features of two Chinese families with Andersen-Tawil syndrome.

Frontiers in neurology
2024

A novel case of Vibrio bacteremia in an immune-competent patient.

Indian journal of medical microbiology
2024

Familial hypokalemic periodic paralysis: a case induced by concurrent hyperthyroidism.

BMC nephrology
2024

Thyrotoxic periodic paralysis complicated by carbimazole-associated myositis.

BMJ case reports
2025

Genetic background of neonatal hypokalemia.

Pediatric nephrology (Berlin, Germany)
2024

A Late Diagnosis of Andersen-Tawil Syndrome in Teenage Siblings.

Pediatric neurology
2024

Unraveling the Clinical Complexity of Thyrotoxic Periodic Paralysis: A Case Report.

Cureus
2024

Thyrotoxic Periodic Paralysis in a Samoan Male With Metabolic Acidosis: A Case Report and Review of the Literature.

Cureus
2024

Pediatric neuromuscular channelopathies.

Handbook of clinical neurology
2024

Coincident Thyrotoxic Hypokalemic Periodic Paralysis and Cardiomyopathy.

Journal of investigative medicine high impact case reports
2024

Primary Sjogren's syndrome presenting as hypokalaemic periodic paralysis and acute pancreatitis.

BMJ case reports
2024

Thyrotoxic Periodic Paralysis: A Case Report with Patient Perspective.

Acute medicine
2024

Core myopathy in two siblings with a biallelic variant in the CACNA1S gene-A case series study.

Clinical case reports
2024

A single-center retrospective study on the clinical features of thyrotoxic periodic paralysis.

PloS one
2024

Torsade de Pointes Caused by a Compound Licorice Tablet.

International heart journal
2024

Convergence of Crisis: A Case Report of Diabetic Ketoacidosis Masking an Impending Thyroid Storm and Periodic Paralysis.

Cureus
2024

[A case report of a MODY 10 family presenting as hypokalemic periodic paralysis].

Zhonghua nei ke za zhi
2024

Anaesthetic management of a parturient with hypokalaemic periodic paralysis for caesarean section: A case report and review of the literature.

Anaesthesia and intensive care
2024

Quality of life in hypokalemic periodic paralysis - a survey.

Neuromuscular disorders : NMD
2024

Case report: thyrotoxic periodic paralysis, an unusual cause of hypokalemia.

Acta clinica Belgica
2025

The effect of systemic acetazolamide administration on intraocular pressure in healthy horses-A preliminary study.

Veterinary ophthalmology
2024

Thyrotoxic periodic paralysis: a case report and review of relevant pathophysiology.

Emergencias : revista de la Sociedad Espanola de Medicina de Emergencias
2024

Early-Onset Autosomal Dominant Myopathy with Vacuolated Fibers and Tubular Aggregates but No Periodic Paralysis, in a Patient with the c.1583G>A (p.R528H) mutation in the CACNA1S Gene.

Journal of neuromuscular diseases
2024

Diagnosis, management and outcomes of primary hypokalemic periodic paralysis during pregnancy.

Obstetric medicine
2024

Hyperandrogenism and Hypokalemic Thyrotoxic Periodic Paralysis in a North American Adolescent Girl.

JCEM case reports
2024

Delayed initiation of disease modifying therapy increases relapse frequency and motor disability in pediatric onset multiple sclerosis.

Multiple sclerosis and related disorders
2024

Acquired hyperkalaemia leading to periodic paralysis: an emergency department perspective.

BMJ case reports
2024

Atypical Normokalemic Case of Thyrotoxic Periodic Paralysis in a Pediatric Patient.

Cureus
2024

Hyperkalemic Periodic Paralysis Secondary to End-Stage Renal Disease and Excess Potato Consumption.

Cureus
2024

Phenotypic Variability of Andersen-Tawil Syndrome Due to Allelic Mutation c.652C>T in the KCNJ2 Gene-A New Family Case Report.

Biomolecules
2024

Case 13-2024: A 27-Year-Old Man with Leg Weakness.

The New England journal of medicine
2024

Genetic analysis of 37 cases with primary periodic paralysis in Chinese patients.

Orphanet journal of rare diseases
2024

Hypokalemic Periodic Paralysis: A Rare Case of a Descending Flaccid Paralysis.

