Raras
Buscar doenças, sintomas, genes...
Doença de Wilson
ORPHA:905CID-10 · E83.0CID-11 · 5C64.00OMIM 277900PCDT · SUSDOENÇA RARA

Uma doença hereditária muito rara que afeta vários sistemas do corpo, causando sintomas variados e que não são específicos, como problemas no cérebro e nos nervos, no fígado, mentais ou nos ossos e músculos. Esses problemas surgem devido ao acúmulo exagerado de cobre no organismo.

Mantido por Agente Raras·Colaborar como especialista →

Introdução

O que você precisa saber de cara

📋

Uma doença hereditária muito rara que afeta vários sistemas do corpo, causando sintomas variados e que não são específicos, como problemas no cérebro e nos nervos, no fígado, mentais ou nos ossos e músculos. Esses problemas surgem devido ao acúmulo exagerado de cobre no organismo.

Pesquisas ativas
27 ensaios
79 total registrados no ClinicalTrials.gov
Publicações científicas
2.398 artigos
Último publicado: 2026 Apr 15

Escala de raridade

CLASSIFICAÇÃO ORPHANET · BRASIL 2024
1-9 / 100 000
Ultra-rara
<1/50k
Muito rara
1/20k
Rara
1/10k
Pouco freq.
1/5k
Incomum
1/2k
Prevalência
6.0
Europe
Início
Adolescent
+ adult, childhood, elderly
🏥
SUS: Cobertura completaScore: 85%
PCDT disponível3 medicamentos CEAFCentros em: PA, PR, SC, RS, ES +8CID-10: E83.0
🇧🇷Dados SUS / DATASUS
PROCEDIMENTOS SIGTAP (2)
0202010538
Dosagem de ceruloplasmina (Wilson)lab_test
0202010562
Dosagem de cobre sérico
Você se identifica com essa condição?
O Raras está aqui pra te apoiar — com ou sem diagnóstico

Encontrou um erro ou informação desatualizada? Sugira uma correção →

Entender a doença

Do básico ao detalhe, leia no seu ritmo

Preparando trilha educativa...

Sinais e sintomas

O que aparece no corpo e com que frequência cada sintoma acontece

Partes do corpo afetadas

🫃
Digestivo
17 sintomas
🧠
Neurológico
14 sintomas
🦴
Ossos e articulações
7 sintomas
📏
Crescimento
5 sintomas
💪
Músculos
4 sintomas
🫘
Rins
4 sintomas

+ 50 sintomas em outras categorias

Características mais comuns

100%prev.
Atraso no desenvolvimento da linguagem
Obrigatório (100%)
100%prev.
Atraso no desenvolvimento da fala
Frequência: 2/2
100%prev.
Mal-estar
Obrigatório (100%)
100%prev.
Distonia de membro
Obrigatório (100%)
100%prev.
Tremor na mão
Obrigatório (100%)
100%prev.
Hiperbilirrubinemia
Frequência: 2/2
106sintomas
Muito frequente (58)
Frequente (13)
Ocasional (17)
Muito raro (1)
Sem dados (17)

Os sintomas variam de pessoa para pessoa. Abaixo estão as 106 características clínicas mais associadas, ordenadas por frequência.

Atraso no desenvolvimento da linguagemHP:0025710
Obrigatório (100%)100%
Atraso no desenvolvimento da falaHP:0025709
Frequência: 2/2100%
Mal-estarMalaise
Obrigatório (100%)100%
Distonia de membroLimb dystonia
Obrigatório (100%)100%
Tremor na mãoHand tremor
Obrigatório (100%)100%

Linha do tempo da pesquisa

Publicações por ano — veja quando o interesse científico cresceu
Anos de pesquisa1desde 2026
Total histórico2.398PubMed
Últimos 10 anos200publicações
Pico2025129 papers
Linha do tempo
2026Hoje · 2026🧪 1993Primeiro ensaio clínico📈 2025Ano de pico
Publicações por ano (últimos 10 anos)

Encontrou um erro ou informação desatualizada? Sugira uma correção →

Genética e causas

O que está alterado no DNA e como passa nas famílias

Genes associados

1 gene identificado com associação a esta condição. Padrão de herança: Autosomal recessive.

ATP7BCopper-transporting ATPase 2Disease-causing germline mutation(s) (loss of function) inTolerante
FUNÇÃO

Copper ion transmembrane transporter involved in the export of copper out of the cells. It is involved in copper homeostasis in the liver, where it ensures the efflux of copper from hepatocytes into the bile in response to copper overload

LOCALIZAÇÃO

Golgi apparatus, trans-Golgi network membraneLate endosomeGolgi apparatus membraneCytoplasmMitochondrion

VIAS BIOLÓGICAS (1)
Ion transport by P-type ATPases
MECANISMO DE DOENÇA

Wilson disease

An autosomal recessive disorder of copper metabolism in which copper cannot be incorporated into ceruloplasmin in liver, and cannot be excreted from the liver into the bile. Copper accumulates in the liver and subsequently in the brain and kidney. The disease is characterized by neurologic manifestations and signs of cirrhosis.

OUTRAS DOENÇAS (1)
Wilson disease
HGNC:870UniProt:P35670

Variantes genéticas (ClinVar)

1,257 variantes patogênicas registradas no ClinVar.

🧬 ATP7B: NM_000053.4(ATP7B):c.253G>A (p.Gly85Ser) ()
🧬 ATP7B: NM_000053.4(ATP7B):c.3917A>T (p.Asp1306Val) ()
🧬 ATP7B: NM_000053.4(ATP7B):c.465delinsTCCTGTGCA (p.Gln155fs) ()
🧬 ATP7B: NM_000053.4(ATP7B):c.3908A>C (p.Asp1303Ala) ()
🧬 ATP7B: NM_000053.4(ATP7B):c.3139G>A (p.Asp1047Asn) ()
Ver todas no ClinVar

Classificação de variantes (ClinVar)

Distribuição de 3,393 variantes classificadas pelo ClinVar.

339
679
2375
Patogênica (10.0%)
VUS (20.0%)
Benigna (70.0%)
VARIANTES MAIS SIGNIFICATIVAS
ATP7B: NM_000053.4(ATP7B):c.253G>A (p.Gly85Ser) [Likely pathogenic]
ATP7B: NM_000053.4(ATP7B):c.465delinsTCCTGTGCA (p.Gln155fs) [Pathogenic]
ATP7B: NM_000053.4(ATP7B):c.329A>C (p.Gln110Pro) [Uncertain significance]
ATP7B: NM_000053.4(ATP7B):c.4328C>T (p.Ala1443Val) [Uncertain significance]
ATP7B: NM_000053.4(ATP7B):c.3917A>T (p.Asp1306Val) [Uncertain significance]

Vias biológicas (Reactome)

1 via biológica associada aos genes desta condição.

Diagnóstico

Os sinais que médicos procuram e os exames que confirmam

Carregando...

