Ketone Body Supplementation Exerts Renoprotective Effects Against Adenine-Induced Kidney Injury via OXCT1-Mediated Ketolysis in Mice.

Ketone body β-hydroxybutyrate restores neuronal Tau proteostasis via ketolysis-independent mechanism.

BDH1 regulates cardiomyocyte apoptosis and diabetic cardiomyopathy by modulating mitochondrial dynamics and attenuating oxidative stress.

Is Beta Ketothiolase Deficiency an Uncommon Disease or an Unsuspected Diagnosis? The Role of Genetic Biochemistry Approaches in Metabolic Acidosis.

Targeting lipid droplets in FUS-linked amyotrophic lateral sclerosis mitigates neuronal and astrocytic lipotoxicity.

Comparison of Ketogenesis and Ketolysis Defects: A Retrospective Single-Center Study of 30 Patients.

Ketone metabolites in metabolic dysfunction-associated steatotic liver disease progression: optimizing keto-therapeutic strategies.

Acute kidney injury through a metabolic lens: pathological reprogramming mechanisms and clinical translation potential.

Dietary lipids, not ketone body metabolites, influence intestinal tumorigenesis in a ketogenic diet.

OXCT1 succinylation and activation by SUCLA2 promotes ketolysis and liver tumor growth.

Targeting OXCT1-mediated ketone metabolism reprograms macrophages to promote antitumor immunity via CD8+ T cells in hepatocellular carcinoma.

Incidence of Inborn Errors of Metabolism in Newborn Infants: Five Years' Single-Center Experience, Jeddah, Saudi Arabia.

Bio-Hacking Better Health-Leveraging Metabolic Biochemistry to Maximise Healthspan.

Elevated serum β-hydroxybutyrate, a circulating ketone metabolite, accelerates colorectal cancer proliferation and metastasis via ACAT1.

Ketolysis is required for the proper development and function of the somatosensory nervous system.

Persistent moderate methylmalonic aciduria in a patient with methylmalonyl CoA epimerase deficiency.

The Effect of Ketogenic Diet on Shared Risk Factors of Cardiovascular Disease and Cancer.

Metabolites and Genes behind Cardiac Metabolic Remodeling in Mice with Type 1 Diabetes Mellitus.

Ketone bodies: from enemy to friend and guardian angel.

Clinical variability and outcome of succinyl-CoA:3-ketoacid CoA transferase deficiency caused by a single OXCT1 mutation: Report of 17 cases.

Sotagliflozin, a Dual SGLT1/2 Inhibitor, Improves Cardiac Outcomes in a Normoglycemic Mouse Model of Cardiac Pressure Overload.

Rethinking Fragility Fractures in Type 2 Diabetes: The Link between Hyperinsulinaemia and Osteofragilitas.

Ketogenic diet restrains aging-induced exacerbation of coronavirus infection in mice.

C4OH is a potential newborn screening marker-a multicenter retrospective study of patients with beta-ketothiolase deficiency in China.

Alzheimer's disease alters oligodendrocytic glycolytic and ketolytic gene expression.

Succinyl-CoA:3-oxoacid coenzyme A transferase (SCOT) deficiency: A rare and potentially fatal metabolic disease.

SGLT2 Inhibition Mediates Protection from Diabetic Kidney Disease by Promoting Ketone Body-Induced mTORC1 Inhibition.

2-methylacetoacetyl-coenzyme A thiolase (beta-ketothiolase) deficiency: one disease - two pathways.

Deficiency of 3-hydroxybutyrate dehydrogenase (BDH1) in mice causes low ketone body levels and fatty liver during fasting.

The recent insights into the function of ACAT1: A possible anti-cancer therapeutic target.

Persistent Hypoglycemia in Children: Targeted Gene Panel Improves the Diagnosis of Hypoglycemia Due to Inborn Errors of Metabolism.

When one disease is not enough: succinyl-CoA: 3-oxoacid coenzyme A transferase (SCOT) deficiency due to a novel mutation in OXCT1 in an infant with known phenylketonuria.

Clinical presentation and outcome in a series of 32 patients with 2-methylacetoacetyl-coenzyme A thiolase (MAT) deficiency.

Aberrant ketolysis fuels hepatocellular cancer progression.

Hepatocellular carcinoma redirects to ketolysis for progression under nutrition deprivation stress.

Hyperketonemia and ketosis increase the risk of complications in type 1 diabetes.

Normoglycemic Ketonemia as Biochemical Presentation in Ketotic Glycogen Storage Disease.