Cureus
2024

Thyrotoxic Periodic Paralysis, an Unusual Presentation of Paralysis After Spinal Surgery: A Case Report.

Cureus
2024

Novel compound heterozygous mutations in SCN4A as a potential genetic cause contributing to myopathic manifestations: A case report and literature review.

Heliyon
2024

Allele frequency of muscular genetic disorders in bull-catching (vaquejada) quarter horses.

Journal of equine veterinary science
2024

Transcriptome and open chromatin analysis reveals the process of myocardial cell development and key pathogenic target proteins in Long QT syndrome type 7.

Journal of translational medicine
2024

Recurrent Thyrotoxic Periodic Paralysis As the Sole Clinical Manifestation of Untreated Hyperthyroidism.

Cureus
2024

Case report: Dihydropyridine receptor (CACNA1S) congenital myopathy, a novel phenotype with early onset periodic paralysis.

Frontiers in neurology
2024

A Rare Case of Hypokalemic Periodic Paralysis With Acute Urinary Retention: Diagnosis and Management.

Cureus
2024

A rare case of thyrotoxic periodic paralysis revealing Graves' disease in a young Malian.

Clinical case reports
2024

Physiotherapy Strategies in Hypokalemic Periodic Paralysis: A Case Report.

Cureus
2024

Hypokalemic Periodic Paralysis Type 2 Due to SCN4A Val1105Met Mutation: A Case Study.

Cureus
2024

Special electromyographic features in a child with paramyotonia congenita: A case report and review of literature.

World journal of clinical cases
2024

Gestational Transient Thyrotoxicosis Can Lead to Hypokalemic Periodic Paralysis.

Chonnam medical journal
2024

Artificial Intelligence Electrocardiography Detecting Thyrotoxic Periodic Paralysis Following a SARS-CoV-2 Infection.

The American journal of medicine
2024

Crossover randomized controlled trial of bumetanide to rescue an attack of exercise induced hand weakness in hypokalaemic periodic paralysis.

Neuromuscular disorders : NMD
2025

Intravenous methylprednisolone-induced hypokalaemic periodic paralysis in a thyrotoxic patient: a case report and literature review.

Orbit (Amsterdam, Netherlands)
2023

Thyrotoxic periodic paralysis presenting with quadriparesis and hyperreflexia.

BMJ case reports
2023

Graves' Disease With Initial Presentation of Thyrotoxic Periodic Paralysis.

Cureus
2023

Upper Limb Movement Execution Classification using Electroencephalography for Brain Computer Interface.

Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual International Conference
2024

A retrospective study of accuracy and usefulness of electrophysiological exercise tests.

Journal of neurology
2023

Newly Diagnosed Hypokalemic Periodic Paralysis Triggered by COVID-19.

Cureus
2023

Hypokalemic periodic paralysis in a teenage boy after an intense period of exercise: A rare case report.

Clinical case reports
2023

Diagnostics in skeletal muscle channelopathies.

Expert review of molecular diagnostics
2023

Hyperkalemic Periodic Paralysis in Twenty-Two Family Members Over Four Generations: A Rare Case Report.

Annals of Indian Academy of Neurology
2023

A Thyrotoxic Periodic Paralysis Case Study: From Weakness to Wellness.

Cureus
2023

An unusual stroke mimic: A case report.

SAGE open medical case reports
2023

Thyrotoxic Periodic Paralysis as an Ongoing Diagnostic Challenge: A Case Report and Literature Review.

Cureus
2024

Prevalence and risk factors of low vitamin D levels in children and adolescents with familial hypokalemic periodic paralysis.

European journal of pediatrics
2023

Case report: A novel CACNA1S mutation associated with hypokalemic periodic paralysis.

Frontiers in neurology
2023

A novel CACNA1S gene variant in a child with hypokalemic periodic paralysis: a case report and literature review.

BMC pediatrics
2023

Hypokalemic periodic paralysis: a 3-year follow-up study.

Journal of neurology
2024

Kidney manifestations of pediatric Sjögren's syndrome.

Pediatric nephrology (Berlin, Germany)
2023

Two Cases of Periodic Paralysis Associated With MCM3AP Variants.

Journal of clinical neuromuscular disease
2023

An Instance of Hypokalemic Periodic Paralysis in Adolescent Brothers: A Case Report.

Cureus
2023

Clinical features and recovery pattern of secondary hypokalaemic paralysis.