Tratamento e manejo

Remédios, cuidados de apoio e o que precisa acompanhar

Pipeline de tratamentos
Pipeline regulatório — de medicamentos já aprovados a drogas em pesquisa exploratória.
3Fase 31
2Fase 22
1Fase 12
·Pré-clínico15
Medicamentos catalogadosEnsaios clínicos· 0 medicamentos · 20 ensaios
Carregando informações de tratamento...

Onde tratar no SUS

Hospitais de referência no Brasil e o protocolo oficial do SUS (PCDT)

🇧🇷 Atendimento SUS — Doença de Wilson

Centros de Referência SUS

21 centros habilitados pelo SUS para Doença de Wilson

Centros para Doença de Wilson

Detalhes dos centros

Hospital Universitário Prof. Edgard Santos (HUPES)

R. Dr. Augusto Viana, s/n - Canela, Salvador - BA, 40110-060 · CNES 0003808

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do Metabolismo

Hospital de Apoio de Brasília (HAB)

AENW 3 Lote A Setor Noroeste - Plano Piloto, Brasília - DF, 70684-831 · CNES 0010456

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital Estadual Infantil e Maternidade Alzir Bernardino Alves (HIABA)

Av. Min. Salgado Filho, 918 - Soteco, Vila Velha - ES, 29106-010 · CNES 6631207

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital das Clínicas da UFG

Rua 235 QD. 68 Lote Área, Nº 285, s/nº - Setor Leste Universitário, Goiânia - GO, 74605-050 · CNES 2338424

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do Metabolismo

Hospital das Clínicas da UFMG

Av. Prof. Alfredo Balena, 110 - Santa Efigênia, Belo Horizonte - MG, 30130-100 · CNES 2280167

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

NUPAD / Faculdade de Medicina UFMG

Av. Prof. Alfredo Balena, 189 - 5 andar - Centro, Belo Horizonte - MG, 30130-100 · CNES 2183226

Serviço de Referência

Rota
Erros Inatos do Metabolismo

Hospital Universitário João de Barros Barreto

R. dos Mundurucus, 4487 - Guamá, Belém - PA, 66073-000 · CNES 2337878

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital de Clínicas da Universidade Federal de Pernambuco

Av. Prof. Moraes Rego, 1235 - Cidade Universitária, Recife - PE, 50670-901 · CNES 2561492

Atenção Especializada

Rota
Erros Inatos do Metabolismo

Instituto de Medicina Integral Prof. Fernando Figueira (IMIP)

R. dos Coelhos, 300 - Boa Vista, Recife - PE, 50070-902 · CNES 0000647

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital de Clínicas da UFPR

R. Gen. Carneiro, 181 - Alto da Glória, Curitiba - PR, 80060-900 · CNES 2364980

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital Universitário Pedro Ernesto (HUPE-UERJ)

Blvd. 28 de Setembro, 77 - Vila Isabel, Rio de Janeiro - RJ, 20551-030 · CNES 2280221

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do Metabolismo

Instituto Nacional de Saúde da Mulher, da Criança e do Adolescente Fernandes Figueira (IFF/Fiocruz)

Av. Rui Barbosa, 716 - Flamengo, Rio de Janeiro - RJ, 22250-020 · CNES 2269988

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital Universitário Onofre Lopes (HUOL)

Av. Nilo Peçanha, 620 - Petrópolis, Natal - RN, 59012-300 · CNES 2408570

Atenção Especializada

Rota
Erros Inatos do Metabolismo

Hospital São Lucas da PUCRS

Av. Ipiranga, 6690 - Jardim Botânico, Porto Alegre - RS, 90610-000 · CNES 2232928

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do Metabolismo

Hospital de Clínicas de Porto Alegre (HCPA)

Rua Ramiro Barcelos, 2350 Bloco A - Av. Protásio Alves, 211 - Bloco B e C - Santa Cecília, Porto Alegre - RS, 90035-903 · CNES 2237601

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital Universitário da UFSC (HU-UFSC)

R. Profa. Maria Flora Pausewang - Trindade, Florianópolis - SC, 88036-800 · CNES 2560356

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do Metabolismo

Hospital das Clínicas da FMUSP

R. Dr. Ovídio Pires de Campos, 225 - Cerqueira César, São Paulo - SP, 05403-010 · CNES 2077485

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital de Clínicas da UNICAMP

R. Vital Brasil, 251 - Cidade Universitária, Campinas - SP, 13083-888 · CNES 2748223

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Hospital de Clínicas de Ribeirão Preto (HCRP-USP)

R. Ten. Catão Roxo, 3900 - Vila Monte Alegre, Ribeirão Preto - SP, 14015-010 · CNES 2082187

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do MetabolismoDeficiência Intelectual

Instituto da Criança e do Adolescente (ICr-HCFMUSP)

Av. Dr. Enéas Carvalho de Aguiar, 647 - Cerqueira César, São Paulo - SP, 05403-000 · CNES 2081695

Serviço de Referência

Rota
Erros Inatos do Metabolismo

UNIFESP / Hospital São Paulo

R. Napoleão de Barros, 715 - Vila Clementino, São Paulo - SP, 04024-002 · CNES 2688689

Serviço de Referência

Rota
Anomalias CongênitasErros Inatos do Metabolismo
Sobre os centros SUS: Estes centros são habilitados pelo Ministério da Saúde como Serviços de Referência em Doenças Raras ou Serviços de Atenção Especializada. O atendimento é pelo SUS, com encaminhamento da rede de atenção básica.

Dados de DATASUS/CNES, SBGM, ABNeuro e Ministério da Saúde. Sempre confirme a disponibilidade diretamente com o estabelecimento.

Pesquisa ativa

Ensaios clínicos abertos e novidades científicas recentes

🟢 Recrutando agora

16 pesquisas recrutando participantes. Converse com seu médico sobre a possibilidade de participar.

Outros ensaios clínicos

79 ensaios clínicos encontrados, 27 ativos.

Distribuição por fase
Ver todos no ClinicalTrials.gov
🧪 Está conduzindo uma pesquisa?
Divulgue para pacientes e familiares que acompanham esta doença.
Divulgar pesquisa →

Publicações mais relevantes

📖Melhor nível de evidência: Revisão
Timeline de publicações
1.176 papers (10 anos)

Mostrando amostra de 200 publicações de um total de 1.176

#1

Case Report: Genetic testing reveals Wilson disease with familial hypertriglyceridemia in a 12-year-old boy.

Frontiers in pediatrics2026

Wilson disease (WD) and familial hypertriglyceridemia (FHTG) are both genetic metabolic diseases, and their comorbidity is extremely rare. This article reports a case of WD with FHTG in a 12-year-old Chinese boy. The patient was diagnosed due to elevated transaminase levels, combined with clinical manifestations, copper metabolism indexes, lipid profile analysis, and genetic testing results (pathogenic mutations of ATP7B and APOA5). The patient was treated using a copper chelating agent to lower copper levels and fibrate drugs to lower lipid levels, which resulted in improvements in his liver function and blood lipid indices. This case serves as a source of reference for the diagnosis and treatment of other similar cases. It not only reveals the potential interaction between copper metabolism disorders and lipid abnormalities, but also highlights the importance of systematic genetic testing to identify comorbid inheritance.

