A síndrome cardiospondilocarpofacial é caracterizada por insuficiência mitral, surdez condutiva, baixa estatura e anomalias esqueléticas (fusão óssea envolvendo as vértebras cervicais, os ossículos e os ossos do carpo e do tarso). Foi descrito em três membros de uma família. O modo de herança é provavelmente autossômico dominante com penetrância incompleta.
Introdução
O que você precisa saber de cara
A síndrome cardiospondilocarpofacial é caracterizada por insuficiência mitral, surdez condutiva, baixa estatura e anomalias esqueléticas (fusão óssea envolvendo as vértebras cervicais, os ossículos e os ossos do carpo e do tarso). Foi descrito em três membros de uma família. O modo de herança é provavelmente autossômico dominante com penetrância incompleta.
Escala de raridade
<1/50kMuito rara
1/20kRara
1/10kPouco freq.
1/5kIncomum
1/2k
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Sinais e sintomas
O que aparece no corpo e com que frequência cada sintoma acontece
Partes do corpo afetadas
+ 18 sintomas em outras categorias
Características mais comuns
Os sintomas variam de pessoa para pessoa. Abaixo estão as 64 características clínicas mais associadas, ordenadas por frequência.
Linha do tempo da pesquisa
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Genética e causas
O que está alterado no DNA e como passa nas famílias
Genes associados
1 gene identificado com associação a esta condição. Padrão de herança: Autosomal dominant.
Serine/threonine kinase which acts as an essential component of the MAP kinase signal transduction pathway (PubMed:10094049, PubMed:11460167, PubMed:12589052, PubMed:16845370, PubMed:16893890, PubMed:21512573, PubMed:8663074, PubMed:9079627). Plays an important role in the cascades of cellular responses evoked by changes in the environment (PubMed:10094049, PubMed:11460167, PubMed:12589052, PubMed:16845370, PubMed:16893890, PubMed:21512573, PubMed:8663074, PubMed:9079627). Mediates signal transd
CytoplasmCell membrane
Frontometaphyseal dysplasia 2
A form of frontometaphyseal dysplasia, a progressive sclerosing skeletal dysplasia affecting the long bones and skull. Characteristic features include supraorbital hyperostosis, cranial hyperostosis, undermodeling of the small bones, flared metaphyses, and digital anomalies. Extra-skeletal manifestations include hearing loss, cardiac malformations, and stenosis, particularly of the upper airway and urinary tract. FMD2 inheritance is autosomal dominant.
Variantes genéticas (ClinVar)
78 variantes patogênicas registradas no ClinVar.
Classificação de variantes (ClinVar)
Distribuição de 20 variantes classificadas pelo ClinVar.
Vias biológicas (Reactome)
18 vias biológicas associadas aos genes desta condição.
Diagnóstico
Os sinais que médicos procuram e os exames que confirmam
Tratamento e manejo
Remédios, cuidados de apoio e o que precisa acompanhar
Onde tratar no SUS
Hospitais de referência no Brasil e o protocolo oficial do SUS (PCDT)
🇧🇷 Atendimento SUS — Síndrome cardio-espondilo-carpo-facial
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Dados de DATASUS/CNES, SBGM, ABNeuro e Ministério da Saúde. Sempre confirme a disponibilidade diretamente com o estabelecimento.
Pesquisa ativa
Ensaios clínicos abertos e novidades científicas recentes
Pesquisa e ensaios clínicos
Nenhum ensaio clínico registrado para esta condição.
Publicações mais relevantes
Genetic diagnosis and clinical characteristics analysis of cardiospondylocarpofacial syndrome in a Chinese family.