Journal of neurology
2023

Muscle MRI in periodic paralysis shows myopathy is common and correlates with intramuscular fat accumulation.

Muscle & nerve
2023

Hypokalemic Hypophosphatemic Thyrotoxic Periodic Paralysis Associated with Bipolar Disorder Therapy.

Cureus
2023

Thyrotoxic Periodic Paralysis With Hypokalemia: A Case Study.

Cureus
2024

Potential Benefit of Channel Activators in Loss-of-Function Primary Potassium Channelopathies Causing Heredoataxia.

Cerebellum (London, England)
2023

A case report of Andersen-Tawil syndrome misdiagnosed with myodystrophy.

Frontiers in neurology
2023

Code Pseudo Stroke - A Case of Hypokalaemic Periodic Paralysis Mimicking Stroke.

European journal of case reports in internal medicine
2023

Hypokalemic paralysis due to renal tubular acidosis: uncommon initial manifestation of primary Sjögren´s syndrome.

ARP rheumatology
2023

Care Recommendations for the Investigation and Management of Children With Skeletal Muscle Channelopathies.

Pediatric neurology
2023

Hypokalemic periodic paralysis as the first sign of thyrotoxicosis- a rare case report from Somalia.

Thyroid research
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Referências e fontes

Bases de dados externas citadas neste artigo

Publicações científicas

Artigos indexados no PubMed ligados a esta doença no grafo RarasNet — título, periódico e PMID direto da fonte, sem intermediação de IA.

  1. Case Report of Andersen-Tawil Syndrome: Rare Presentation of a Rare Disease.
    Annals of Indian Academy of Neurology· 2026· PMID 41643185mais citado
  2. Novel CACNA1S Mutation c.3491 A>C in Hypokalemic Periodic Paralysis: First Report with Functional Validation.
    Journal of molecular neuroscience : MN· 2026· PMID 41634283mais citado
  3. The Critical Care Phenotype of Hypokalemic Paralysis: Etiology, Outcomes, and Predictors of Respiratory Failure in a Retrospective Cohort Study.
    Cureus· 2026· PMID 41869263mais citado
  4. Hypokalemic Periodic Paralysis: A Case Report of Acute Flaccid Quadriparesis Mimicking Guillain-Barré Syndrome.
    Cureus· 2026· PMID 41869153mais citado
  5. Sex determination and genetic screening of equine embryos with whole genome amplification and real-time PCR.
    The Journal of reproduction and development· 2026· PMID 41866194mais citado
  6. Paralysis to Analysis: Unmasking Thyrotoxic Periodic Paralysis in a Middle-Aged Male Patient With Undiagnosed Graves Disease.
    Cureus· 2026· PMID 41960015recente
  7. Permanent weakness and myopathy in hypokalemic periodic paralysis.
    Acta Myol· 2026· PMID 41954147recente
  8. Lifestyle and dietary measures in Periodic Paralyses.
    Acta Myol· 2026· PMID 41954145recente
  9. Hypokalemic periodic paralysis associated with atypical CACNA1S in pregnancy: New challenge for a rare syndrome.
    Int J Gynaecol Obstet· 2026· PMID 41944566recente
  10. Unmasking Hypokalemic Periodic Paralysis: The Rare Role of Levothyroxine in a Pakistani Woman.
    AACE Endocrinol Diabetes· 2026· PMID 41938294recente

Bases de dados e fontes oficiais

Identificadores e referências canônicas usadas para montar este verbete.

  1. ORPHA:206976(Orphanet)
  2. MONDO:0016122(MONDO)
  3. GARD:20374(GARD (NIH))
  4. Variantes catalogadas(ClinVar)
  5. Busca completa no PubMed(PubMed)
  6. Q1788314(Wikidata)

Dados compilados pelo RarasNet a partir de fontes abertas (Orphanet, OMIM, MONDO, PubMed/EuropePMC, ClinicalTrials.gov, DATASUS, PCDT/MS). Este conteúdo é informativo e não substitui avaliação médica.

Conteúdo mantido por Agente Raras · Médicos e pesquisadores podem colaborar

Paralisia periódica
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Paralisia periódica

ORPHA:206976 · MONDO:0016122
CID-10
G72.3 · Paralisia periódica
CID-11
Ensaios
2 ativos
MedGen
UMLS
C0030443
EuropePMC
Wikidata
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