#2

Evaluation of novel assays of non-ceruloplasmin copper to monitor chelation treatment in patients with Wilson disease: Monitoring chelation treatment in Wilson disease.

JHEP reports : innovation in hepatology2026 Mar 12

We examined the use of two newer measures of non-ceruloplasmin bound copper (NCC) and 24-hour urinary Cu excretion (UCE) to monitor chelation therapy in clinically stable Wilson Disease (WD). We post-hoc analyzed data from Chelate study, in which 77 clinically stable WD patients on penicillamine (DPA) entered a 12-week screening phase after which 53 were randomized to continued DPA or same dose trientine-tetrahydrochloride (TETA4) (weeks 12-60). Data included NCC measured by protein speciation (NCC-Sp), exchangeable copper (NCC-Ex), and UCE. In 32/53 patients with unchanged dose from week 1 to 60, NCC-Sp decreased from 57.9±21.1μg/L to 39.6±16.25μg/L (P=0.0002), while NCC-Ex decreased from 56.4±20.3μg/L to 46.2±11.5(P=0.01), likely due to improved adherence during participation in a clinical trial. UCE dropped by ∼50% after switching to TETA4 and gradually decreased in the DPA arm. Biomarker values did not reach steady state until week 60. The visit-to-visit coefficient of variance was 30% for NCC-Sp, 20% for NCC-Ex and 52% for UCE. Including all 45 patients who completed week 60, those with lower tertile values of NCC-Sp (16.3-30.9μg/L) and NCC-Ex (18.7-43.1μg/L) had lower and more stable AST and ALT, and higher and more stable S-Albumin and S-Protein than those with higher values. No neurological changes were noted despite these differences in NCC. Copper deficiency was not observed. Non-ceruloplasmin by protein speciation and exchangeable copper have potential to guide chelation in WD patients on maintenance therapy. Specific target ranges should be established, and we hypothesize they may include values below normal ranges. Further studies are required to improve our understanding of the responses to dose changes and non-adherence and if standardization of sampling conditions can reduce visit-to-visit variability. (NCT03539952) IMPACT AND IMPLICATIONS: The use of biomarkers of copper metabolism (non-ceruloplasmin and 24 hour urinary copper excretion) to monitor chelating treatment in Wilson Disease is poorly supported by data and development of newer methodologies (NCC-Ex and NCC-Sp) further supports re-evaluation in longitudinal studies. Our study suggests that in patients with stable Wilson disease, the response of NCC-Sp and NCC-Ex to a dose change may take as long as 6-12 months to achieve and with an intraindividual visit-to-visit variation coefficient of ≈ 20-25% this will impact clinical practice and decision making (requiring serial measurements) and estimation of sample size and longevity of clinical trials. Steady state NCC-Ex and NCC-Sp below the commonly recommended 50-150 μg/L range were associated with stable ALT, AST, S-albumin, and S-protein in contrast to values above 50 μg/L, where ALT and AST increase and S-albumin and S-protein decreased, suggesting that future prospective studies may lead to re-evaluation and changes to ranges for treatment goals for NCC-Ex and NCC-Sp. The lack of a similar association of UCE and clinical outcome and high variation (51%) question the current use of UCE to guide dosing in WD patients with stable disease.

#3

Precision Diagnosis of Wilson Disease Using a MultiGene Panel: Insights From a Prospective Cohort Study.

Neurology. Genetics2026 Apr

Wilson disease (WD) is an autosomal recessive disorder of copper metabolism. Conventional genetic diagnostics are low-throughput and may miss intronic, structural, or phenocopy variants, leading to delayed or missed diagnoses. In this study, we evaluate the utility of a custom next-generation sequencing (NGS) panel targeting the full-length ATP7B gene and 10 additional copper metabolism-related genes in patients with clinically suspected WD. We conducted a prospective cohort study of 144 individuals at our neurogenetic center. Variants identified by NGS were filtered and annotated with in silico tools and classified according to American College of Medical Genetics and Genomics guidelines. Confirmatory Sanger sequencing, multiplex ligation-dependent probe amplification, and reverse transcription PCR assays were performed as needed. Genetic confirmation of WD was achieved in 129 of 144 patients (90%), including 80 typical (Leipzig score ≥4) and 49 atypical (score <4) cases. Ten novel ATP7B variants, including deep intronic, noncanonical splice, and copy number variants, were identified using this panel. Among 15 genetically unresolved cases, 6 harbored variants in other copper metabolism-related genes but no pathogenic ATP7B variants. Notably, 1 patient with a Leipzig score of 4 had been clinically diagnosed with WD for years but was reclassified as spinocerebellar ataxia type 12 after panel testing revealed only a heterozygous CP variant and a CAG repeat expansion in PPP2R2B. Our comprehensive multigene NGS panel enables precise diagnosis of WD by detecting both classical and unconventional pathogenic variants, as well as distinguishing phenocopies. This improved diagnostic accuracy underscores the value of early genetic testing to guide timely intervention, especially in atypical or early-stage cases.

#4

Liver Iron Accumulation in Recompensated Wilson Disease on Chelation Therapy: A Prospective Evaluation in Children.

Journal of clinical and experimental hepatology2026

As serum ceruloplasmin has ferroxidase properties, we hypothesized that Wilson disease (WD) may have greater iron accumulation than other liver diseases. We aimed to assess liver iron overload in WD by evaluating the iron metabolism biomarkers and liver iron concentration (LIC). Compensated and recompensated WD patients with ≥3 years of chelation and serum exchangeable copper (ExCu) < 1.15 μmol/L were recruited and compared to controls. All patients underwent assessment of iron metabolism biomarkers and T2∗-weighted liver magnetic resonance imaging (MRI) for LIC. High LIC was defined as >1.5 mg/g dry weight (dw). Thirty-seven WD patients were compared to age, sex, and liver disease score-matched controls (n = 10). High LIC was seen in 49% WD vs. 10% controls (P = 0.027). In those with a duration of chelation ≥6 years vs. <6 years, high LIC was found in 89% vs. 58% (P = 0.03). High LIC was seen in 3/9 (30%), 7/15 (47%), and 8/13 (62%) of the WD patients in 3-5 years, 6-9 years, and 10-12 years of chelation therapy, respectively. In those with LIC >1.5 mg/g dw (n = 18) and LIC >2.0 mg/g dw (n = 10), longer duration of chelation therapy inversely correlated with serum ferroxidase activity (r = -0.7, P < 0.001; r = -0.75, P = 0.01 respectively). Serum ferritin had poor correlation with LIC (r = 0.177, P = 0.3). Mean (standard deviation) ExCu in high vs. normal LIC were 0.66 (0.27) vs. 0.94 (0.33), P = 0.01. High LIC is found in approximately half of WD patients, especially in those with ≥6 years of chelation therapy and low ExCu. MRI is recommended as a screening tool for iron overload in WD.

#5

Impact of COVID-19 infection in patients with inherited metabolic diseases: a National Multicenter Study from the French IMDs Healthcare Network for Rare Diseases.