Cardiospondylocarpofacial syndrome (CSCFS) is an extremely rare autosomal dominant disorder resulting from variant in the MAP3K7 gene, which encodes the transforming growth factor-β-activated kinase 1 (TAK1). Only 26 cases of CSCFS have been reported worldwide. The main manifestations are growth retardation, hypotonia, dysmorphic facial features, skeletal and limb abnormalities, cardiac septal defects with valve dysplasia, cardiomyopathy, and deafness with inner ear malformations. In this study, we recruited an unrelated Chinese family with a patient diagnosed with CSCFS. Whole exome sequencing revealed a novel heterozygous variant, c.142G > A[p. (Gly48Arg)], in the MAP3K7 gene. The variant was confirmed by Sanger sequencing to be absent in other family members and is de novo. The patient described here has a similar dysmorphology profile to that associated with CSCFS. Compared with reported cases of CSCFS, our patient presented with new complications of short tongue tie, brain abnormalities including asymmetrical cerebral hemispheres with widening of the right frontotemporal exoptic hiatus, intestinal obstruction and intussusception. In addition, scoliosis, vertebral abnormalities, carpal/tarsal fusion, pectus excavatum, and cervical spine fusion were not found in our patient. The molecular diagnosis in this patient extends the known genetic spectrum of CSCFS. Furthermore, the specific manifestations in this case offer valuable additional clinical details regarding the syndrome.
Neonatal dilated cardiomyopathy and cardiospondylocarpofacial syndrome linked to a novel MAP3K7 gene mutation.
Cardiospondylocarpofacial syndrome (CSCF) is a rare congenital disorder characterized by growth impairment, polyvalvular heart diseases, and skeletal anomalies caused by a mutation in the mitogen-activated protein three kinase seven (MAP3K7) gene. It encodes transforming growth factor-β activated kinase1 (TAK1), a member of the mitogen-activated protein kinase (MAPK) family, and is responsible for abnormal skeletal and cardiac morphogenesis. We report a case of CSCF syndrome with a novel variant of the MAP3K7 gene c.710 C>T (p.F237s) in a newborn who has severe dilated cardiomyopathy (DCM) and congenital heart disease (CHD) and presented with acute heart failure (HF). DCM has not been reported before with CSCF. This case emphasizes the role of genetic testing in diagnosing the syndromic neonate with DCM.
A severe case of cardiospondylocarpofacial syndrome with a novel MAP3K7 variant.
Cardiospondylocarpofacial syndrome (CSCFS) is a congenital malformation characterized by growth retardation, facial features, short toes with carpal and tarsal fusion, extensive posterior neck vertebral fusion, congenital heart disease, and deafness. Here, we report a severe case of CSCFS with a novel variant, p.Thr187Ile, in MAP3K7. Thr187 is the main phosphorylation site for TGF-beta-activated kinase 1 encoded by MAP3K7, and this variant may cause significant abnormalities in downstream signaling.
Primary ciliary dyskinesia: additional diagnosis in a patient with cardiospondylocarpofacial syndrome.
A novel MAP3K7 mutation in a child with cardiospondylocarpofacial syndrome and orofacial clefting.
Here we present the case of a patient with a novel de novo, likely pathogenic, heterozygous MAP3K7 variant (c.528dupT, p.G177WfsX5) causing cardiospondylocarpofacial syndrome (CSCFS). The variant, which falls in exon 6, is the first frameshift or non-sense mutation to be connected to CSCFS and presents with a phenotype that shares features with other MAP3K7-linked pathologies, including frontometaphyseal dysplasia 2 (FMD2) and the syndrome arising from an interstitial 6q15 deletions which envelop the gene. Other known mutations associated with CSCFS are plotted in black text (1,2,3).
Publicações recentes
Genetic diagnosis and clinical characteristics analysis of cardiospondylocarpofacial syndrome in a Chinese family.
Neonatal dilated cardiomyopathy and cardiospondylocarpofacial syndrome linked to a novel MAP3K7 gene mutation.
Primary ciliary dyskinesia: additional diagnosis in a patient with cardiospondylocarpofacial syndrome.
A severe case of cardiospondylocarpofacial syndrome with a novel MAP3K7 variant.
TGF-β and BMP Signaling Pathways in Skeletal Dysplasia with Short and Tall Stature.
📚 EuropePMC13 artigos no totalmostrando 14
Genetic diagnosis and clinical characteristics analysis of cardiospondylocarpofacial syndrome in a Chinese family.
Frontiers in pediatricsNeonatal dilated cardiomyopathy and cardiospondylocarpofacial syndrome linked to a novel MAP3K7 gene mutation.
Annals of pediatric cardiologyPrimary ciliary dyskinesia: additional diagnosis in a patient with cardiospondylocarpofacial syndrome.
Anales de pediatriaA severe case of cardiospondylocarpofacial syndrome with a novel MAP3K7 variant.
Human genome variationTGF-β and BMP Signaling Pathways in Skeletal Dysplasia with Short and Tall Stature.
Annual review of genomics and human geneticsEarly and severe tricuspid valve dysplasia in a fetus with cardiospondylocarpofacial syndrome due to a variant c.616T>G p.(Tyr206Asp) in MAP3K7.
Prenatal diagnosisA novel MAP3K7 mutation in a child with cardiospondylocarpofacial syndrome and orofacial clefting.
Clinical geneticsThe MAP3K7 gene: Further delineation of clinical characteristics and genotype/phenotype correlations.
Human mutationGeneration of the induced pluripotent stem cell line UNIBSi017-A from an individual with cardiospondylocarpofacial syndrome and the MAP3K7 c.737-7A > G variant.
Stem cell researchExpanding the phenotypic spectrum of cardiospondylocarpofacial syndrome: From a detailed clinical and radiological observation of a boy with a novel missense variant in MAP3K7.
American journal of medical genetics. Part AA novel variant in MAP3K7 associated with an expanded cardiospondylocarpofacial syndrome phenotype.
Cold Spring Harbor molecular case studiesInsights into the molecular pathogenesis of cardiospondylocarpofacial syndrome: MAP3K7 c.737-7A > G variant alters the TGFβ-mediated α-SMA cytoskeleton assembly and autophagy.
Biochimica et biophysica acta. Molecular basis of diseaseA novel MAP3K7 splice mutation causes cardiospondylocarpofacial syndrome with features of hereditary connective tissue disorder.
European journal of human genetics : EJHGHeterozygous Mutations in MAP3K7, Encoding TGF-β-Activated Kinase 1, Cause Cardiospondylocarpofacial Syndrome.
American journal of human geneticsAssociações
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Comunidades
Grupos ativos de quem convive com esta doença aqui no Raras
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Referências e fontes
Bases de dados externas citadas neste artigo
Publicações científicas
Artigos indexados no PubMed ligados a esta doença no grafo RarasNet — título, periódico e PMID direto da fonte, sem intermediação de IA.
- Genetic diagnosis and clinical characteristics analysis of cardiospondylocarpofacial syndrome in a Chinese family.
- Neonatal dilated cardiomyopathy and cardiospondylocarpofacial syndrome linked to a novel MAP3K7 gene mutation.
- A severe case of cardiospondylocarpofacial syndrome with a novel MAP3K7 variant.
- Primary ciliary dyskinesia: additional diagnosis in a patient with cardiospondylocarpofacial syndrome.
- A novel MAP3K7 mutation in a child with cardiospondylocarpofacial syndrome and orofacial clefting.
- TGF-β and BMP Signaling Pathways in Skeletal Dysplasia with Short and Tall Stature.
Bases de dados e fontes oficiais
Identificadores e referências canônicas usadas para montar este verbete.
- ORPHA:3238(Orphanet)
- OMIM OMIM:157800(OMIM)
- MONDO:0008005(MONDO)
- GARD:2362(GARD (NIH))
- Variantes catalogadas(ClinVar)
- Busca completa no PubMed(PubMed)
- Q17540795(Wikidata)
Dados compilados pelo RarasNet a partir de fontes abertas (Orphanet, OMIM, MONDO, PubMed/EuropePMC, ClinicalTrials.gov, DATASUS, PCDT/MS). Este conteúdo é informativo e não substitui avaliação médica.
Conteúdo mantido por Agente Raras · Médicos e pesquisadores podem colaborar