Orphanet journal of rare diseases2026 Feb 14

The COVID-19 pandemic presented unique challenges for patients with inherited metabolic diseases (IMDs), particularly due to the risk of infection-related metabolic decompensation and disruptions to specialized care. We aimed to assess the impact of COVID-19 infection on the clinical course of patients with IMDs in a National Multicenter Study from the French IMDs Healthcare Network for Rare Diseases. This national French study included 317 IMD patients (69 children and 248 adults) with symptomatic or asymptomatic COVID-19 infection between January 2020 and January 2023. Most COVID-19 cases were mild to moderate. The frequency of symptomatic COVID-19 was similar in adults and children (234/248 [94.3%] vs. 56/64 [87.5%], p = 0.09). Children experienced more frequently metabolic destabilization than adults during a COVID-19 infection (17/67 [25.4%] vs. 33/248 [13.3%], p = 0.03). Moreover, the proportion of children admitted to the ICU was higher than that of adult patients (5/69 [7.2%] vs. 4/248 [1.6%], p = 0.04). Temporary suspension or delay of IMD-specific treatment due to COVID-19 was rare, affecting 3/64 (4.7%) children and 13/229 (5.7%) adults. Severe COVID-19 outcomes were uncommon, with only one death in the adult cohort and five cases of long-term sequelae (1 child, 4 adults). COVID-19 was generally mild to moderate in IMD patients and caused metabolic decompensation or imbalance in a minority of cases, with only rare interruptions to disease-specific treatment. We observed that COVID-19 more frequently worsened the condition of children with IMD compared to adults in our cohort of patients. The online version contains supplementary material available at 10.1186/s13023-026-04230-8.

Publicações recentes

Ver todas no PubMed

📚 EuropePMC1.580 artigos no totalmostrando 197

2026

Case Report: Genetic testing reveals Wilson disease with familial hypertriglyceridemia in a 12-year-old boy.

Frontiers in pediatrics
2026

Multiscale computational genomics in Wilson disease: from atomic dynamics to clinical prediction.

Frontiers in genetics
2026

Spectrum of pathogenic variants in ATP7B gene causing Wilson Disease in Mexican patients.

Archives of medical research
2026

The role of zinc transporter 1 (ZnT1) in health and disease: From molecular mechanisms to therapeutic opportunities.

European journal of medicinal chemistry
2026

Evaluation of novel assays of non-ceruloplasmin copper to monitor chelation treatment in patients with Wilson disease: Monitoring chelation treatment in Wilson disease.

JHEP reports : innovation in hepatology
2026

Relative Exchangeable Copper Confirms Wilson Disease and Supports Reclassification of the ATP7B p.Met665Ile Variant With Conflicting Pathogenicity Evidence.

American journal of medical genetics. Part A
2026

Exposure to copper induces oxidative stress and apoptosis in human MEG-01 cells.

Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS)
2026

Precision Diagnosis of Wilson Disease Using a MultiGene Panel: Insights From a Prospective Cohort Study.

Neurology. Genetics
2026

Liver Iron Accumulation in Recompensated Wilson Disease on Chelation Therapy: A Prospective Evaluation in Children.

Journal of clinical and experimental hepatology
2026

Dual carrier status for HUPRA (hyperuricemia, pulmonary hypertension, renal failure, and alkalosis) syndrome and Wilson disease in a young dialysis patient: clinical and genetic counseling implications.

Clinical transplantation and research
2026

Analyses of ATP7B mRNA in Nasopharyngeal Swab Samples Increase Yields of Wilson Disease Molecular Genetic Diagnostics.

Human mutation
2026

TikTok as a Platform for Patient Education and Health Information in Rare Genetic Diseases: Cross-Sectional Study.

JMIR formative research
2026

Neuropsychiatric Symptoms After Liver Transplant for Wilson's Disease: A US-Based Multicenter Retrospective Cohort Study.

Clinical transplantation
2026

Unmasking Wilson's Disease Through Severe Psychiatric Manifestations: A Case Report.

Cureus
2026

Patient Burden in the Treatment of Wilson Disease in the United States: An Analysis of Real-World Health Insurance Claims Data from the Komodo database.

Advances in therapy
2026

Generalized Dystonia in a Patient With Wilson Disease 5 Years After Liver Transplant: A Case Report.

Tremor and other hyperkinetic movements (New York, N.Y.)
2026

The ATP7B c.3316 G > A variant is associated with mild subphenotype in Wilson disease: a single-center cohort study.

Orphanet journal of rare diseases
2026

MLDP-AS: an optimized next-generation sequencing assay for enhanced detection of technically challenging variants in expanded carrier screening.

Journal of translational medicine
2026

Impact of COVID-19 infection in patients with inherited metabolic diseases: a National Multicenter Study from the French IMDs Healthcare Network for Rare Diseases.

Orphanet journal of rare diseases
2026

Genetic findings in ten Ecuadorian patients with suspected Wilson's disease.

Human genomics
2026

Gait deviations in adolescent patients with Wilson disease and their possible connections with biochemical markers: preliminary results.

European journal of pediatrics
2026

Spatiotemporal abnormalities in brain networks as a signature of neurological damage in Wilson's disease.

Clinical neurophysiology : official journal of the International Federation of Clinical Neurophysiology
2026

NON-REDUNDANT ROLES OF COPPER TRANSPORTERS ATP7A AND ATP7B IN NORADRENERGIC SIGNALING.

bioRxiv : the preprint server for biology
2026

Whole-exome sequencing illuminates unexplained pediatric cholestatic liver disease.

World journal of hepatology
2026

Evaluation of retinal microvascular changes and laboratory characteristics in children with Wilson disease: an optical coherence tomography angiography study.

Journal of AAPOS : the official publication of the American Association for Pediatric Ophthalmology and Strabismus
2026

Clinical and Demographic Profile of Wilson Disease in Young Adults: A Retrospective Study at a Tertiary Care Center in Peshawar, Pakistan.

Cureus
2026

Neurologic Manifestations of Hepatic and Gastrointestinal Disease.

Continuum (Minneapolis, Minn.)
2026

Deciphering Copper Homeostasis and Cuproptosis: Biological Mechanisms, Disease Connections, and Cutting-Edge Copper-Based Nanomedicine.

Molecular pharmaceutics
2026

High copper levels induce oxidative stress and inflammatory processes in a cell culture model of Wilson's disease.

Molecular and cellular biochemistry
2026

Diagnostic challenges in progressive familial intrahepatic cholestasis type 3 (PFIC3) misdiagnosed as Wilson's disease: A systematic review.

Advances in clinical and experimental medicine : official organ Wroclaw Medical University
2025

Re-examining the Diagnostic Criteria for Wilson's Disease: A Case Report and Literature Review.

Cureus
2025

Clinical and Molecular Analysis of ATP7B Variants Identified by Next-Generation Sequencing in Iraqi Adults With Wilson Disease.

Sultan Qaboos University medical journal
2026

Global burden of Wilson disease: a comprehensive evidence synthesis.

Orphanet journal of rare diseases
2026

Acute hepatitis of unknown origin in 38-year-old man: A case report.

Medicine
2026

Further elucidation on the effect of food on the pharmacokinetics of trientine.

European journal of clinical pharmacology
2026

Role of cuproptosis in digestive system tumors (Review).

International journal of molecular medicine
2025

Mitochondrial dysfunction in Wilson disease: a systematic review and meta-analysis across human and animal models.

Frontiers in molecular biosciences
2025

Study on the effect of mesenchymal stem cells on neural injury, inflammation and copper content in Wilson disease.

Frontiers in cellular neuroscience
2026

Comprehensive urinary proteomics using DIA and PRM for low-abundance protein profiling of Wilson disease.

Journal of chromatography. B, Analytical technologies in the biomedical and life sciences
2025

Discovery of newborn Wilson disease biomarkers via integrated next-generation sequencing and untargeted metabolomics.

Orphanet journal of rare diseases
2025

Wilson Disease Hiding in Plain Sight: A Case Report of Psychosis and Catatonia Revealing Underlying Liver Dysfunction.

Reports (MDPI)
2025

New insights into the effects of dissolution profiles on the pharmacokinetics of trientine dihydrochloride.

European journal of clinical pharmacology
2025

Comparative review of copper-associated chronic hepatitis in dogs and Wilson disease in humans.

Frontiers in veterinary science
2026

Repurposing melatonin's therapeutic potential in Wilson disease: Addressing copper overload and redox imbalance.

Redox biology
2025

Juvenile/adult-onset POLG-related disease unmasked by valproate-associated fulminant hepatic failure.

BMJ case reports
2025

Rapid transcellular hepatic copper depletion by ARBM-101 rescues severe liver damage in Wilson disease rodents.

Biomedicine &amp; pharmacotherapy = Biomedecine &amp; pharmacotherapie
2025

Carbon-ion Radiotherapy for Hepatocellular Cancer Arising in Transplanted Liver Tissue.

Anticancer research
2025

Coexisting elastosis perforans serpiginosa and acquired cutis laxa following long-term penicillamine in Wilson disease.

Skin health and disease
2025

[LncRNA Meg3 expression level is negatively correlated with liver fibrosis severity in patients with Wilson disease].

Nan fang yi ke da xue xue bao = Journal of Southern Medical University
2026

Melatonin ameliorates copper accumulation-induced cognitive impairment in Wilson disease via activation of the SIRT3/FOXO3α signaling pathway.

Neuropharmacology
2025

Rare but relevant: Genetic liver disease in the general medical setting.

Clinical medicine (London, England)
2026

Nuances in ATP7B Genetic Testing and Interpretation in India.

Journal of clinical and experimental hepatology
2026

From Radiocopper to Cold Copper: Mechanistic Modeling and Simulation to Define Clinical Response Criteria and Biomarkers for VTX-801 in Wilson Disease.

CPT: pharmacometrics &amp; systems pharmacology
2025

Haematological Predictors of Cirrhosis in Paediatric Wilson Disease: A Record-Based Analysis.

Cureus
2025

Uncovering the Critical Role of Cuproptosis in Wilson Disease: Insights Into Potential Therapeutic Targets.

Journal of cellular and molecular medicine
2025

Rare liver diseases - Etiology, diagnosis and management: A review.

Biomolecules &amp; biomedicine
2025

Management of Wilson disease across Europe: an international physician-oriented survey by the ERN-RARE Liver group.

Orphanet journal of rare diseases
2026

Copper in Human Health and Disease: Insights from Inherited Disorders.

Physiology (Bethesda, Md.)
2025

Multiple Genetic Analysis Unravels Pseudoexon in ATP7B in a Patient with Clinically Diagnosed Wilson Disease.

Movement disorders clinical practice
2025

Minimal Criteria to Screen for Wilson Disease: A Delphi Consensus in the United States.

International journal of hepatology
2025

Adherence, satisfaction, and quality of life in Wilson disease patients after switching to trientine tetrahydrochloride: observational data from a dual cohort study.

Frontiers in pharmacology
2025

Analysis of factors affecting early hepatic steatosis in pediatric patients with Wilson's disease in China: A retrospective study.

Medicine
2025

The role of copper dysregulation in Wilson disease: an expert opinion.

Frontiers in medicine
2025

Ventricular arrhythmia-induced syncope as the initial presentation of Wilson disease in a 4-year-old child: a rare case report.

Cardiology in the young
2025

[Recommendations from the European Association for the Study of the Liver and the European Reference Network for Rare Liver Diseases Clinical Practice Guidelines for hepatolenticular degeneration].

Zhonghua gan zang bing za zhi = Zhonghua ganzangbing zazhi = Chinese journal of hepatology
2025

Novel and less invasive biomarker assays to measure liver ATP7B in Wilson disease patients.

Scientific reports
2026

Inactivation of Atp7b Copper Transporter in Intestinal Epithelial Cells Is Associated with Altered Lipid Processing and Cell Growth Machinery Independent from Hepatic Copper Accumulation and Severity of Liver Histology.

The American journal of pathology
2026

Pathogenicity analysis and functional prediction of a rare LDLR variant in familial hypercholesterolemia combined with Wilson disease.

Genes &amp; genomics
2025

Hepatocyte-targeted lipid nanoparticle for full-length ATP7B mRNA delivery in Wilson disease.

Journal of controlled release : official journal of the Controlled Release Society
2025

Copper-associated Chronic Hepatitis in Dogs.

The Veterinary clinics of North America. Small animal practice
2025

Intrahepatic Cholangiocarcinoma in Wilson's Disease: 2 Case Reports.

GE Portuguese journal of gastroenterology
2025

Defining the clinical spectrum and genotype-phenotype correlations for CCDC115-CDG: A patient report and review of the literature.

Molecular genetics and metabolism
2025

Comparative management practices of Wilson disease in Californian and Italian providers.

Journal of health, population, and nutrition
2025

Diet quality and nutritional risk in patients with Wilson's disease: a cross-sectional study.

Frontiers in nutrition
2025

Brugada ECG Pattern in Wilson Disease: Genetic Coincidence or Triggered Phenocopy?

JACC. Case reports
2025

Positioning the Cu/Zn Ratio within the Diagnostic Framework of Wilson Disease: Methodological and Conceptual Considerations.

Pediatric gastroenterology, hepatology &amp; nutrition
2025

Revolutionizing Wilson disease prognosis: a machine learning approach to predict acute-on-chronic liver failure.

Journal of translational medicine
2025

Relative exchangeable copper: A highly specific and sensitive biomarker for Wilson disease diagnosis.

JHEP reports : innovation in hepatology
2025

Oral bis-choline tetrathiomolybdate rapidly improves copper balance in patients with Wilson disease.

Journal of hepatology
2025

The burden of Wilson's disease: Insights into clinical, psychological, and functional dimensions.

Rehabilitacion
2025

A Case of Wilson's Disease With Early Neuropsychiatric and Late Hepatic Manifestations.

Cureus
2025

Repurposing Melatonin in dual-mode for Wilson disease therapy as a Copper Chelator and an antioxidant agent.

bioRxiv : the preprint server for biology
2025

High-Calorie Diet Accelerates the Liver Tissue Degeneration and Induces Subcutaneous White-to-Brown Fat Conversion in Mice with a Single-Allele Atp7b Mutation.

The Journal of nutrition
2025

Isolated Stiffness as a Predominant Manifestation of Wilson Disease: A Case Report.

Clinical case reports
2025

Development and validation of a nomogram model for prediction of dyslipidemia in children with Wilson disease: a retrospective analysis.

Frontiers in endocrinology
2026

Methanobactin rapidly facilitates biliary copper excretion in a Wilson disease rat model visualised by 64Cu PET/MRI.

British journal of pharmacology
2026

Labile Bound Copper (LBC) and Total Serum Copper Concentrations in Newborns and Infants.

Biological trace element research
2026

Combined clinical and genomic analysis uncovers neonatal-onset Wilson disease in two siblings with idiopathic cholestasis.

Clinica chimica acta; international journal of clinical chemistry
2025

An adolescent with newly diagnosed Wilson disease having underlying type 1 diabetes: A previously unreported combination.

Journal of family medicine and primary care
2025

Mechanisms of copper metabolism and cuproptosis: implications for liver diseases.

Frontiers in immunology
2025

Metallothionein and neurodegenerative diseases.

Neural regeneration research
2025

Native liver survival with therapeutic plasma exchange in acutely decompensated hepatic Wilson Disease: revisiting the Dhawan index.

Hepatology international
2025

[Progress on the research of hepatolenticular degeneration].

Zhonghua gan zang bing za zhi = Zhonghua ganzangbing zazhi = Chinese journal of hepatology
2025

Serum copper and ceruloplasmin levels as biomarkers reflecting liver fibrosis in children with autoimmune hepatitis.

Clinical and experimental pediatrics
2025

Metallothioneins in the Pathogenesis of Liver Diseases: A Review.

International journal of hepatology
2025

Diagnosing Wilson Disease in Acute Liver Failure: Comparison of Existing and Experimental Biomarkers.

The American journal of gastroenterology
2025

Systematic Review and Meta-Analysis of Secondary Treatment Failure and Immunogenicity With Botulinum Neurotoxin A in Multiple Indications.

European journal of neurology
2025

Wilson Disease Masquerading as Nephrotic Syndrome: A Case Report.

Cureus
2025

Functional Screen of Wilson Disease ATP7B Variants Reveals Residual Transport Activities.

Human mutation
2025

Diagnostic Pitfalls in Wilson Disease with Autoimmune Features: A Case Report.

GE Portuguese journal of gastroenterology
2025

Hypocupraemia-related drug-refractory seizures in Wilson disease.

Practical neurology
2025

Optimizing therapeutic plasma exchange in Wilson disease-related liver failure: toward precision bridging strategies.

Hepatology international
2025

A rare presentation of Wilson disease with normal levels of serum ceruloplasmin and copper and MODY: A case report.

Medicine
2025

Atrophy related neuroimaging biomarkers for neurological and cognitive function in Wilson disease.

Neurological research and practice
2025

CLSI Validation of Exchangeable Copper Determination in Serum by ICP-MS: A Focus on Alzheimer's Disease and Wilson Disease.

Biomolecules
2025

Clinical application of expanded carrier screening based on next-generation sequencing in the Chinese population.

Archives of gynecology and obstetrics
2025

Mapping neurological symptoms and muscle tension representations in impaired gray matter volume of Wilson disease.

Frontiers in neurology
2025

Liver transplantation in patients with neurological Wilson disease: What can a five-decade systematic literature review teach us?

Transplantation reviews (Orlando, Fla.)
2025

Explainable machine learning model predicting neurological deterioration in Wilson's disease via MRI radiomics and clinical features.

Parkinsonism &amp; related disorders
2025

Pitfalls in the Diagnosis of Wilson Disease.

Current neurology and neuroscience reports
2025

Metabolic Dysfunction-Associated Steatotic Liver Disease in Patients and Mice with Wilson Disease.

The American journal of pathology
2025

Liver transplantation in Wilson disease: a single-center experience.

Orphanet journal of rare diseases
2025

A New Dystonia Phenotype in Wilson Disease.

JAMA neurology
2025

Predictive value of liver enzymes in long-term prognosis of hepatic Wilson disease: results from the Wilson AEEH registry.

Orphanet journal of rare diseases
2025

Wilson disease: time frame for improvement of neurological symptomology may exceed a decade.

Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology
2025

Postoperative Outcomes Following Liver Transplantation for Wilson's Disease: A Systematic Review and Meta-Analysis.

Clinical transplantation
2025

Acute Deep Vein Thrombosis Presents as an Early Complication of Wilson Disease.

Cureus
2025

Changes in the FXR-cistrome and alterations in bile acid physiology in Wilson disease.

Hepatology communications
2025

A single-cell transcriptomic atlas of immune cells in Wilson disease identifies copper-specific immune regulation.

iScience
2025

miRNA-29b-3p: An Important Target for Ameliorating Liver Fibrosis in Wilson Disease by Inhibiting Autophagy.

Current molecular medicine
2025

Apolipoprotein-decorated drug loaded liposomes mitigating copper intoxication: an in vitro and in vivo evidence-based study intervening Wilson disease.

Naunyn-Schmiedeberg's archives of pharmacology
2025

Mental and Physical Health in Wilson Disease Patients With SARS-CoV-2 Infection and Relevance of Long-COVID.

JIMD reports
2025

Psychiatric Presentation of Wilson's Disease: A Rare Disease With an Unusual Manifestation.

Cureus
2025

Clinical significance of platelet-to-white blood cell ratio in patients with Wilson disease: a retrospective cohort study.

PeerJ
2025

Plasma exchange improves survival with native liver in Wilson disease with new Wilson's index ≥ 11 & early hepatic encephalopathy.

Hepatology international
2025

From severe aplastic anemia with TERT variant to Wilson disease - associations or not.

Annals of hematology
2025

Emerging roles of cuproptosis in liver diseases.

Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
2025

Comparative evaluation of anterior segment optical coherence tomography and in vivo confocal microscopy for Kayser-Fleischer rings assessment in Wilson disease.

BMC ophthalmology
2025

Incidence and health burden of 20 rare neurological diseases in South China from 2016 to 2022: a hospital-based observational study.

Orphanet journal of rare diseases
2025

Relative Exchangeable Copper, Exchangeable Copper and Total Copper in the Diagnosis of Wilson Disease.

Liver international : official journal of the International Association for the Study of the Liver
2025

Copper in human health: From COVID 19 to neurodegenerative diseases.

Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS)
2025

Correlation between differences in the intestinal flora structure and Chinese medicine evidence in patients with Wilson disease-related liver fibrosis analyzed via high-throughput sequencing technology.

Bioscience of microbiota, food and health
2025

Building Clinical and Research Delivery Networks: A Blue Print for Multidisciplinary Management and Consensus in Wilson Disease.

GE Portuguese journal of gastroenterology
2025

Challenges and Recent Advances in Diagnosing Wilson Disease.

Journal of clinical and experimental hepatology
2025

Case report: Co-occurrence of Wilson's and Alexander's diseases revealed by genetic analysis.

Frontiers in neurology
2025

Practical and Multidisciplinary Review on Wilson Disease: The Portuguese Perspective.

GE Portuguese journal of gastroenterology
2025

Hepatic microtubule destabilization facilitates liver fibrosis in the mouse model of Wilson disease.

Journal of molecular medicine (Berlin, Germany)
2025

The Effect of Consanguineous Marriage on the Epidemiology of Wilson Disease among Children: A Report from Southern Israel.

The Israel Medical Association journal : IMAJ
2025

Wilson disease combined with polycystic ovary syndrome-clinical features, treatment, and outcome in Chinese patients.

BMC endocrine disorders
2025

[Gandou Bushen Decoction Ameliorates Cognitive Impairment in Wilson Disease Model TX Mice by Regulating Melatonin Synthesis via the SIRT3/FOXO3α Pathway].

Sichuan da xue xue bao. Yi xue ban = Journal of Sichuan University. Medical science edition
2025

Application of Anterior Segment Optical Coherence Tomography in Detecting Kayser-Fleischer Rings in Wilson Disease.

Cornea
2025

Prospective Study to Assess Long-Term Outcomes of Chelator-Based Treatment With Trientine Dihydrochloride in Patients With Wilson Disease.

JGH open : an open access journal of gastroenterology and hepatology
2025

Performance of Relative Exchangeable Copper for the Diagnosis of Wilson Disease in Acute Liver Failure.

Journal of inherited metabolic disease
2025

Lean Metabolic Dysfunction-Associated Steatotic Liver Disease: A Comparative Analysis of Hepatic and Oncological Outcomes.

Journal of clinical gastroenterology
2025

Copper-Induced Neurodegenerative Disorders and Therapeutic Potential of Curcumin-Loaded Nanoemulsion.

Toxics
2025

The Importance of Genetic Testing: A Case Report of Wilson's Disease in Two Siblings of a Three-Sibling Family.

Cureus
2025

Aceruloplasminemia as a rare hereditary disease: four case reports in a single center.

Proceedings (Baylor University. Medical Center)
2025

Unmasking Wilson Disease: A Rare Paediatric Case of Haemolysis and Hepatic Dysfunction Without Neurological Features.

Cureus
2025

Spectrum and classification of ATP7B variants with clinical correlation in children with Wilson disease.

Saudi medical journal
2025

Genetic map of Wilson disease in Spain - A great tool to improve diagnosis and screening.

Revista espanola de enfermedades digestivas
2025

Correlation of shear wave elastography with liver biopsy in children with Chronic Liver disease.

Pakistan journal of medical sciences
2025

Clinical and Molecular Spectrum of Wilson Disease in the Arab World: A Systematic Review.

Biochemical genetics
2025

Prion protein promotes copper toxicity in Wilson disease.

Nature communications
2025

Acute Encephalopathy and Refractory Hypokalemia in a 12-Year-Old Boy.

Iranian journal of child neurology
2024

Dystonia in a child with neurocysticercosis mimicking neuro Wilson disease.

Sudanese journal of paediatrics
2025

Wilson's disease in two siblings from Ecuador: Two case reports.

World journal of clinical cases
2025

Advancing Newborn Screening in Washington State: A Novel Multiplexed LC-MS/MS Proteomic Assay for Wilson Disease and Inborn Errors of Immunity.

International journal of neonatal screening
2024

Unprecedented Co-occurrence: Identification of a Pathogenic Genetic Variant in the KMT2B Gene in a Wilson Disease Patient with a Pathogenic ATP7B Mutation.

Annals of neurosciences
2025

A comparative analysis in monitoring 24-hour urinary copper in wilson disease: sampling on or off treatment?

Orphanet journal of rare diseases
2024

A clinical study showing the expression characteristics of cuproptosis markers in cases with Wilson disease.

Medicine
2025

Elevated Hepatic Copper Content in Porto-Sinusoidal Vascular Disorder (PSVD): Leading Down a Wrong Track.

Liver international : official journal of the International Association for the Study of the Liver
2024

Wilson disease in the USA: epidemiology and real-world patient characteristics based on a retrospective observational health claims study.

BMJ open
2025

Indian Childhood Cirrhosis: Report of 2 Cases With Review of Literature and Implication of Metallothionein Immunohistochemical Expression.

Pediatric and developmental pathology : the official journal of the Society for Pediatric Pathology and the Paediatric Pathology Society
2025

Genetic profiling of Wilson disease reveals a potential recurrent pathogenic variant of ATP7B in the Jordanian population.

Journal of pediatric gastroenterology and nutrition
2024

Iron and Copper Liver Concentrations in Wilson Disease.

Journal of gastrointestinal and liver diseases : JGLD
2024

Mildly Elevated Liver Transaminase Levels: Causes and Evaluation.

American family physician
2025

Targeted and non-targeted proteomics to identify the urinary protein biomarkers for Wilson disease.

Clinica chimica acta; international journal of clinical chemistry
2024

[Gandou Bushen Decoction improves spermatogenesis and promotes spermatogenic cell proliferation in Wilson disease TX mice by activating testicular ERK signaling pathway].

Nan fang yi ke da xue xue bao = Journal of Southern Medical University
2024

Acute Liver Failure Etiology Determines Long-Term Outcomes in Patients Undergoing Liver Transplantation: An Analysis of the UNOS Database.

Journal of clinical medicine
2025

Epidemiology and economic burden of Wilson disease in France: A nationwide population-based study.

Journal of inherited metabolic disease
2025

Brain morphometry in hepatic Wilson disease patients.

Journal of inherited metabolic disease
2024

Genetically Confirmed Wilson Disease: A Retrospective Cohort Study From Bahrain.

Cureus
2025

The Genomic Landscape of Wilson Disease in a Pan India Disease Cohort and Population-Scale Data.

Movement disorders clinical practice
2024

Copper and Colorectal Cancer.

Cancers
2024

Clinical experience on switching trientine tetrahydrochloride to trientine dihydrochloride in Wilson disease patients.

JIMD reports
2024

Systematic Analysis and Insights Into the Mutation Spectrum and Ethnic Differences in ATP7B Mutations Associated With Wilson Disease.

Biomarker insights
2025

Wilson Disease: Novel Diagnostic and Therapeutic Approaches.

Seminars in liver disease
2025

Deep brain stimulation for severe dystonia associated with Wilson disease: A prospective multicenter meta-analysis of an N-of-1 trial.

European journal of neurology
2024

Unusual Confluence: Exploring the Association of Biliary Atresia, Wilson Disease, and Iron Overload.

ACG case reports journal
2024

Zinc gluconate for Wilson disease.

Clinical parkinsonism &amp; related disorders
2024

Contrast-enhanced ultrasound findings of sclerotic nodules in Wilson disease: A case report.

Medicine
2025

Kayser-Fleischer ring and sunflower cataract in Wilson disease.

QJM : monthly journal of the Association of Physicians
2024

Trientine Tetrahydrochloride, From Bench to Bedside: A Narrative Review.

Drugs
2024

Neuroimaging Correlates with Clinical Severity in Wilson Disease: A Multiparametric Quantitative Brain MRI.

AJNR. American journal of neuroradiology
2024

Amantadine-induced psychosis in Wilson disease.

The National medical journal of India
2025

Dysfunction of ATP7B Splicing Variant Caused by Enhanced Interaction With COMMD1 in Wilson Disease.

Cellular and molecular gastroenterology and hepatology
2025

Validation of Metallothionein Immunohistochemistry as a Highly Sensitive Screening Test for Wilson Disease.

Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc
2024

Relative exchangeable copper, a high-quality biomarker for differentiation of Traditional Chinese Medicine syndrome in Wilson's disease.

Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan
2024

Structural lesions and transcriptomic specializations shape gradient perturbations in Wilson disease.

Brain communications
2025

Exchangeable serum copper: Adult and pediatric reference intervals and in vitro stability in a nordic cohort.

Clinica chimica acta; international journal of clinical chemistry
2024

A Qualitative Study Exploring Experiences in Caregiving for Patients With Advanced Wilson Disease.

The Journal of neuroscience nursing : journal of the American Association of Neuroscience Nurses
2024

Generation of an induced pluripotent stem cell (iPSC) line (IGIBi026-A) derived from Wilson disease patient harboring compound heterozygous mutations [c.2165dupT (p.R723Efs31) and c.C813A (p.C271*)] in the ATP7B gene.

Stem cell research
2025

Low penetrance of frequent ATP7B mutations explains the low prevalence of Wilson disease. Lessons from real-life registries.

Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
2025

Exchangeable copper for patients with Wilson disease at follow-up: Rethinking normal ranges or changing methodology.

Hepatology (Baltimore, Md.)
2024

Progressive familial intrahepatic cholestasis 3 Camouflaging as Wilson disease in a 12-year-old: a diagnostic Odyssey.

Gastroenterology and hepatology from bed to bench
2024

Phenotypic and genetic characterization of children with Wilson Disease from Northeast China.

BMC pediatrics
2024

Nontraumatic intra-diploic arachnoid cyst communicating with sphenoid bone and in close proximity to cavernous sinus in a known case of Wilson disease: A rare entity.

Radiology case reports
Ver todos os 1.580 no EuropePMC

Associações

Organizações que acompanham esta doença — pra ter apoio e orientação

Ainda não temos associações cadastradas para Doença de Wilson.

É de uma associação que acompanha esta doença? Fale com a gente →

Comunidades

Grupos ativos de quem convive com esta doença aqui no Raras

Ainda não existe comunidade no Raras para Doença de Wilson

Pacientes, familiares e cuidadores se organizam em comunidades pra compartilhar experiências, fazer perguntas e se apoiar. Você pode ser o primeiro.

Tire suas dúvidas

Perguntas, dicas e experiências compartilhadas aqui na página

Participe da discussão

Faça login para postar dúvidas, compartilhar experiências e interagir com especialistas.

Fazer login

Doenças relacionadas

Doenças com sintomas parecidos — ajudam quem ainda está buscando diagnóstico

Referências e fontes

Bases de dados externas citadas neste artigo

Publicações científicas

Artigos indexados no PubMed ligados a esta doença no grafo RarasNet — título, periódico e PMID direto da fonte, sem intermediação de IA.

  1. Case Report: Genetic testing reveals Wilson disease with familial hypertriglyceridemia in a 12-year-old boy.
    Frontiers in pediatrics· 2026· PMID 41867937mais citado
  2. Evaluation of novel assays of non-ceruloplasmin copper to monitor chelation treatment in patients with Wilson disease: Monitoring chelation treatment in Wilson disease.
    JHEP reports : innovation in hepatology· 2026· PMID 41831608mais citado
  3. Precision Diagnosis of Wilson Disease Using a MultiGene Panel: Insights From a Prospective Cohort Study.
    Neurology. Genetics· 2026· PMID 41788301mais citado
  4. Liver Iron Accumulation in Recompensated Wilson Disease on Chelation Therapy: A Prospective Evaluation in Children.
    Journal of clinical and experimental hepatology· 2026· PMID 41783652mais citado
  5. Impact of COVID-19 infection in patients with inherited metabolic diseases: a National Multicenter Study from the French IMDs Healthcare Network for Rare Diseases.
    Orphanet journal of rare diseases· 2026· PMID 41689122mais citado
  6. Specific Therapies For ALF: Viral, Autoimmune and Wilson Disease.
    J Intensive Care Med· 2026· PMID 41984025recente
  7. CRISPR/Cas9-mediated gene correction of Wilson disease H1069Q point mutation in patient-specific induced pluripotent stem cells.
    Gene Ther· 2026· PMID 41981235recente
  8. Undiagnosed Wilson Disease in Cryptogenic Cirrhosis: A Genetic Study.
    Ann Hepatol· 2026· PMID 41974228recente
  9. Diagnostic Accuracy of Exchangeable Copper for Grading the Severity of Wilson Disease.
    J Inherit Metab Dis· 2026· PMID 41952595recente
  10. A cross-sectional analysis of the quality and reliability of Wilson disease videos on Bilibili, Douyin, and Kuaishou.
    Sci Rep· 2026· PMID 41935168recente

Bases de dados e fontes oficiais

Identificadores e referências canônicas usadas para montar este verbete.

  1. ORPHA:905(Orphanet)
  2. OMIM OMIM:277900(OMIM)
  3. MONDO:0010200(MONDO)
  4. Doenca de Wilson(PCDT · Ministério da Saúde)
  5. GARD:7893(GARD (NIH))
  6. Variantes catalogadas(ClinVar)
  7. Busca completa no PubMed(PubMed)
  8. Artigo Wikipedia(Wikipedia)
  9. Q117121(Wikidata)

Dados compilados pelo RarasNet a partir de fontes abertas (Orphanet, OMIM, MONDO, PubMed/EuropePMC, ClinicalTrials.gov, DATASUS, PCDT/MS). Este conteúdo é informativo e não substitui avaliação médica.

Conteúdo mantido por Agente Raras · Médicos e pesquisadores podem colaborar

Doença de Wilson
Compêndio · Raras BR

Doença de Wilson

ORPHA:905 · MONDO:0010200
🇧🇷 Brasil SUS
CEAF
1BD-Penicilamina1ATrientinaAcetato de zinco
Geral
Prevalência
1-9 / 100 000
Herança
Autosomal recessive
CID-10
E83.0 · Distúrbios do metabolismo do cobre
CID-11
Ensaios
27 ativos
Início
Adolescent, Adult, Childhood, Elderly
Prevalência
6.0 (Europe)
MedGen
UMLS
C0019202
EuropePMC
Wikidata
Wikipedia
Papers 10a
DiscussaoAtiva

Nenhuma novidade ainda. O agente esta monitorando.

0membros
0novidades